| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Cristina Ribeiro Barros Cardoso, Maria Aparecida Souza, Eloísa Amália Vieira Ferro, Sílvio Favoreto, Janethe Deolina Oliviera Pen. Influence of topical administration of n-3 and n-6 essential and n-9 nonessential fatty acids on the healing of cutaneous wounds. Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society. vol 12. issue 2. 2004-09-03. PMID:15086775. |
furthermore, we showed that n-3 fatty acid induced a peak in nitric oxide at 3 hours postsurgery and an intense deposition of extracellular matrix after 5 days of treatment. |
2004-09-03 |
2023-08-12 |
Not clear |
| J-M Daye. [Biological modulation of IL-1 activity: role and development of its natural inhibitor IL-1Ra]. Reumatismo. vol 56. issue 1 Suppl 1. 2004-08-30. PMID:15201935. |
il-1 and tnf-alpha are potent inducers of matrix metalloproteinases (mmp), eicosanoids, nitric oxide oxydase (inos), receptor activator of nf-kb ligand (rankl), products involved in the destruction of the extracellular matrix, the cartilage and in bone resorption. |
2004-08-30 |
2023-08-12 |
Not clear |
| Farshid Guilak, Beverley Fermor, Francis J Keefe, Virginia B Kraus, Steven A Olson, David S Pisetsky, Lori A Setton, J Brice Weinber. The role of biomechanics and inflammation in cartilage injury and repair. Clinical orthopaedics and related research. issue 423. 2004-08-26. PMID:15232421. |
in vitro explant studies confirm that mechanical load is a potent regulator of matrix metabolism, cell viability, and the production of proinflammatory mediators such as nitric oxide and prostaglandin e2. |
2004-08-26 |
2023-08-12 |
Not clear |
| Raymond A Swanson, Weihai Ying, Tiina M Kauppine. Astrocyte influences on ischemic neuronal death. Current molecular medicine. vol 4. issue 2. 2004-08-10. PMID:15032713. |
reactive astrocytes secrete nitric oxide, tnfalpha, matrix metalloproteinases, and other factors that can contribute to delayed neuronal death, and facilitate brain edema via aquaporin-4 channels localized to the astrocyte endfoot-endothelial interface. |
2004-08-10 |
2023-08-12 |
Not clear |
| P S Monraats, W R P Agema, J W Jukem. Genetic predictive factors in restenosis. Pathologie-biologie. vol 52. issue 4. 2004-08-05. PMID:15145131. |
the single nucleotide polymorphisms (snps) described in the literature so far involve; the renin-angiotensin system, platelet aggregation, the inflammatory response, matrix metalloproteinases, smooth muscle cell proliferation, lipids and oxidative stress and nitric oxide. |
2004-08-05 |
2023-08-12 |
Not clear |
| H Nakamura, A Shibakawa, M Tanaka, T Kato, K Nishiok. Effects of glucosamine hydrochloride on the production of prostaglandin E2, nitric oxide and metalloproteases by chondrocytes and synoviocytes in osteoarthritis. Clinical and experimental rheumatology. vol 22. issue 3. 2004-08-04. PMID:15144122. |
to determine the response of glucosamine hydrochloride on chondrocytes and synoviocytes in terms of prostaglandin e2 (pge2), nitric oxide (no) and matrix metalloproteases (mmps). |
2004-08-04 |
2023-08-12 |
Not clear |
| Ashok Biyani, Gunnar B J Andersso. Low back pain: pathophysiology and management. The Journal of the American Academy of Orthopaedic Surgeons. vol 12. issue 2. 2004-07-13. PMID:15089084. |
cytokines such as matrix metalloproteinases, phospholipase a2, nitric oxide, and tumor necrosis factor-alpha are thought to contribute to the development of low back pain. |
2004-07-13 |
2023-08-12 |
Not clear |
| Chinnaswamy Tiruppathi, Tabassum Naqvi, Yubin Wu, Stephen M Vogel, Richard D Minshall, Asrar B Mali. Albumin mediates the transcytosis of myeloperoxidase by means of caveolae in endothelial cells. Proceedings of the National Academy of Sciences of the United States of America. vol 101. issue 20. 2004-07-06. PMID:15136724. |
