All Relations between hypertrophic and matrix compartment

Publication Sentence Publish Date Extraction Date Species
Franck Simon, Daniele Bergeron, Sébastien Larochelle, Carlos A Lopez-Vallé, Hervé Genest, Alexis Armour, Véronique J Mouli. Enhanced secretion of TIMP-1 by human hypertrophic scar keratinocytes could contribute to fibrosis. Burns : journal of the International Society for Burn Injuries. vol 38. issue 3. 2012-05-23. PMID:22040932. hypertrophic scars are a pathological process characterized by an excessive deposition of extracellular matrix components. 2012-05-23 2023-08-12 human
Franck Simon, Daniele Bergeron, Sébastien Larochelle, Carlos A Lopez-Vallé, Hervé Genest, Alexis Armour, Véronique J Mouli. Enhanced secretion of TIMP-1 by human hypertrophic scar keratinocytes could contribute to fibrosis. Burns : journal of the International Society for Burn Injuries. vol 38. issue 3. 2012-05-23. PMID:22040932. the dermal matrix of these rhs was recreated using mesenchymal cells from three different origins (skin, wound and hypertrophic scar). 2012-05-23 2023-08-12 human
Dutmanee Seriwatanachai, Nateetip Krishnamra, Narattaphol Charoenphandh. Chondroregulatory action of prolactin on proliferation and differentiation of mouse chondrogenic ATDC5 cells in 3-dimensional micromass cultures. Biochemical and biophysical research communications. vol 420. issue 1. 2012-05-16. PMID:22405773. a recent investigation in lactating rats has provided evidence that the lactogenic hormone prolactin (prl) increases endochondral bone growth and bone elongation, presumably by accelerating apoptosis of hypertrophic chondrocytes in the growth plate and/or subsequent chondrogenic matrix mineralization. 2012-05-16 2023-08-12 mouse
Saurabh Dahiya, Shephali Bhatnagar, Sajedah M Hindi, Chunhui Jiang, Pradyut K Paul, Shihuan Kuang, Ashok Kuma. Elevated levels of active matrix metalloproteinase-9 cause hypertrophy in skeletal muscle of normal and dystrophin-deficient mdx mice. Human molecular genetics. vol 20. issue 22. 2012-05-15. PMID:21846793. elevated levels of active matrix metalloproteinase-9 cause hypertrophy in skeletal muscle of normal and dystrophin-deficient mdx mice. 2012-05-15 2023-08-12 mouse
G P Sidgwick, A Baya. Extracellular matrix molecules implicated in hypertrophic and keloid scarring. Journal of the European Academy of Dermatology and Venereology : JEADV. vol 26. issue 2. 2012-05-14. PMID:21838832. extracellular matrix molecules implicated in hypertrophic and keloid scarring. 2012-05-14 2023-08-12 Not clear
Norio Amizuka, Tomoka Hasegawa, Kimimitsu Oda, Paulo Henrique Luiz de Freitas, Kazuto Hoshi, Minqi Li, Hidehiro Ozaw. Histology of epiphyseal cartilage calcification and endochondral ossification. Frontiers in bioscience (Elite edition). vol 4. 2012-05-14. PMID:22202021. cartilage calcification is carried out by chondrocytes as they hypertrophy and begin to secrete matrix vesicles. 2012-05-14 2023-08-12 Not clear
Norio Amizuka, Tomoka Hasegawa, Kimimitsu Oda, Paulo Henrique Luiz de Freitas, Kazuto Hoshi, Minqi Li, Hidehiro Ozaw. Histology of epiphyseal cartilage calcification and endochondral ossification. Frontiers in bioscience (Elite edition). vol 4. 2012-05-14. PMID:22202021. hypertrophic chondrocytes secrete vesicles in the cartilaginous matrix of intercolumnar septae only, forming well-calcified longitudinal septae and poorly-calcified transverse partitions. 2012-05-14 2023-08-12 Not clear
P S Skagen, T Horn, H A Kruse, B Staergaard, M M Rapport, T Nicolaise. Osteochondritis dissecans (OCD), an endoplasmic reticulum storage disease?: a morphological and molecular study of OCD fragments. Scandinavian journal of medicine & science in sports. vol 21. issue 6. 2012-05-01. PMID:20561273. further, von kossa staining and tem showed empty lacunae close to mineralized "islands" in the cartilage and hypertrophic chondrocytes containing accumulated matrix proteins. 2012-05-01 2023-08-12 Not clear
Geert W Schmid-Schönbei. An emerging role of degrading proteinases in hypertension and the metabolic syndrome: autodigestion and receptor cleavage. Current hypertension reports. vol 14. issue 1. 2012-05-01. PMID:22081429. an emerging body of evidence suggests that human and experimental hypertensives may exhibit uncontrolled activity of proteinases, including the family of matrix metalloproteinases, recognized for their ability to restructure the extracellular matrix proteins and to play a role in hypertrophy. 2012-05-01 2023-08-12 human
Kirk R Hutchinson, Anuradha Guggilam, Mary J Cismowski, Maarten L Galantowicz, Thomas A West, James A Stewart, Xiaojin Zhang, Kevin C Lord, Pamela A Lucches. Temporal pattern of left ventricular structural and functional remodeling following reversal of volume overload heart failure. Journal of applied physiology (Bethesda, Md. : 1985). vol 111. issue 6. 2012-04-30. PMID:21885799. structural and functional changes were correlated to lv collagen content, extracellular matrix (ecm) proteins, and hypertrophic markers. 2012-04-30 2023-08-12 rat
