All Relations between microtubule-associated protein tau and Multiple System Atrophy

Publication Sentence Publish Date Extraction Date Species
Mathew B Wong, Jacob Goodwin, Anwar Norazit, Adrian C B Meedeniya, Christiane Richter-Landsberg, Wei Ping Gai, Dean L Pountne. SUMO-1 is associated with a subset of lysosomes in glial protein aggregate diseases. Neurotoxicity research. vol 23. issue 1. 2013-10-17. PMID:23229893. multiple system atrophy (msa) is characterized by α-synuclein glial cytoplasmic inclusions and progressive supranuclear palsy (psp) is associated with glial tau inclusions. 2013-10-17 2023-08-12 human
Bartosz Bujan, Markus J Hofer, Wolfgang H Oertel, Axel Pagenstecher, Katrin Bür. Multiple system atrophy of the cerebellar type (MSA-C) with concomitant beta-amyloid and tau pathology. Clinical neuropathology. vol 32. issue 4. 2013-09-04. PMID:23320996. multiple system atrophy of the cerebellar type (msa-c) with concomitant beta-amyloid and tau pathology. 2013-09-04 2023-08-12 Not clear
Angela Roco, Félix Javier Jiménez-Jiménez, Hortensia Alonso-Navarro, Carmen Martínez, Martín Zurdo, Laura Turpín-Fenoll, Jorge Millán, Teresa Adeva-Bartolomé, Esther Cubo, Francisco Navacerrada, Ana Rojo-Sebastián, Lluisa Rubio, Marisol Calleja, José Francisco Plaza-Nieto, Belén Pilo-de-la-Fuente, Margarita Arroyo-Solera, Elena García-Martín, José A G Agúnde. MAPT1 gene rs1052553 variant is unrelated with the risk for restless legs syndrome. Journal of neural transmission (Vienna, Austria : 1996). vol 120. issue 3. 2013-08-12. PMID:23001634. mutations in the microtubule-associated protein tau gene (mapt) can cause frontotemporal dementia with parkinsonism linked to the chromosome 17, and are associated with the risk for progressive supranuclear palsy, parkinson's disease, corticobasal degeneration, and multiple system atrophy. 2013-08-12 2023-08-12 Not clear
Katsuhiko Yanagisaw. [Searching for biomarkers to diagnose dementia]. Nihon rinsho. Japanese journal of clinical medicine. vol 70. issue 5. 2012-08-02. PMID:22620001. in terms of biomarkers for other dementing neurodegenerative diseases, including synucleinopathies such as dementia with lewy bodies and multiple system atrophy, and tauopathies such as progressive supranuclear palsy, corticobasal degeneration, synuclein, tdp-43, progranulin and tau may be candidate proteins for possible biomarkers for these diseases. 2012-08-02 2023-08-12 Not clear
Sigurd D Süssmuth, Ingo Uttner, Bernhard Landwehrmeyer, Elmar H Pinkhardt, Johannes Brettschneider, Axel Petzold, Bernd Kramer, Jörg B Schulz, Christian Palm, Markus Otto, Albert C Ludolph, Jan Kassubek, Hayrettin Tuman. Differential pattern of brain-specific CSF proteins tau and amyloid-β in Parkinsonian syndromes. Movement disorders : official journal of the Movement Disorder Society. vol 25. issue 9. 2010-11-04. PMID:20589870. to evaluate cerebrospinal fluid (csf) proteins reflecting processes of neurodegeneration and glial activation in progressive supranuclear palsy (psp; richardson's syndrome, n = 20; psp-parkinsonism, n = 7) and multiple system atrophy (msa, n = 25), we analyzed tau, phosphorylated tau, amyloid-beta(1-42) (abeta1-42), abeta1-40, glial fibrillary acidic protein (gfap), and csf routine variables. 2010-11-04 2023-08-12 Not clear
W Farid Abdo, Bastiaan R Bloem, Wieneke J Van Geel, Rianne A J Esselink, Marcel M Verbee. CSF neurofilament light chain and tau differentiate multiple system atrophy from Parkinson's disease. Neurobiology of aging. vol 28. issue 5. 2007-05-31. PMID:16678934. csf neurofilament light chain and tau differentiate multiple system atrophy from parkinson's disease. 2007-05-31 2023-08-12 Not clear
Toshiki Uchihara, Charles Duyckaerts, Danielle Seilhean, Ayako Nakamura, Françoise Lazarini, Jean-Jacques Hau. Exclusive induction of tau2 epitope in microglia/macrophages in inflammatory lesions-tautwopathy distinct from degenerative tauopathies. Acta neuropathologica. vol 109. issue 2. 2005-09-21. PMID:15549333. similar modification of tau2 epitope can be induced even in the absence phosphorylation of tau, as we first demonstrated in ischemic foci and in glial cytoplasmic inclusions (gcis) of multiple system atrophy. 2005-09-21 2023-08-12 human
Benoit I Giasson, Meghann E Mabon, John E Duda, Thomas J Montine, David Robertson, Howard I Hurtig, Virginia M-Y Lee, John Q Trojanowsk. Tau and 14-3-3 in glial cytoplasmic inclusions of multiple system atrophy. Acta neuropathologica. vol 106. issue 3. 2004-01-09. PMID:12811584. tau and 14-3-3 in glial cytoplasmic inclusions of multiple system atrophy. 2004-01-09 2023-08-12 Not clear
Osamu Yokota, Seishi Terada, Hideki Ishizu, Kuniaki Tsuchiya, Yoshihiro Kitamura, Kenji Ikeda, Kenji Uéda, Shigetoshi Kurod. NACP/alpha-synuclein immunoreactivity in diffuse neurofibrillary tangles with calcification (DNTC). Acta neuropathologica. vol 104. issue 4. 2002-12-27. PMID:12200618. these findings suggest that (1) dntc shares a common pathophysiological background with parkinson's disease, dementia with lewy bodies, and multiple system atrophy in which abnormal alphas aggregation is observed, and (2) there is an interaction between alphas and tau pathology that does not involve amyloid in dntc. 2002-12-27 2023-08-12 Not clear
