| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| T Barka, C Yagil, H Van der Noen, Y Nait. Induction of the synthesis of a specific protein in rat submandibular gland by isoproterenol. Laboratory investigation; a journal of technical methods and pathology. vol 54. issue 2. 1986-03-26. PMID:3945051. |
prolonged treatment of rats with the beta-adrenergic agonist, isoproterenol (ipr), produces hypertrophy and hyperplasia of the parotid and submandibular glands. |
1986-03-26 |
2023-08-11 |
rat |
| D Laky, S Constantinescu, G Filipescu, E Ratea, C Zean. The biology of the myocardium in chronic hypoxia. Note I. Myocardial lesions in experimental chronic heart failure. Morphologie et embryologie. vol 31. issue 4. 1986-02-17. PMID:2934627. |
the biology of the myocardium was studied in chronic heart failure, especially in the course of compensatory hypertrophy induced experimentally by partial stenosis of the aorta for 10 months and the administration of large isoproterenol doses for 7 months. |
1986-02-17 |
2023-08-11 |
Not clear |
| S Pickett, J C Le. Phosphorylation of ribosomal and ribosome-associated proteins in isoproterenol-induced cardiac hypertrophy. The International journal of biochemistry. vol 17. issue 9. 1986-01-15. PMID:2933283. |
cardiac hypertrophy in adult rabbits was induced by subcutaneous injection of isoproterenol. |
1986-01-15 |
2023-08-11 |
Not clear |
| D J Wells, M G Humphreys-Behe. Analysis of protein synthesis in rat salivary glands after chronic treatment with beta-receptor agonists and phosphodiesterase inhibitors. Biochemical pharmacology. vol 34. issue 24. 1986-01-14. PMID:2866770. |
chronic administration of the beta-adrenergic receptor agonist isoproterenol (5 mg/200 g animal for 10 days) resulted in rat parotid and submandibular gland hypertrophy, and it induced synthesis of a series of proline-rich proteins (prps) and glycoproteins. |
1986-01-14 |
2023-08-11 |
rat |
| Y Sugishita, K Iida, M Matsuda, R Ajisaka, T Ogawa, R Matsumoto, T Fujita, I Ito, T Yamaguch. [Apical hypertrophy and catecholamine]. Journal of cardiography. Supplement. issue 6. 1985-12-31. PMID:4067358. |
to investigate the contribution of catecholamine to apical hypertrophy (aph) which is a special type of hcm, 16 normal controls, 49 +/- 4 years old; 14 patients with aph, 55 +/- 6 years old; 18 patients with other types of hcm, 53 +/- 8 years old; and 12 patients with left ventricular hypertrophy due to valvular heart diseases (vhd), 49 +/- 10 years old, were studied during treadmill exercise tests, supine ergometer tests and intravenous infusions of isoproterenol (isp) (0.02 micrograms/kg/min for 5 min). |
1985-12-31 |
2023-08-11 |
Not clear |
| K Kumano, P A Khairalla. Adenylate cyclase activity during development and reversal of cardiac hypertrophy. Journal of molecular and cellular cardiology. vol 17. issue 6. 1985-09-25. PMID:3160864. |
it is concluded that in cardiac hypertrophy associated with different models of hypertension the decreased inotropic responsiveness to isoproterenol is associated with different biochemical defects in the beta-adrenergic receptor response coupling pathway, and that reversal in function occurs only when there is no apparent change in the catalytic subunit of the adenylate cyclase complex. |
1985-09-25 |
2023-08-11 |
rat |
| A R Hand, B H. Mitosis and hypertrophy of intercalated duct cells and endothelial cells in the isoproterenol-treated rat parotid gland. Journal of dental research. vol 64. issue 8. 1985-09-09. PMID:2410467. |
chronic treatment of rats with isoproterenol (ipr) induces mitosis and differentiation of intercalated duct cells, and mitosis and hypertrophy of endothelial cells of the microvasculature in the parotid gland. |
1985-09-09 |
2023-08-11 |
rat |
| P Simpso. Stimulation of hypertrophy of cultured neonatal rat heart cells through an alpha 1-adrenergic receptor and induction of beating through an alpha 1- and beta 1-adrenergic receptor interaction. Evidence for independent regulation of growth and beating. Circulation research. vol 56. issue 6. 1985-07-30. PMID:2988814. |
studies with other agonists (phenylephrine, methoxamine, clonidine, isoproterenol, dopamine) and antagonists (prazosin, terazosin, yohimbine, propranolol, betaxolol, ici 118,551) indicated that hypertrophy was mediated through an alpha 1-adrenergic receptor, whereas the induction of beating required both alpha 1- and beta 1-receptor activation. |
1985-07-30 |
2023-08-11 |
rat |
| C Thollon, J Aussedat, J Verdetti, P Krehe. [Absence, in the hypertrophied rat heart caused by aortocaval fistula, of several metabolic and electrophysiological changes seen in other models of hypertrophy]. Comptes rendus de l'Academie des sciences. Serie III, Sciences de la vie. vol 300. issue 16. 1985-07-19. PMID:3158383. |
in this work, we compared the electrophysiological and metabolic parameters of a volume overload model of cardiac hypertrophy (aorto-caval fistula) with those of two other models of hypertrophy (aortic stenosis and isoproterenol pretreatment). |
