| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| R Schwarcz, K Fuxe, T Hökfelt, L Terenius, M Goldstei. Effects of chronic striatal kainate lesions on some dopaminergic parameters and enkephalin immunoreactive neurons in the basal ganglia. Journal of neurochemistry. vol 34. issue 4. 1980-05-23. PMID:6102111. |
effects of chronic striatal kainate lesions on some dopaminergic parameters and enkephalin immunoreactive neurons in the basal ganglia. |
1980-05-23 |
2023-08-12 |
Not clear |
| A I Balakleevsky, N P Bobrova, N V Shevtchenk. Regulation of gamma-aminobutyric acid metabolism in different brain structures by cholinergic and adrenergic drugs. Annali dell'Istituto superiore di sanita. vol 14. issue 1. 1979-12-29. PMID:756168. |
most dopaminergic drugs (apomorphine, l-dopa, cb-154, fusaric acid, 6-hydroxydopamine, haloperidol) fail to influence gaba metabolism in whole brain and in basal ganglia. |
1979-12-29 |
2023-08-11 |
mouse |
| A R Crossman, M A Sambrook, S W Gergies, P Slate. The neurological basis of motor asymmetry following unilateral 6-hydroxydopamine brain lesions in the rat: the effect of motor decortication. Journal of the neurological sciences. vol 34. issue 3. 1978-02-18. PMID:591996. |
it is postulated that subcortical basal ganglia efferent pathways may be involved in the mediation of motor asymmetry after unilateral 6-ohda lesions of the nigrostriatal dopaminergic projection. |
1978-02-18 |
2023-08-11 |
rat |
| E S Sear. Therapeutics of disordered movement. American family physician. vol 16. issue 3. 1977-10-20. PMID:143204. |
human motor behavior is organized around two major neurotransmitter systems in the basal ganglia--dopaminergic and cholinergic. |
1977-10-20 |
2023-08-11 |
human |
| K Lühdorf, M Lun. Phenytoin-induced hyperkinesia. Epilepsia. vol 18. issue 3. 1977-10-14. PMID:891495. |
the hypothesis is presented that phenytoin induces hyperkinesia by increasing dopaminergic and serotonergic activity in the basal ganglia, and that patients with preexisting basal ganglia damage are the most susceptible. |
1977-10-14 |
2023-08-11 |
Not clear |
| M M Cohen, R T Scheif. Pharmacotherapy of Parkinson's disease. American journal of hospital pharmacy. vol 34. issue 5. 1977-07-23. PMID:326045. |
the pathophysiology, anticholinergic therapy and dopaminergic therapy of parkinson's disease are reviewed; an emphasis is placed on the structure and function of the basal ganglia because of their importance in understanding the pharmacotherapy of parkinsonism. |
1977-07-23 |
2023-08-11 |
Not clear |
| J Gerlac. Relationship between tardive dyskinesia, L-Dopa-induced hyperkinesia and parkinsonism. Psychopharmacology. vol 51. issue 3. 1977-05-20. PMID:403541. |
it is concluded that the irreversible neurotoxic effect of neuroleptic drugs may be associated with age-related changes in the oral somatotopic region of the basal ganglia (to be given consideration in any future search for the pathogenetic process underlying irreversible tardive dyskinesia), and that the pathophysiology of involuntary hyperkinesia in neuroleptic-treated psychiatric patients and in l-dopatreated parkinson patients may consist of a primary dopamine deficiency (pharmacological or structural), and a secondary relative hyperactivity in the dopaminergic system ("dopaminergic hypersensitivity") possibly corresponding to hypoactivity in the cholinergic system. |
1977-05-20 |
2023-08-11 |
Not clear |
| B S Bunney, G K Aghajania. Dopaminergic infllence in the basal ganglia: evidence for striatonigral feedback regulation. Research publications - Association for Research in Nervous and Mental Disease. vol 55. 1977-02-24. PMID:826996. |
dopaminergic infllence in the basal ganglia: evidence for striatonigral feedback regulation. |
1977-02-24 |
2023-08-11 |
Not clear |