H R Morris, A Schrag, U Nath, D Burn, N P Quinn, S Daniel, N W Wood, A J Lee. Effect of ApoE and tau on age of onset of progressive supranuclear palsy and multiple system atrophy. Neuroscience letters. vol 312. issue 2. 2002-01-15. PMID:11595348. we have therefore analysed the relationship between tau and apoe genotypes and the age of onset of psp and another form of atypical parkinsonism, multiple system atrophy (msa). 2002-01-15 2023-08-12 Not clear
H R Morris, A Schrag, U Nath, D Burn, N P Quinn, S Daniel, N W Wood, A J Lee. Effect of ApoE and tau on age of onset of progressive supranuclear palsy and multiple system atrophy. Neuroscience letters. vol 312. issue 2. 2002-01-15. PMID:11595348. effect of apoe and tau on age of onset of progressive supranuclear palsy and multiple system atrophy. 2002-01-15 2023-08-12 Not clear
Y S Piao, S Hayashi, M Hasegawa, K Wakabayashi, M Yamada, M Yoshimoto, A Ishikawa, T Iwatsubo, H Takahash. Co-localization of alpha-synuclein and phosphorylated tau in neuronal and glial cytoplasmic inclusions in a patient with multiple system atrophy of long duration. Acta neuropathologica. vol 101. issue 3. 2001-08-30. PMID:11307630. co-localization of alpha-synuclein and phosphorylated tau in neuronal and glial cytoplasmic inclusions in a patient with multiple system atrophy of long duration. 2001-08-30 2023-08-12 Not clear
H R Morris, J R Vaughan, S R Datta, R Bandopadhyay, H A Rohan De Silva, A Schrag, N J Cairns, D Burn, U Nath, P L Lantos, S Daniel, A J Lees, N P Quinn, N W Woo. Multiple system atrophy/progressive supranuclear palsy: alpha-Synuclein, synphilin, tau, and APOE. Neurology. vol 55. issue 12. 2001-02-01. PMID:11134398. article abstract-alpha synuclein, tau, synphilin, and apoe genotypes were analyzed in patients with multiple system atrophy (msa) and progressive supranuclear palsy (psp) and controls. 2001-02-01 2023-08-12 Not clear
W G Johnso. Late-onset neurodegenerative diseases--the role of protein insolubility. Journal of anatomy. vol 196 ( Pt 4). 2000-08-22. PMID:10923991. in multiple system atrophy, alpha-synuclein precipitates are encountered and in progressive supranuclear palsy, tau precipitates occur. 2000-08-22 2023-08-12 Not clear
K Wakabayashi, I Kawachi, Y Toyoshima, H Takahash. [Occurrence of argyrophilic grains in multiple system atrophy: histopathological examination of 26 autopsy cases]. No to shinkei = Brain and nerve. vol 51. issue 5. 1999-08-23. PMID:10396750. we examined the brains of 26 patients with multiple system atrophy (msa), a neurodegenerative disorder fundamentally lacking tau pathology, histologically and immunocytochemically. 1999-08-23 2023-08-12 Not clear
A Takeda, N Arai, T Komori, E Iseki, S Kato, M Od. Tau immunoreactivity in glial cytoplasmic inclusions in multiple system atrophy. Neuroscience letters. vol 234. issue 1. 1997-12-09. PMID:9347947. in order to clarify the manner and significance of tau expression in glial cytoplasmic inclusions (gcis), ubiquitinated oligodendroglial abnormal structures in multiple system atrophy (msa), an immunohistochemical study was carried out in the lesions of the pontine nuclei of 10 cases of msa using antibodies against various epitope locations of tau protein. 1997-12-09 2023-08-12 Not clear
A Takeda, N Arai, T Komori, E Iseki, S Kato, M Od. Tau immunoreactivity in glial cytoplasmic inclusions in multiple system atrophy. Neuroscience letters. vol 234. issue 1. 1997-12-09. PMID:9347947. tau immunoreactivity in glial cytoplasmic inclusions in multiple system atrophy. 1997-12-09 2023-08-12 Not clear
N J Cairns, P F Atkinson, D P Hanger, B H Anderton, S E Daniel, P L Lanto. Tau protein in the glial cytoplasmic inclusions of multiple system atrophy can be distinguished from abnormal tau in Alzheimer's disease. Neuroscience letters. vol 230. issue 1. 1997-09-24. PMID:9259461. tau protein in the glial cytoplasmic inclusions of multiple system atrophy can be distinguished from abnormal tau in alzheimer's disease. 1997-09-24 2023-08-12 Not clear
N Arai, M I Papp, P L Lanto. New observation on ubiquitinated neurons in the cerebral cortex of multiple system atrophy (MSA). Neuroscience letters. vol 182. issue 2. 1995-05-17. PMID:7715809. to investigate neuronal damage in the cerebral cortex in patients with multiple system atrophy (msa), immunohistochemical stainings were carried out on the prefrontal cortex, the hippocampus, the precentral gyrus, the supplementary motor cortex and the occipital cortex in 6 cases of msa and 6 controls, using antibodies against ubiquitin, tau protein and neurofilaments (bf10, rt97, 147). 1995-05-17 2023-08-12 Not clear