All Relations between Dementia and temporal lobe

Publication Sentence Publish Date Extraction Date Species
B T Volpe, C K Petit. Dementia with bilateral medial temporal lobe ischemia. Neurology. vol 35. issue 12. 1986-01-03. PMID:4069376. neuropathologic examination of two patients with dementia showed chronic bilateral medial temporal lobe ischemic damage that included the hippocampus (particularly the ca-1 region), subiculum, and amygdala. 1986-01-03 2023-08-11 Not clear
B T Volpe, C K Petit. Dementia with bilateral medial temporal lobe ischemia. Neurology. vol 35. issue 12. 1986-01-03. PMID:4069376. the additional damage to medial temporal lobe structures may have caused the dementia. 1986-01-03 2023-08-11 Not clear
J Ulrich, H B Stäheli. The variable topography of Alzheimer type changes in senile dementia and normal old age. Gerontology. vol 30. issue 4. 1984-11-01. PMID:6479600. no patients with isolated temporal lobe changes could be clinically identified as having so-called 'simple senile dementia'. 1984-11-01 2023-08-12 human
P A Barford, J A Blair, C Eggar, C Hamon, C Morar, S B Whitbur. Tetrahydrobiopterin metabolism in the temporal lobe of patients dying with senile dementia of Alzheimer type. Journal of neurology, neurosurgery, and psychiatry. vol 47. issue 7. 1984-09-17. PMID:6747650. tetrahydrobiopterin metabolism in the temporal lobe of patients dying with senile dementia of alzheimer type. 1984-09-17 2023-08-12 human
G K Wilcoc. The temporal lobe in dementia of Alzheimer's type. Gerontology. vol 29. issue 5. 1983-11-23. PMID:6618185. the temporal lobe in dementia of alzheimer's type. 1983-11-23 2023-08-12 Not clear
G K Wilcock, M M Esiri, D M Bowen, C C Smit. Alzheimer's disease. Correlation of cortical choline acetyltransferase activity with the severity of dementia and histological abnormalities. Journal of the neurological sciences. vol 57. issue 2-3. 1983-04-21. PMID:7161627. we have examined the choline acetyltransferase [cat] activity in autopsy samples of frontal and temporal lobe cortex from 47 patients (31 with alzheimer's disease, 4 with dementia due to cerebrovascular disease and 12 undemented controls) and compared it with the severity of dementia during life and with the numbers of argyrophilic plaques and neurofibrillary tangles in the corresponding areas of cortex in the contralateral cerebral hemisphere. 1983-04-21 2023-08-12 cat
G K Wilcock, M M Esir. Plaques, tangles and dementia. A quantitative study. Journal of the neurological sciences. vol 56. issue 2-3. 1983-02-25. PMID:7175555. the temporal lobe cortex and hippocampus were the areas most severely affected by the increased neurofibrillary tangle formation in senile dementia due to alzheimer's disease. 1983-02-25 2023-08-12 human
W Bondareff, R Baldy, R Lev. Quantitative computed tomography in senile dementia. Archives of general psychiatry. vol 38. issue 12. 1982-02-25. PMID:7316681. mean hu values were significantly lower bilaterally in the medial temporal lobe, anterior frontal lobe, and head of the caudate in the group of patients with senile dementia. 1982-02-25 2023-08-12 human
D M Bowen, A N Daviso. Biochemical changes in the cholinergic system of the ageing brain and in senile dementia. Psychological medicine. vol 10. issue 2. 1980-08-25. PMID:6104344. compared with age-matched controls about one third of nerve cell components are lost from the temporal lobe of patients with senile dementia of alzheimer's type. 1980-08-25 2023-08-12 human
D M Bowen, P White, J A Spillane, M J Goodhardt, G Curzon, P Iwangoff, W Meier-Ruge, A N Daviso. Accelerated ageing or selective neuronal loss as an important cause of dementia? Lancet (London, England). vol 1. issue 8106. 1979-03-24. PMID:83462. extensive biochemical analysis of whole temporal lobe from cases of dementia and controls suggests that alzheimer's disease is a primary degenerative nerve-cell disorder and not the result of accelerated ageing. 1979-03-24 2023-08-11 Not clear