All Relations between conditioned response and fatty acid amide hydrolase

Reference Sentence Publish Date Extraction Date Species
William G Warren, Eleni P Papagianni, Ed Hale, Rebecca A Brociek, Helen J Cassaday, Carl W Stevenso. Endocannabinoid metabolism inhibition has no effect on spontaneous fear recovery or extinction resistance in Lister hooded rats. Frontiers in pharmacology vol 13 issue 2023 36588687 in two experiments using auditory fear conditioned rats, we examined the effects of the faah inhibitor urb597 and the magl inhibitor jzl184 administered systemically on 1) spontaneous fear recovery after delayed extinction, and 2) extinction resistance resulting from immediate extinction [the immediate extinction deficit (ied)]. 2023-01-02 2023-01-05 Not clear
Nicole L Zabik, Allesandra S Iadipaolo, Hilary A Marusak, Craig Peters, Kyle Burghardt, Christine A Rabina. A common genetic variant in fatty acid amide hydrolase is linked to alterations in fear extinction neural circuitry in a racially diverse, nonclinical sample of adults. Journal of neuroscience research vol issue 2021 34051704 in the present study, we used a novel, immersive-reality fear extinction paradigm paired with functional neuroimaging to assess faah c385a effects on fear-related neural circuitry and conditioned fear responding (us expectancy ratings, subjective units of distress, and skin conductance responding) in healthy adults from an urban area (detroit, mi; n\xc2\xa0=\xc2\xa059; c/c\xc2\xa0=\xc2\xa035, a-carrier\xc2\xa0=\xc2\xa024). 2021-11-30 2022-01-13 Not clear
Maria Morena, Robert J Aukema, Kira D Leitl, Asim J Rashid, Haley A Vecchiarelli, Sheena A Josselyn, Matthew N Hil. Upregulation of Anandamide Hydrolysis in the Basolateral Complex of Amygdala Reduces Fear Memory Expression and Indices of Stress and Anxiety. The Journal of neuroscience : the official journal of the Society for Neuroscience vol 39 issue 7 2019 30573646 surprisingly, faah overexpression in bla dampened stress-induced corticosterone release, reduced anxiety-like behaviors, and decreased conditioned fear expression. 2019-12-11 2022-01-13 Not clear
Francisco J Pavon, Antonia Serrano, Nimish Sidhpura, Ilham Polis, David Stouffer, Fernando Rodriguez de Fonseca, Benjamin F Cravatt, R\\xc3\\xa9mi Martin-Fardon, Loren H Parson. Fatty acid amide hydrolase (FAAH) inactivation confers enhanced sensitivity to nicotine-induced dopamine release in the mouse nucleus accumbens. Addiction biology vol 23 issue 2 2019 28660730 these observations in mice suggest that enhanced nicotine-induced nac da release might contribute to increased sensitivity to the conditioned rewarding effects of low-dose nicotine following faah inhibition, which has been previously reported. 2019-09-17 2022-01-13 Not clear
Francisco J Pavon, Antonia Serrano, Nimish Sidhpura, Ilham Polis, David Stouffer, Fernando Rodriguez de Fonseca, Benjamin F Cravatt, R\\xc3\\xa9mi Martin-Fardon, Loren H Parson. Fatty acid amide hydrolase (FAAH) inactivation confers enhanced sensitivity to nicotine-induced dopamine release in the mouse nucleus accumbens. Addiction biology vol 23 issue 2 2019 28660730 previous research has reported that both genetic deletion and pharmacological inhibition of faah enhance nicotine-induced conditioned place preference at low doses. 2019-09-17 2022-01-13 Not clear
Francisco J Pavon, Antonia Serrano, Nimish Sidhpura, Ilham Polis, David Stouffer, Fernando Rodriguez de Fonseca, Benjamin F Cravatt, R\\xc3\\xa9mi Martin-Fardon, Loren H Parson. Fatty acid amide hydrolase (FAAH) inactivation confers enhanced sensitivity to nicotine-induced dopamine release in the mouse nucleus accumbens. Addiction biology vol 23 issue 2 2019 28660730 we conducted a microdialysis study to characterize nicotine-induced changes in da and serotonin (5-ht) levels in the nac of faah knockout (ko) mice using a conditioned place preference-like paradigm with three nicotine doses (0.1, 1 and 10\xc2\xa0mg/kg, s.c.). 2019-09-17 2022-01-13 Not clear
Cheryl L Limebeer, Erin M Rock, Nirushan Puvanenthirarajah, Micah J Niphakis, Benjamin F Cravatt, Linda A Parke. Elevation of 2-AG by monoacylglycerol lipase inhibition in the visceral insular cortex interferes with anticipatory nausea in a rat model. Behavioral neuroscience vol 130 issue 2 2016 26974857 here we report that bilateral infusion of the magl inhibitor, mjn110 (but neither the faah inhibitor, pf3845, nor ondansetron) into the vic suppressed contextually elicited conditioned gaping, and this effect was reversed by coadministration of the cb1 antagonist, am251. 2016-12-13 2022-01-12 Not clear