myeloperoxidase (mpo), the phagocyte hemoprotein involved in neutrophil host defense and consuming nitric oxide (*no), induces the nitration of extracellular matrix proteins and tissue remodeling subsequent to its transcytosis across the endothelial barrier. |
2004-07-06 |
2023-08-12 |
human |
| Hsuan-Hsu Chen, Danny Ling Wan. Nitric oxide inhibits matrix metalloproteinase-2 expression via the induction of activating transcription factor 3 in endothelial cells. Molecular pharmacology. vol 65. issue 5. 2004-05-28. PMID:15102941. |
nitric oxide inhibits matrix metalloproteinase-2 expression via the induction of activating transcription factor 3 in endothelial cells. |
2004-05-28 |
2023-08-12 |
Not clear |
| Hsuan-Hsu Chen, Danny Ling Wan. Nitric oxide inhibits matrix metalloproteinase-2 expression via the induction of activating transcription factor 3 in endothelial cells. Molecular pharmacology. vol 65. issue 5. 2004-05-28. PMID:15102941. |
nitric oxide (no) has been shown to inhibit migration of cells in which various matrix metalloproteinases (mmps) are involved. |
2004-05-28 |
2023-08-12 |
Not clear |
| Patricia G Phillips, Linda M Birnb. Nitric oxide modulates caveolin-1 and matrix metalloproteinase-9 expression and distribution at the endothelial cell/tumor cell interface. American journal of physiology. Lung cellular and molecular physiology. vol 286. issue 5. 2004-05-17. PMID:15064242. |
nitric oxide modulates caveolin-1 and matrix metalloproteinase-9 expression and distribution at the endothelial cell/tumor cell interface. |
2004-05-17 |
2023-08-12 |
human |
| Thomas Pufe, Viola Harde, Wolf Petersen, Mary B Goldring, Bernhard Tillmann, Rolf Mentlei. Vascular endothelial growth factor (VEGF) induces matrix metalloproteinase expression in immortalized chondrocytes. The Journal of pathology. vol 202. issue 3. 2004-04-16. PMID:14991903. |
activated matrix metalloproteinase (mmp)-1, mmp-3, mmp-13, tissue inhibitor of metalloproteinases (timp)-1, timp-2, interleukin (il)-1beta, il-6, and tumour necrosis factor-alpha (tnf-alpha) were measured in culture supernatants by enzyme-linked immunosorbent assays, nitric oxide with the griess reagent, and cell proliferation by [3h]thymidine incorporation. |
2004-04-16 |
2023-08-12 |
human |
| João B Calixto, Maria M Campos, Michel F Otuki, Adair R Santo. Anti-inflammatory compounds of plant origin. Part II. modulation of pro-inflammatory cytokines, chemokines and adhesion molecules. Planta medica. vol 70. issue 2. 2004-04-06. PMID:14994184. |
ap-1:activator protein-1 ccr1:chemokine receptor 1 cinc-1:cytokine-induced neutrophil chemoattractant 1 cox:cyclooxygenase egcg:(-)-epigallocatechin gallate elam-1:endothelial-leukocyte adhesion molecule-1 erk:extracellular signal-regulated kinase gro:growth-related oncogene huvec:human umbilical vein endothelial cells icam-1:intercellular adhesion molecule-1 ifn:interferon il:interleukin inos:inducible nitric oxide synthase ira:the natural interleukin receptor activation jak:janus kinase jnk:c-jun nh2-terminal kinase lps:lipopolysaccharide mapk:mitogen-activated protein kinases mcp:monocyte chemotactic protein mhc:major histocompatibility complex mip:macrophage inflammatory protein mmp:matrix metalloproteinases mpo:myeloperoxidase nf-kappabnuclear factor kappa b no:nitric oxide paf:platelet aggregation factor pgee:prostaglandin pk:protein kinase pma/tpa:phorbol myristate acetate rantes:regulated upon activation normal t-cell expressed and secreted tgf-beta:transforming growth factor-beta tnfalpha:tumour necrosis factor vcam-1:vascular cell adhesion molecule-1 |