1985-07-19 |
2023-08-11 |
rat |
| H G Zimme. Correlation between haemodynamic and metabolic changes in three models of experimental cardiac hypertrophy. European heart journal. vol 5 Suppl F. 1985-06-19. PMID:6099802. |
three models of cardiac hypertrophy (aortic constriction, application of isoproterenol, daily injections of triiodothyronine) were characterized in haemodynamic and in metabolic terms. |
1985-06-19 |
2023-08-12 |
rat |
| A Rossi, J Aussedat, J Olivarès, A Ray, M Verdy. Pyrimidine nucleotide metabolism in cardiac hypertrophy. European heart journal. vol 5 Suppl F. 1985-06-19. PMID:6241890. |
cardiac hypertrophy was induced in rats either by isoprenaline treatment or by experimental abdominal aortic stenosis. |
1985-06-19 |
2023-08-12 |
rat |
| F Cluzeaud, J Perennec, E de Amoral, M Willemin, P Y Hat. Myocardial cell nucleus in cardiac overloading in the rat. European heart journal. vol 5 Suppl F. 1985-06-19. PMID:6241897. |
it decreased (80%) two days after isoproterenol overloading, as well as in heart hypertrophy of 6-9-months duration. |
1985-06-19 |
2023-08-12 |
rat |
| K M Middleton, A H Henderson, M J Lewi. Catecholamine-induced necrosis and hypertrophy. Hemodynamic factors. Advances in myocardiology. vol 6. 1985-06-14. PMID:3158047. |
significant degrees of both hypertrophy (whether measured as biventricular weight or biventricular/body weight ratio) and necrosis (measured by enzyme histochemical techniques on a standardized series of cryostat sections through the apex of each heart) occurred following 10 days' treatment with daily isoproterenol (0.5 or 5 mg/kg s.c.) or dobutamine (5 mg/kg s.c.) (n = 6-12). |
1985-06-14 |
2023-08-11 |
rat |
| F M Fouad, K Shimamatsu, M M Hanna, P A Khairallah, R C Taraz. Impaired inotropic responses to alpha-adrenergic stimulation in experimental left ventricular hypertrophy. Circulation. vol 71. issue 5. 1985-05-30. PMID:2985293. |
we have previously reported that left ventricular hypertrophy in two-kidney, one-clip renal hypertensive rats (2k-1c rhrs) was associated with diminished inotropic responsiveness to isoproterenol and glucagon, suggesting an alteration in the receptor-adenylate cyclase cascade. |
1985-05-30 |
2023-08-11 |
rat |
| K Iida, Y Sugishita, M Matsuda, T Yamaguchi, R Ajisaka, R Matsumoto, T Fujita, I It. [Pathophysiology of asymmetric septal hypertrophy with special reference to the response to isoproterenol administration]. Journal of cardiography. vol 14. issue 1. 1985-03-21. PMID:6240510. |
[pathophysiology of asymmetric septal hypertrophy with special reference to the response to isoproterenol administration]. |
1985-03-21 |
2023-08-12 |
Not clear |
| M Harri, T Dannenberg, R Oksanen-Rossi, E Hohtola, U Sundi. Related and unrelated changes in response to exercise and cold in rats: a reevaluation. Journal of applied physiology: respiratory, environmental and exercise physiology. vol 57. issue 5. 1985-02-27. PMID:6520042. |
these included cardiac hypertrophy, reduced urinary catecholamine excretion during and after stress situations, increased tail skin temperature response to isoproterenol, and a higher tail skin temperature during exposure to cold. |
1985-02-27 |
2023-08-12 |
rat |
| A Genovese, M Chiariello, M Bozzaotre, S Latte, W De Alfieri, M Condorell. Adrenergic activity as a modulating factor in the genesis of myocardial hypertrophy in the rat. Experimental and molecular pathology. vol 41. issue 3. 1985-02-01. PMID:6239790. |
data are presented indicating that both exogenous catecholamines (i.e., isoproterenol) and endogenous catecholamines (released from tyramine) are able to bring about a degree of myocardial hypertrophy associated with a significant increase in the ventricular concentrations of hydroxyproline, without affecting hemodynamic parameters (blood pressures and heart rate). |
1985-02-01 |
2023-08-12 |
rat |
| Q Tang, P B Taylo. Regression of isoproterenol-induced cardiac hypertrophy. Canadian journal of physiology and pharmacology. vol 62. issue 9. 1985-01-14. PMID:6208988. |
cardiac hypertrophy was induced in adult female wistar rats after 8 days of daily subcutaneous injections of isoproterenol (iso). |
1985-01-14 |
2023-08-12 |
rat |
| M G Humphreys-Behe. Isoprenaline-induced changes in rat parotid and submandibular glands are age- and dosage-dependent. The Biochemical journal. vol 221. issue 1. 1984-09-17. PMID:6466311. |
gland hypertrophy showed the same differential dosage kinetics, based on gland weight, between the two glands; however, hypertrophy could be accomplished at a lower dosage of isoprenaline than that used to induce proline-rich-protein biosynthesis. |
1984-09-17 |
2023-08-12 |
rat |
| P B Taylor, Q Tan. Development of isoproterenol-induced cardiac hypertrophy. Canadian journal of physiology and pharmacology. vol 62. issue 4. 1984-08-15. PMID:6203632. |
the development of cardiac hypertrophy was studied in adult female wistar rats following daily subcutaneous injections of isoproterenol (iso) (0.3 mg/kg body weight). |
1984-08-15 |
2023-08-12 |
rat |