All Relations between hypertrophic and matrix compartment

Reference Sentence Publish Date Extraction Date Species
A J Hayes, S MacPherson, H Morrison, G Dowthwaite, C W Arche. The development of articular cartilage: evidence for an appositional growth mechanism. Anatomy and embryology vol 203 issue 6 2001 11453164 it is well-established that cartilage grows by a combination of matrix secretion, cell hypertrophy and cell proliferation. 2001-12-04 2023-01-24 Not clear
T Walther, A Schubert, V Falk, C Binner, A Kanev, S Bleiziffer, C Walther, N Doll, R Autschbach, F W Moh. Regression of left ventricular hypertrophy after surgical therapy for aortic stenosis is associated with changes in extracellular matrix gene expression. Circulation vol 104 issue 12 Suppl 1 2001 11568030 regression of left ventricular hypertrophy after surgical therapy for aortic stenosis is associated with changes in extracellular matrix gene expression. 2001-10-04 2023-01-24 Not clear
V S Mujumdar, L M Smiley, S C Tyag. Activation of matrix metalloproteinase dilates and decreases cardiac tensile strength. International journal of cardiology vol 79 issue 2-3 2001 11461752 previous studies demonstrated that transition from compensatory pressure overload hypertrophy to decompensatory volume overload heart failure is associated with decreased cardiac tensile strength and activation of matrix metalloproteinase (mmp) in spontaneously hypertensive rat (shr). 2001-09-27 2023-01-24 rat
V S Mujumdar, L M Smiley, S C Tyag. Activation of matrix metalloproteinase dilates and decreases cardiac tensile strength. International journal of cardiology vol 79 issue 2-3 2001 11461752 to test the hypothesis that in the absence of nitric oxide activation of mmp during cardiac failure causes disruption in the organization of extracellular matrix (ecm) and leads to decrease systolic and diastolic cardiac tensile strength, we employed shr of 24--32 weeks, which demonstrates significant cardiac hypertrophy and fibrosis. 2001-09-27 2023-01-24 rat
V S Mujumdar, L M Smiley, S C Tyag. Activation of matrix metalloproteinase dilates and decreases cardiac tensile strength. International journal of cardiology vol 79 issue 2-3 2001 11461752 to determine whether cardiac hypertrophy is associated with increased elastinolytic matrix metalloproteinase-2 (mmp-2) activity; quantitative elastin-zymography was performed on cardiac tissue homogenates. 2001-09-27 2023-01-24 rat
M A Ross. Connective tissue skeleton in the normal left ventricle and in hypertensive left ventricular hypertrophy and chronic chagasic myocarditis. Medical science monitor : international medical journal of experimental and clinical research vol 7 issue 4 2001 11433216 this report reviews the connective tissue matrix of the normal human myocardial tissue and the pathological myocardial fibrosis in left ventricular hypertrophy due to chronic arterial hypertension in humans and in human chronic chagasic myocarditis. 2001-09-20 2023-01-24 human
K Hanada, L A Solchaga, A I Caplan, T M Hering, V M Goldberg, J U Yoo, B Johnston. BMP-2 induction and TGF-beta 1 modulation of rat periosteal cell chondrogenesis. Journal of cellular biochemistry vol 81 issue 2 2001 11241668 after 14 days in culture, bmp-2-treated aggregates consisted of many hypertrophic chondrocytes within a metachromatic matrix, which was immunoreactive with anti-type ii and type x collagen antibodies. 2001-08-02 2023-01-24 rat
K Hanada, L A Solchaga, A I Caplan, T M Hering, V M Goldberg, J U Yoo, B Johnston. BMP-2 induction and TGF-beta 1 modulation of rat periosteal cell chondrogenesis. Journal of cellular biochemistry vol 81 issue 2 2001 11241668 in contrast, at 14 days, tgf-beta 1 + bmp-2-treated aggregates did not contain any morphologically identifiable hypertrophic chondrocytes and their abundant extracellular matrix was not immunoreactive to the anti-type x collagen antibody. 2001-08-02 2023-01-24 rat
K Stewart, P Wilson, H Wilson, A Minto, N Haites, P Whiting, A Macleod, P Brow. Thy1 glomerulonephritis induced in young lewis rats accelerates age-related glomerulosclerosis. Nephron vol 88 issue 1 2001 11340352 in this regard, glomerular hypercellularity, rather than hypertrophy or matrix increase, was the most consistent histological index of later accelerated disease. 2001-08-02 2023-01-24 rat
Y Mizuno-Horikawa, S Mizuno, S Tamura, T Kurosaw. Advanced glomerulosclerosis is reversible in nephrotic mice. Biochemical and biophysical research communications vol 284 issue 3 2001 11396960 when ureter ligation was applied to our model, glomerulosclerosis (characterized by myofibroblast hyperplasia and over-accumulated matrix protein) weakened in conjunction with suppressed glomerular hypertrophy. 2001-08-02 2023-01-24 mouse
