| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Shunsuke Kobayashi, Toru Tanaka, Yoshiyuki Soeda, Osborne F X Almeida, Akihiko Takashim. Local Somatodendritic Translation and Hyperphosphorylation of Tau Protein Triggered by AMPA and NMDA Receptor Stimulation. EBioMedicine. vol 20. 2018-03-26. PMID:28566250. |
local somatodendritic translation and hyperphosphorylation of tau protein triggered by ampa and nmda receptor stimulation. |
2018-03-26 |
2023-08-13 |
Not clear |
| Jane C Hettinger, John R Cirrit. Bad Ac-etylated Tau. Neuron. vol 90. issue 2. 2017-10-26. PMID:27100190. |
(2016) found that acetylated tau promotes synaptic dysfunction through disruption of postsynaptic kibra signaling pathways, actin dynamics, and ampa receptor trafficking. |
2017-10-26 |
2023-08-13 |
Not clear |
| Philip Regan, Thomas Piers, Jee-Hyun Yi, Dong-Hyun Kim, Seonghoo Huh, Se Jin Park, Jong Hoon Ryu, Daniel J Whitcomb, Kwangwook Ch. Tau phosphorylation at serine 396 residue is required for hippocampal LTD. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 35. issue 12. 2015-06-15. PMID:25810511. |
here we probe the role of tau in ltd, finding that an ampa receptor internalization mechanism is impaired in tau ko mice, and that ltd causes specific phosphorylation at the serine 396 and 404 residues of tau. |
2015-06-15 |
2023-08-13 |
mouse |
| Eric C Miller, Peter J Teravskis, Benjamin W Dummer, Xiaohui Zhao, Richard L Huganir, Dezhi Lia. Tau phosphorylation and tau mislocalization mediate soluble Aβ oligomer-induced AMPA glutamate receptor signaling deficits. The European journal of neuroscience. vol 39. issue 7. 2014-12-18. PMID:24713000. |
tau phosphorylation and tau mislocalization mediate soluble aβ oligomer-induced ampa glutamate receptor signaling deficits. |
2014-12-18 |
2023-08-13 |
mouse |
| Amy M Pooler, Emma C Phillips, Dawn H W Lau, Wendy Noble, Diane P Hange. Physiological release of endogenous tau is stimulated by neuronal activity. EMBO reports. vol 14. issue 4. 2013-09-19. PMID:23412472. |
we show that stimulation of neuronal activity, or ampa receptor activation, induces tau release from healthy, mature cortical neurons. |
2013-09-19 |
2023-08-12 |
Not clear |
| Julie Allyson, Xiaoning Bi, Michel Baudry, Guy Massicott. Maintenance of synaptic stability requires calcium-independent phospholipase A₂ activity. Neural plasticity. vol 2012. 2013-04-03. PMID:22685677. |
in this line, the paper presents new data indicating that ipla₂γ deficiencies accentuate ampa receptor destabilization and tau phosphorylation, which suggests that this ipla₂ isoform should be considered as a potential target for the treatment of tau-related disorders. |
2013-04-03 |
2023-08-12 |
Not clear |
| Wagner Zago, Manuel Buttini, Thomas A Comery, Christopher Nishioka, Shyra J Gardai, Peter Seubert, Dora Games, Frédérique Bard, Dale Schenk, Gene G Kinne. Neutralization of soluble, synaptotoxic amyloid β species by antibodies is epitope specific. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 32. issue 8. 2012-04-16. PMID:22357853. |
in the current report, we demonstrated that 3d6 binds to soluble, synaptotoxic assemblies of aβ(1-42) and prevents multiple downstream functional consequences in rat hippocampal neurons including changes in glutamate ampa receptor trafficking, ad-type tau phosphorylation, and loss of dendritic spines. |
2012-04-16 |
2023-08-12 |
mouse |
| Yusuke Takatsuru, Masae Iino, Kohichi Tanaka, Seiji Ozaw. Contribution of glutamate transporter GLT-1 to removal of synaptically released glutamate at climbing fiber-Purkinje cell synapses. Neuroscience letters. vol 420. issue 1. 2009-06-02. PMID:17513050. |
in the presence of cyclothiazide (ctz) that attenuates the desensitization of ampa receptors, the decay time constant of cf-epscs (tau(w)) in glt-1(-/-) mice was slower than that in wt mice. |
2009-06-02 |
2023-08-12 |
mouse |
| Krisztina Szalisznyó, André Longtin, Leonard Male. Effect of synaptic plasticity on sensory coding and steady-state filtering properties in the electric sense. Bio Systems. vol 92. issue 1. 2008-06-03. PMID:18243518. |
via a mixture of ampa and nmda synapses, and a relatively smaller synaptic facilitation time constant (tau(f)) and larger tau(d) (with tau(f) smaller than tau(d) and tau(g)). |
2008-06-03 |
2023-08-12 |
Not clear |
| David Fernández de Sevilla, Marco Fuenzalida, Ana B Porto Pazos, Washington Buñ. Selective shunting of the NMDA EPSP component by the slow afterhyperpolarization in rat CA1 pyramidal neurons. Journal of neurophysiology. vol 97. issue 5. 2007-07-17. PMID:17329628. |
in contrast, the sahp did not modify the amplitude of the isolated epsp(ampa) but reduced tau(ampa) both in dendritic and somatic recordings. |
2007-07-17 |
2023-08-12 |
rat |
| Yusuke Takatsuru, Yukihiro Takayasu, Masae Iino, Osamu Nikkuni, Yuto Ueda, Kohichi Tanaka, Seiji Ozaw. Roles of glial glutamate transporters in shaping EPSCs at the climbing fiber-Purkinje cell synapses. Neuroscience research. vol 54. issue 2. 2006-03-29. PMID:16377014. |
in the presence of cyclothiazide (ctz), which attenuates the desensitization of ampa receptors, dhk prolonged the decay time constant (tau(w)) of cf-epscs in wt, indicating that glt-1 plays a partial role in the removal of glutamate. |
2006-03-29 |
2023-08-12 |
mouse |
| b' G Abdrachmanova, V Vlachov\\xc3\\xa1, L Vyklick\\xc3\\xb. Axotomy-induced change in the properties of (S)-alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionate receptor channels in rat motoneurons. Neuroscience. vol 99. issue 1. 2000-10-18. PMID:10924957.' |
deactivation of ampa receptors assessed from motoneuron excitatory postsynaptic currents at -70 mv was independent of postnatal age, with tau(fast)=0.88+/-0.35ms (a(fast)=78.2+/-11.8%) and tau(slow)=6. |
2000-10-18 |
2023-08-12 |
rat |
| G J Hathway, P C Emson, P P Humphrey, K M Kendric. Somatostatin potently stimulates in vivo striatal dopamine and gamma-aminobutyric acid release by a glutamate-dependent action. Journal of neurochemistry. vol 70. issue 4. 1998-04-23. PMID:9523593. |
an alpha-amino-3-hydroxy-5-methylisoxazole-4-propionic acid (ampa)/kainate antagonist, 6,7-dinitroquinoxaline-2,3-dione (100 microm), blocked srif-induced da and tau release and reduced that of asp, glu, and gaba. |
1998-04-23 |
2023-08-12 |
rat |