| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Wolfgang Enard, Sabine Gehre, Kurt Hammerschmidt, Sabine M Hölter, Torsten Blass, Mehmet Somel, Martina K Brückner, Christiane Schreiweis, Christine Winter, Reinhard Sohr, Lore Becker, Victor Wiebe, Birgit Nickel, Thomas Giger, Uwe Müller, Matthias Groszer, Thure Adler, Antonio Aguilar, Ines Bolle, Julia Calzada-Wack, Claudia Dalke, Nicole Ehrhardt, Jack Favor, Helmut Fuchs, Valérie Gailus-Durner, Wolfgang Hans, Gabriele Hölzlwimmer, Anahita Javaheri, Svetoslav Kalaydjiev, Magdalena Kallnik, Eva Kling, Sandra Kunder, Ilona Mossbrugger, Beatrix Naton, Ildikó Racz, Birgit Rathkolb, Jan Rozman, Anja Schrewe, Dirk H Busch, Jochen Graw, Boris Ivandic, Martin Klingenspor, Thomas Klopstock, Markus Ollert, Leticia Quintanilla-Martinez, Holger Schulz, Eckhard Wolf, Wolfgang Wurst, Andreas Zimmer, Simon E Fisher, Rudolf Morgenstern, Thomas Arendt, Martin Hrabé de Angelis, Julia Fischer, Johannes Schwarz, Svante Pääb. A humanized version of Foxp2 affects cortico-basal ganglia circuits in mice. Cell. vol 137. issue 5. 2009-06-22. PMID:19490899. |
in the striatum, a part of the basal ganglia affected in humans with a speech deficit due to a nonfunctional foxp2 allele, we find that medium spiny neurons have increased dendrite lengths and increased synaptic plasticity. |
2009-06-22 |
2023-08-12 |
mouse |
| Jeffery R Wicken. Synaptic plasticity in the basal ganglia. Behavioural brain research. vol 199. issue 1. 2009-05-13. PMID:19026691. |
synaptic plasticity in the basal ganglia. |
2009-05-13 |
2023-08-12 |
Not clear |
| Jeffery R Wicken. Synaptic plasticity in the basal ganglia. Behavioural brain research. vol 199. issue 1. 2009-05-13. PMID:19026691. |
activity-dependent synaptic plasticity occurs in several parts of the basal ganglia. |
2009-05-13 |
2023-08-12 |
Not clear |
| I A Prescott, J O Dostrovsky, E Moro, M Hodaie, A M Lozano, W D Hutchiso. Levodopa enhances synaptic plasticity in the substantia nigra pars reticulata of Parkinson's disease patients. Brain : a journal of neurology. vol 132. issue Pt 2. 2009-03-10. PMID:19050033. |
studies in rat striatal slices have shown dopamine to be an essential component of activity-dependent synaptic plasticity at the input to the basal ganglia, but dopamine is also released from ventrally projecting dendrites of the substantia nigra pars compacta (snc) on the substantia nigra pars reticulata (snr), a major output structure of the basal ganglia. |
2009-03-10 |
2023-08-12 |
rat |
| I A Prescott, J O Dostrovsky, E Moro, M Hodaie, A M Lozano, W D Hutchiso. Levodopa enhances synaptic plasticity in the substantia nigra pars reticulata of Parkinson's disease patients. Brain : a journal of neurology. vol 132. issue Pt 2. 2009-03-10. PMID:19050033. |
our findings suggest that extrastriatal dopamine modulates activity-dependent synaptic plasticity at basal ganglia output neurons. |
2009-03-10 |
2023-08-12 |
rat |
| Anatol C Kreitzer, Robert C Malenk. Striatal plasticity and basal ganglia circuit function. Neuron. vol 60. issue 4. 2009-01-16. PMID:19038213. |
here, we review current understanding of synaptic plasticity in the striatum and its role in the physiology and pathophysiology of basal ganglia function. |
2009-01-16 |
2023-08-12 |
Not clear |
| Nicola Berretta, Robert Nisticò, Giorgio Bernardi, Nicola Biagio Mercur. Synaptic plasticity in the basal ganglia: a similar code for physiological and pathological conditions. Progress in neurobiology. vol 84. issue 4. 2008-07-30. PMID:18243479. |
synaptic plasticity in the basal ganglia: a similar code for physiological and pathological conditions. |
2008-07-30 |
2023-08-12 |
Not clear |
| Nicola Berretta, Robert Nisticò, Giorgio Bernardi, Nicola Biagio Mercur. Synaptic plasticity in the basal ganglia: a similar code for physiological and pathological conditions. Progress in neurobiology. vol 84. issue 4. 2008-07-30. PMID:18243479. |