A D Hodgkinson, B A Millward, A G Demain. Polymorphisms of the glucose transporter (GLUT1) gene are associated with diabetic nephropathy. Kidney international vol 59 issue 3 2001 11231353 glucose transporter (glut1) activity has been implicated in renal hypertrophy and extracellular matrix formation in mesangial cells. 2001-07-19 2023-01-24 Not clear
I Katsuragi, T Okeda, H Yoshimatsu, N Utsunomiya, K Ina, T Sakat. Transplantation of normal islets into the portal vein of Otsuka Long Evans Tokushima Fatty rats prevents diabetic progression. Experimental biology and medicine (Maywood, N.J.) vol 226 issue 7 2001 11444104 immunohistochemical studies demonstrated that hypertrophic and fibrotic changes in pancreatic islets, together with mesangial proliferation of the glomerular matrix, an indicator for diabetic nephropathy, were attenuated predominantly in the it group at the late phase after transplantation compared with those in the corresponding phase of the so group. 2001-07-19 2023-01-24 rat
X Zhen, L Wei, Q Wu, Y Zhang, Q Che. Mitogen-activated protein kinase p38 mediates regulation of chondrocyte differentiation by parathyroid hormone. The Journal of biological chemistry vol 276 issue 7 2001 11098049 inhibition of p38 mapk in hypertrophic chondrocytes by either pth, sb303580, or both together leads to a decrease of hypertrophic marker type x collagen mrna and an increase of the expression of prehypertrophic marker cartilage matrix protein. 2001-06-28 2023-01-24 Not clear
F B Niessen, M P Andriessen, J Schalkwijk, L Visser, W Timen. Keratinocyte-derived growth factors play a role in the formation of hypertrophic scars. The Journal of pathology vol 194 issue 2 2001 11400150 in predisposed individuals, wound healing can lead to hypertrophic scar or keloid formation, characterized by an overabundant extracellular matrix. 2001-06-21 2023-01-24 Not clear
F B Niessen, M P Andriessen, J Schalkwijk, L Visser, W Timen. Keratinocyte-derived growth factors play a role in the formation of hypertrophic scars. The Journal of pathology vol 194 issue 2 2001 11400150 it is hypothesized that impaired production of keratinocyte-derived growth factors, such as il-1alpha, leads to a decrease in the catabolism of the dermal matrix, whereas augmented epidermal pdgf production leads to increased formation of the dermal matrix in hypertrophic scars. 2001-06-21 2023-01-24 Not clear
D Grimm, M Huber, H C Jabusch, M Shakibaei, S Fredersdorf, M Paul, G A Riegger, E P Krome. Extracellular matrix proteins in cardiac fibroblasts derived from rat hearts with chronic pressure overload: effects of beta-receptor blockade. Journal of molecular and cellular cardiology vol 33 issue 3 2001 11181017 left ventricular hypertrophy (lvh) is accompanied by progressive accumulations of extracellular matrix proteins. 2001-06-14 2023-01-24 rat
N A Trueblood, Z Xie, C Communal, F Sam, S Ngoy, L Liaw, A W Jenkins, J Wang, D B Sawyer, O H Bing, C S Apstein, W S Colucci, K Sing. Exaggerated left ventricular dilation and reduced collagen deposition after myocardial infarction in mice lacking osteopontin. Circulation research vol 88 issue 10 2001 11375279 osteopontin (opn), an extracellular matrix protein, is expressed in the myocardium with hypertrophy and failure. 2001-06-14 2023-01-24 mouse
M Alini, P J Roughle. Changes in leucine-rich repeat proteoglycans during maturation of the bovine growth plate. Matrix biology : journal of the International Society for Matrix Biology vol 19 issue 8 2001 11223340 at the message level, the expression of decorin shows a similar trend to that of protein abundance in the extracellular matrix, with its expression dropping markedly in the proliferative and hypertrophic zones. 2001-06-07 2023-01-24 cattle
P Chamiot-Clerc, J F Renaud, M E Safa. Pulse pressure, aortic reactivity, and endothelium dysfunction in old hypertensive rats. Hypertension (Dallas, Tex. : 1979) vol 37 issue 2 2001 11230291 in old hypertensive rats, (1) increased kcl reactivity is endothelium independent but influenced by the map-dependent aortic hypertrophy with resulting increased vascular smooth muscle reactivity, whereas (2) increased ne reactivity is endothelium dependent in association with increased pp, altered endothelial function, and extracellular matrix, with resulting enhanced intrinsic arterial stiffness. 2001-05-17 2023-01-24 rat
K Kusafuka, Y Hiraki, C Shukunami, A Yamaguchi, T Kayano, T Takemur. Cartilage-specific matrix protein chondromodulin-I is associated with chondroid formation in salivary pleomorphic adenomas: immunohistochemical analysis. The American journal of pathology vol 158 issue 4 2001 11290564 in the growth plates of the long bones, chm-i was shown to be expressed in mature to upper hypertrophic chondrocytes, and to be deposited in the cartilage matrix. 2001-05-03 2023-01-24 Not clear