| Reference |
Sentence |
Publish Date |
Extraction Date |
Species |
| M Crabos, M Roth, A W Hahn, P Ern. Characterization of angiotensin II receptors in cultured adult rat cardiac fibroblasts. Coupling to signaling systems and gene expression. The Journal of clinical investigation vol 93 issue 6 1994 8200970 |
cardiac hypertrophy is largely due to cardiac fibroblast growth and increased synthesis of extracellular matrix. |
1994-07-07 |
2023-01-24 |
rat |
| H Hattori, M Kawai, K Yasue, H Mizutani, M Kobayashi, T Kaneda, T Hoshin. A correlative immuno-light and electron microscopic study on the type I collagen in the bone morphogenetic protein-induced cartilage. Journal of electron microscopy vol 42 issue 6 1994 8176334 |
the extracellular matrix components around the hypertrophic chondrocytes in the induced tissue were examined by electron microscopy and immunohistochemistry for type i collagen. |
1994-06-07 |
2023-01-24 |
mouse |
| H Hattori, M Kawai, K Yasue, H Mizutani, M Kobayashi, T Kaneda, T Hoshin. A correlative immuno-light and electron microscopic study on the type I collagen in the bone morphogenetic protein-induced cartilage. Journal of electron microscopy vol 42 issue 6 1994 8176334 |
at 14 days after implantation, chondrocytes became hypertrophic with calcifying matrix around them. |
1994-06-07 |
2023-01-24 |
mouse |
| H Hattori, M Kawai, K Yasue, H Mizutani, M Kobayashi, T Kaneda, T Hoshin. A correlative immuno-light and electron microscopic study on the type I collagen in the bone morphogenetic protein-induced cartilage. Journal of electron microscopy vol 42 issue 6 1994 8176334 |
these results suggest that some of the bmp-induced hypertrophic chondrocytes maintain their activity and produce type i collagen-rich matrix before their lacunae are eroded. |
1994-06-07 |
2023-01-24 |
mouse |
| V Pelouch, I M Dixon, L Golfman, R E Beamish, N S Dhall. Role of extracellular matrix proteins in heart function. Molecular and cellular biochemistry vol 129 issue 2 1994 8177233 |
since these cells are the source of many components of the cardiac extracellular matrix, and because changes in cardiac extracellular matrix are suspected of contributing to the genesis of cardiovascular complications in disease states such as diabetes, hypertension, cardiac hypertrophy and congestive heart failure, interest in the mechanisms of activation of fibroblasts and endothelial cells has led to progress in understanding these processes. |
1994-06-07 |
2023-01-24 |
Not clear |
| V Pelouch, I M Dixon, L Golfman, R E Beamish, N S Dhall. Role of extracellular matrix proteins in heart function. Molecular and cellular biochemistry vol 129 issue 2 1994 8177233 |
recent work provides evidence for the role of the renin-angiotensin-aldosterone system in the pathogenesis of abnormal deposition of extracellular matrix in the cardiac interstitium during the development of inappropriate cardiac hypertrophy and failure. |
1994-06-07 |
2023-01-24 |
Not clear |
| R J Wardale, V C Duanc. Characterisation of articular and growth plate cartilage collagens in porcine osteochondrosis. Journal of cell science vol 107 ( Pt 1) issue 1994 8175922 |
type x collagen was produced in the matrix of the calcifying region of osteochondrotic articular cartilage by small groups of hypertrophic chondrocytes, but was not detected in normal articular cartilage. |
1994-06-06 |
2023-01-24 |
Not clear |
| S Moskalewski, A Hyc, T Grzela, J Malejczy. Differences in cartilage formed intramuscularly or in joint surface defects by syngeneic rat chondrocytes isolated from the articular-epiphyseal cartilage complex. Cell transplantation vol 2 issue 6 1994 8167932 |
in cartilage nodules produced intramuscularly, chondrocyte hypertrophy and matrix calcification were observed after 2 wk. |
1994-05-27 |
2023-01-24 |
rat |
| S Moskalewski, A Hyc, T Grzela, J Malejczy. Differences in cartilage formed intramuscularly or in joint surface defects by syngeneic rat chondrocytes isolated from the articular-epiphyseal cartilage complex. Cell transplantation vol 2 issue 6 1994 8167932 |
thus, either intraarticular environment exerted an inhibitory effect on chondrocyte hypertrophy and matrix calcification or articular chondrocytes present among transplanted cells accumulated close to the joint lumen and reconstructed normal articular cartilage. |
1994-05-27 |
2023-01-24 |
rat |
| S Katayama, M Abe, M Inaba, A Itabashi, J Ishi. Effects of captopril or nitrendipine on left ventricular collagen or laminin B2 gene expression. Japanese heart journal vol 34 issue 6 1994 8164345 |