Thomas F Gamage, Bogna M Ignatowska-Jankowska, Pretal P Muldoon, Benjamin F Cravatt, M Imad Damaj, Aron H Lichtma. Differential effects of endocannabinoid catabolic inhibitors on morphine withdrawal in mice. Drug and alcohol dependence vol 146 issue 2015 25479915 the present study investigated whether \xce\x94(9)-tetrahydrocannabinol (thc), the magl inhibitor jzl184, the faah inhibitor pf-3845, or the dual faah/magl inhibitor sa-57 would reduce acquisition of morphine withdrawal-induced conditioned place avoidance (cpa) and jumping. 2015-12-18 2022-01-12 Not clear
Laurie A Manwell, Paul E Malle. Comparative effects of pulmonary and parenteral \\xce\\x94\\xe2\\x81\\xb9-tetrahydrocannabinol exposure on extinction of opiate-induced conditioned aversion in rats. Psychopharmacology vol 232 issue 9 2015 25395060 we demonstrated that the fatty acid amide hydrolase (faah) inhibitor urb597, which increases ecb levels, facilitates extinction of a naloxone-precipitated morphine withdrawal-induced conditioned place aversion (cpa). 2015-11-20 2022-01-12 Not clear
Linda A Parker, Erin M Rock, Cheryl L Limebee. Regulation of nausea and vomiting by cannabinoids. British journal of pharmacology vol 163 issue 7 2012 21175589 cannabinoid agonists (\xce\x94(9) -thc, hu-210) and the fatty acid amide hydrolase (faah) inhibitor, urb-597, suppress conditioned gaping reactions (nausea) in rats as they suppress vomiting in emetic species. 2012-01-18 2022-01-12 Not clear
Amanda L McCallum, Cheryl L Limebeer, Linda A Parke. Reducing endocannabinoid metabolism with the fatty acid amide hydrolase inhibitor, URB597, fails to modify reinstatement of morphine-induced conditioned floor preference and naloxone-precipitated morphine withdrawal-induced conditioned floor avoidance. Pharmacology, biochemistry, and behavior vol 96 issue 4 2010 20643159 the potential of the fatty acid amide hydrolase (faah) inhibitor, urb597, to modify drug prime-induced reinstatement of morphine-induced conditioned floor preference or naloxone-precipitated morphine withdrawal-induced conditioned floor avoidance was evaluated. 2010-12-23 2022-01-12 Not clear
Antonio Luchicchi, Salvatore Lecca, Stefano Carta, Giuliano Pillolla, Anna L Muntoni, Sevil Yasar, Steven R Goldberg, Marco Pisti. Effects of fatty acid amide hydrolase inhibition on neuronal responses to nicotine, cocaine and morphine in the nucleus accumbens shell and ventral tegmental area: involvement of PPAR-alpha nuclear receptors. Addiction biology vol 15 issue 3 2010 20477753 we recently demonstrated that inhibition by urb597 of fatty acid amide hydrolase (faah), the main enzyme that degrades the endogenous cannabinoid n-acylethanolamine (nae) anandamide and the endogenous non-cannabinoid naes oleoylethanolamide and palmitoylethanolamide, blocks nicotine-induced excitation of ventral tegmental area (vta) dopamine (da) neurons and da release in the shell of the nucleus accumbens (shnac), as well as nicotine-induced drug self-administration, conditioned place preference and relapse in rats. 2010-11-02 2022-01-12 Not clear
P Adamczyk, A C McCreary, E Przegalinski, P Mierzejewski, P Bienkowski, M Fili. The effects of fatty acid amide hydrolase inhibitors on maintenance of cocaine and food self-administration and on reinstatement of cocaine-seeking and food-taking behavior in rats. Journal of physiology and pharmacology : an official journal of the Polish Physiological Society vol 60 issue 3 2010 19826190 our results indicate that faah inhibitors could be potent modulators of motivational and conditioned aspects of goal-directed behaviors with less prominent effects on consumatory behaviors. 2010-03-30 2022-01-12 Not clear
Laurie A Manwell, Elham Satvat, Stefan T Lang, Craig P Allen, Francesco Leri, Linda A Parke. FAAH inhibitor, URB-597, promotes extinction and CB(1) antagonist, SR141716, inhibits extinction of conditioned aversion produced by naloxone-precipitated morphine withdrawal, but not extinction of conditioned preference produced by morphine in rats. Pharmacology, biochemistry, and behavior vol 94 issue 1 2009 19698735 faah inhibitor, urb-597, promotes extinction and cb(1) antagonist, sr141716, inhibits extinction of conditioned aversion produced by naloxone-precipitated morphine withdrawal, but not extinction of conditioned preference produced by morphine in rats. 2009-12-17 2022-01-12 Not clear
Maria Scherma, Julie Medalie, Walter Fratta, Subramanian K Vadivel, Alexandros Makriyannis, Daniele Piomelli, Eva Mikics, Jozsef Haller, Sevil Yasar, Gianluigi Tanda, Steven R Goldber. The endogenous cannabinoid anandamide has effects on motivation and anxiety that are revealed by fatty acid amide hydrolase (FAAH) inhibition. Neuropharmacology vol 54 issue 1 2008 17904589 however, when rats were pre-treated with the fatty acid amide hydrolase (faah) inhibitor urb597 (cyclohexyl carbamic acid 3'-carbamoyl-3-yl ester; 0.3 mg/kg intraperitoneal), which blocks anandamide's metabolic degradation, anandamide produced dose-related conditioned place aversions. 2008-04-18 2022-01-12 Not clear
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