2004-04-06 |
2023-08-12 |
human |
| J M Daye. Evidence for the biological modulation of IL-1 activity: the role of IL-1Ra. Clinical and experimental rheumatology. vol 20. issue 5 Suppl 27. 2004-03-25. PMID:14989424. |
il-1 and tnfalpha are potent inducers of matrix metalloproteinases (mmps), eicosanoids, nitric oxide oxydase (inos), receptor activator of nf-kappab ligand (rankl), products involved in the destruction of the extracellular matrix, the cartilage and in bone resorption. |
2004-03-25 |
2023-08-12 |
Not clear |
| E A G Demoncheaux, P J Foster, C D Borland, A P L Smith, T W Higenbottam, M B Davie. Determination of trace concentrations of dissolved nitric oxide in a biological buffer. The Analyst. vol 128. issue 10. 2004-02-24. PMID:14667166. |
a new real-time method for measuring a trace concentration of nitric oxide (no) in a complex matrix routinely used in pharmacological studies of its bioactivity is described. |
2004-02-24 |
2023-08-12 |
Not clear |
| Mari Hoshiyama, Bing Li, Jian Yao, Takashi Harada, Tetsuo Morioka, Takashi Oit. Effect of high glucose on nitric oxide production and endothelial nitric oxide synthase protein expression in human glomerular endothelial cells. Nephron. Experimental nephrology. vol 95. issue 2. 2004-02-20. PMID:14610325. |
nitric oxide (no), a potent endothelium-derived vasodilator, has been suggested to participate in the regulation of renal blood flow, glomerular filtration rate, and mesangial matrix accumulation. |
2004-02-20 |
2023-08-12 |
human |
| Ru Liu, Frédéric Lioté, David M Rose, Denise Merz, Robert Terkeltau. Proline-rich tyrosine kinase 2 and Src kinase signaling transduce monosodium urate crystal-induced nitric oxide production and matrix metalloproteinase 3 expression in chondrocytes. Arthritis and rheumatism. vol 50. issue 1. 2004-02-17. PMID:14730623. |
proline-rich tyrosine kinase 2 and src kinase signaling transduce monosodium urate crystal-induced nitric oxide production and matrix metalloproteinase 3 expression in chondrocytes. |
2004-02-17 |
2023-08-12 |
Not clear |
| Sang-Jin Shin, Beverley Fermor, J Brice Weinberg, David S Pisetsky, Farshid Guila. Regulation of matrix turnover in meniscal explants: role of mechanical stress, interleukin-1, and nitric oxide. Journal of applied physiology (Bethesda, Md. : 1985). vol 95. issue 1. 2004-02-02. PMID:12665533. |
regulation of matrix turnover in meniscal explants: role of mechanical stress, interleukin-1, and nitric oxide. |
2004-02-02 |
2023-08-12 |
Not clear |
| Sang-Jin Shin, Beverley Fermor, J Brice Weinberg, David S Pisetsky, Farshid Guila. Regulation of matrix turnover in meniscal explants: role of mechanical stress, interleukin-1, and nitric oxide. Journal of applied physiology (Bethesda, Md. : 1985). vol 95. issue 1. 2004-02-02. PMID:12665533. |
the goal of this study was to examine the influence of mechanical stress on matrix turnover in the meniscus in the presence of interleukin-1 (il-1) and to determine the role of nitric oxide (no) in these processes. |
2004-02-02 |
2023-08-12 |
Not clear |
| Hiroshi Maeda, Jun Fang, Takao Inutsuka, Yasunori Kitamot. Vascular permeability enhancement in solid tumor: various factors, mechanisms involved and its implications. International immunopharmacology. vol 3. issue 3. 2003-12-17. PMID:12639809. |
common denominators affecting both cancer and inflammatory lesions are now well known: bradykinin (bk), nitric oxide (no), peroxynitrite (onoo(-)), prostaglandins (pgs), collagenases or matrix metalloproteinases (mmps) and others. |
2003-12-17 |
2023-08-12 |
Not clear |