these results suggest that angiotensin converting enzyme inhibitors prevent and/or cause regression of cardiac hypertrophy by inhibiting the gene expression of extracellular matrix, and that angiotensin ii may have a pivotal role in cardiac hypertrophy. |
1994-05-20 |
2023-01-24 |
rat |
| M E Safar, B I L\\xc3\\xa9v. Mechanical function and histological structure of the arterial wall. The response to antihypertensive treatment. Drugs vol 46 Suppl 2 issue 1994 7512485 |
firstly, during arterial hypertension, increases in arterial stiffness, associated with medial hypertrophy and/or hyperplasia and alterations in extracellular matrix, are always observed. |
1994-05-19 |
2023-01-24 |
rat |
| T Kirsch, R E Wuthie. Stimulation of calcification of growth plate cartilage matrix vesicles by binding to type II and X collagens. The Journal of biological chemistry vol 269 issue 15 1994 8157677 |
matrix vesicles (mv), microstructures which rapidly accumulate ca2+ and induce mineral formation in vitro, are linked to type ii and x collagens and proteoglycans in the hypertrophic cartilage. |
1994-05-19 |
2023-01-24 |
chicken |
| C W Kische. Contributions of electron microscopy to the study of the hypertrophic scar and related lesions. Scanning microscopy vol 7 issue 3 1994 8146619 |
the first sem studies demonstrated a homogeneous, dense dermal matrix which supported the conclusion that the hypertrophic scar and keloid reflected excess collagen. |
1994-04-29 |
2023-01-24 |
Not clear |
| E B Hunziker, J Wagner, J Zap. Differential effects of insulin-like growth factor I and growth hormone on developmental stages of rat growth plate chondrocytes in vivo. The Journal of clinical investigation vol 93 issue 3 1994 8132746 |
proliferating cell cycle time decreased from 11 to 4.5 and 3 d, and duration of the hypertrophic phase decreased from 6 to 4 and 2.8 d. average matrix volume per cell at each differentiation stage was similar for normal, hormone-treated, and untreated hypophysectomized groups. |
1994-04-21 |
2023-01-24 |
rat |
| B Zimmerman. Occurrence of osteoblast necroses during ossification of long bone cortices in mouse fetuses. Cell and tissue research vol 275 issue 2 1994 8111841 |
initially, cell necroses in the perichondrium occurred in the region surrounding the hypertrophic cartilage and the matrix of which showed spots of endochondral mineralization. |
1994-03-28 |
2023-01-24 |
rat |
| C Gentili, R Doliana, P Bet, G Campanile, A Colombatti, F D Cancedda, R Cancedd. Ovotransferrin and ovotransferrin receptor expression during chondrogenesis and endochondral bone formation in developing chick embryo. The Journal of cell biology vol 124 issue 4 1994 8106555 |
ovotransferrin was expressed by in vitro differentiating chondrocytes in the early phase of the culture and, at least when culture conditions allowed extracellular matrix assembly, also by hypertrophic chondrocytes and derived osteoblast-like cells. |
1994-03-24 |
2023-01-24 |
chicken |
| F J Pardo Mind\\xc3\\xa1n, A Paniz. Alterations in the extracellular matrix of the myocardium in essential hypertension. European heart journal vol 14 Suppl J issue 1994 8281955 |
hypertension produces myocyte hypertrophy and increases the extracellular matrix. |
1994-02-14 |
2023-01-24 |
Not clear |
| J Cos\\xc3\\xadn Aguilar, A Hernandiz Martinez, F Andres Conejo. Mechanisms of ventricular arrhythmias in the presence of pathological hypertrophy. European heart journal vol 14 Suppl J issue 1994 8281966 |
hypertension, electrical changes in the hypertrophic myocyte, the growth of the collagen matrix and ischaemia (even in the absence of macro-angiographic injuries) are factors present in this group of patients that contribute to the appearance of arrhythmias. |
1994-02-14 |
2023-01-24 |
Not clear |
| M E Choi, E G Kim, Q Huang, B J Ballerman. Rat mesangial cell hypertrophy in response to transforming growth factor-beta 1. Kidney international vol 44 issue 5 1994 8264154 |
central features of progressive glomerular sclerosis are initial glomerular hypertrophy and subsequent accumulation of extracellular matrix proteins. |
1994-01-26 |
2023-01-24 |
rat |
| M E Choi, E G Kim, Q Huang, B J Ballerman. Rat mesangial cell hypertrophy in response to transforming growth factor-beta 1. Kidney international vol 44 issue 5 1994 8264154 |
these findings demonstrate that rat mesangial cells express key components of the tgf-beta system and raise the intriguing possibility that in the glomerular mesangium, tgf-beta 1 may not only induce extracellular matrix synthesis, but may also participate in the process of glomerular hypertrophy in response to injury. |
1994-01-26 |
2023-01-24 |
rat |