Maurizio Mandala, George Oso. Physiological remodelling of the maternal uterine circulation during pregnancy. Basic & clinical pharmacology & toxicology. vol 110. issue 1. 2012-04-30. PMID:21902814. the process of maternal gestational uterine vascular remodelling involves a number of cellular processes and mechanisms, including trophoblast invasion, hyperplasia and hypertrophy, and changes in extracellular matrix composition. 2012-04-30 2023-08-12 rat
Anna Leychenko, Eugene Konorev, Mayumi Jijiwa, Michelle L Matte. Stretch-induced hypertrophy activates NFkB-mediated VEGF secretion in adult cardiomyocytes. PloS one. vol 6. issue 12. 2012-04-23. PMID:22174951. it is characterized by extracellular matrix remodeling and hypertrophic growth of adult cardiomyocytes. 2012-04-23 2023-08-12 rat
Ramesh N Reddy, Taylor L Knotts, Brian R Roberts, Jeffery D Molkentin, S Russ Price, Jennifer L Gooc. Calcineurin A-β is required for hypertrophy but not matrix expansion in the diabetic kidney. Journal of cellular and molecular medicine. vol 15. issue 2. 2012-04-16. PMID:19778355. calcineurin a-β is required for hypertrophy but not matrix expansion in the diabetic kidney. 2012-04-16 2023-08-12 mouse
Ramesh N Reddy, Taylor L Knotts, Brian R Roberts, Jeffery D Molkentin, S Russ Price, Jennifer L Gooc. Calcineurin A-β is required for hypertrophy but not matrix expansion in the diabetic kidney. Journal of cellular and molecular medicine. vol 15. issue 2. 2012-04-16. PMID:19778355. previously, we found that inhibition of calcineurin with cyclosporine reduced renal hypertrophy and blocked glomerular matrix expansion in the diabetic kidney. 2012-04-16 2023-08-12 mouse
Ramesh N Reddy, Taylor L Knotts, Brian R Roberts, Jeffery D Molkentin, S Russ Price, Jennifer L Gooc. Calcineurin A-β is required for hypertrophy but not matrix expansion in the diabetic kidney. Journal of cellular and molecular medicine. vol 15. issue 2. 2012-04-16. PMID:19778355. therefore, we examined the role of calcineurin β in diabetic renal hypertrophy and glomerular matrix expansion. 2012-04-16 2023-08-12 mouse
Ramesh N Reddy, Taylor L Knotts, Brian R Roberts, Jeffery D Molkentin, S Russ Price, Jennifer L Gooc. Calcineurin A-β is required for hypertrophy but not matrix expansion in the diabetic kidney. Journal of cellular and molecular medicine. vol 15. issue 2. 2012-04-16. PMID:19778355. type i diabetes was induced in wild-type and β(-/-) mice and then renal function, extracellular matrix expansion and hypertrophy were evaluated. 2012-04-16 2023-08-12 mouse
Ramesh N Reddy, Taylor L Knotts, Brian R Roberts, Jeffery D Molkentin, S Russ Price, Jennifer L Gooc. Calcineurin A-β is required for hypertrophy but not matrix expansion in the diabetic kidney. Journal of cellular and molecular medicine. vol 15. issue 2. 2012-04-16. PMID:19778355. in conclusion, loss of the β isoform of calcineurin is sufficient to reproduce beneficial aspects of cyclosporine on diabetic renal hypertrophy but not matrix expansion. 2012-04-16 2023-08-12 mouse
Ramesh N Reddy, Taylor L Knotts, Brian R Roberts, Jeffery D Molkentin, S Russ Price, Jennifer L Gooc. Calcineurin A-β is required for hypertrophy but not matrix expansion in the diabetic kidney. Journal of cellular and molecular medicine. vol 15. issue 2. 2012-04-16. PMID:19778355. therefore, while multiple signals appear to regulate matrix, calcineurin β appears to be a central mechanism involved in organ hypertrophy. 2012-04-16 2023-08-12 mouse
Maria C Guido, Carolina F Clemente, Ana I Moretti, Hermes V Barbeiro, Victor Debbas, Elia G Caldini, Kleber G Franchini, Francisco G Sorian. Small interfering RNA targeting focal adhesion kinase prevents cardiac dysfunction in endotoxemia. Shock (Augusta, Ga.). vol 37. issue 1. 2012-04-16. PMID:21921830. the hearts of lipopolysaccharide-injected rats showed collagen deposition, increased matrix metalloproteinase 2 activity, and myocyte hypertrophy, as well as reduced 24-h +dp/dt and -dp/dt, together with hypotension, increased left ventricular end-diastolic pressure, and elevated levels of fak (phosphorylated and unphosphorylated). 2012-04-16 2023-08-12 rat
Hak Joo Lee, Meenalakshmi M Mariappan, Denis Feliers, Rita C Cavaglieri, Kavithalakshmi Sataranatarajan, Hanna E Abboud, Goutam Ghosh Choudhury, Balakuntalam S Kasinat. Hydrogen sulfide inhibits high glucose-induced matrix protein synthesis by activating AMP-activated protein kinase in renal epithelial cells. The Journal of biological chemistry. vol 287. issue 7. 2012-04-16. PMID:22158625. high glucose stimulation of global protein synthesis, cellular hypertrophy, and matrix laminin and type iv collagen content was inhibited by sodium hydrosulfide (nahs), an h(2)s donor. 2012-04-16 2023-08-12 mouse