here we present an overview of the experimental data regarding synaptic plasticity in the basal ganglia and highlight how results reported in the literature are often contradictory, especially when compared to those obtained in the hippocampal area. |
2008-07-30 |
2023-08-12 |
Not clear |
| Nicola Berretta, Robert Nisticò, Giorgio Bernardi, Nicola Biagio Mercur. Synaptic plasticity in the basal ganglia: a similar code for physiological and pathological conditions. Progress in neurobiology. vol 84. issue 4. 2008-07-30. PMID:18243479. |
in trying to propose possible explanations to some of these contradictions, we present a holistic approach that re-interprets the basal ganglia synaptic plasticity in terms of expression of physiological and pathological phenomena and therapeutic effects of drugs. |
2008-07-30 |
2023-08-12 |
Not clear |
| Sabah Kelai, Gilles Maussion, Florence Noble, Claudette Boni, Nicolas Ramoz, Jean-Marie Moalic, Michel Peuchmaur, Philip Gorwood, Michel Simonnea. Nrxn3 upregulation in the globus pallidus of mice developing cocaine addiction. Neuroreport. vol 19. issue 7. 2008-07-29. PMID:18418251. |
thus, nrxn3 adhesion molecules may play an important role in the synaptic plasticity of neurons involved in the indirect pathways of basal ganglia, in which they regulate reward-related learning. |
2008-07-29 |
2023-08-12 |
mouse |
| Carol A Sege. How do the basal ganglia contribute to categorization? Their roles in generalization, response selection, and learning via feedback. Neuroscience and biobehavioral reviews. vol 32. issue 2. 2008-05-02. PMID:17919725. |
these projections underlie corticostriatal synaptic plasticity across the basal ganglia, and also serve as input to the executive and motivational corticostriatal loops that play a role in strategic use of feedback. |
2008-05-02 |
2023-08-12 |
Not clear |
| I G Sil'ki. The contribution of synaptic plasticity in the basal ganglia to the processing of visual information. Neuroscience and behavioral physiology. vol 37. issue 8. 2008-01-08. PMID:17922242. |
the contribution of synaptic plasticity in the basal ganglia to the processing of visual information. |
2008-01-08 |
2023-08-12 |
Not clear |
| I G Sil'ki. [A contribution of synaptic plasticity in the basal ganglia to processing of visual information]. Zhurnal vysshei nervnoi deiatelnosti imeni I P Pavlova. vol 56. issue 6. 2007-03-01. PMID:17285769. |
[a contribution of synaptic plasticity in the basal ganglia to processing of visual information]. |
2007-03-01 |
2023-08-12 |
Not clear |
| Jenny E Westin, Hanna S Lindgren, Jonathan Gardi, Jens Randel Nyengaard, Patrik Brundin, Paul Mohapel, M Angela Cenc. Endothelial proliferation and increased blood-brain barrier permeability in the basal ganglia in a rat model of 3,4-dihydroxyphenyl-L-alanine-induced dyskinesia. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 26. issue 37. 2006-10-20. PMID:16971529. |
3,4-dihydroxyphenyl-l-alanine (l-dopa)-induced dyskinesia is associated with molecular and synaptic plasticity in the basal ganglia, but the occurrence of structural remodeling through cell genesis has not been explored. |
2006-10-20 |
2023-08-12 |
rat |
| Mario van der Stelt, Vincenzo Di Marz. The endocannabinoid system in the basal ganglia and in the mesolimbic reward system: implications for neurological and psychiatric disorders. European journal of pharmacology. vol 480. issue 1-3. 2004-07-22. PMID:14623357. |
by activating presynaptic cannabinoid cb1 receptors, they can reduce glutamate release in dorsal and ventral striatum (nucleus accumbens) and alter synaptic plasticity, thereby modulating neurotransmission in the basal ganglia and in the mesolimbic reward system. |
2004-07-22 |
2023-08-12 |
Not clear |
| Jonathan M Brotchi. CB1 cannabinoid receptor signalling in Parkinson's disease. Current opinion in pharmacology. vol 3. issue 1. 2003-07-01. PMID:12550742. |
thus, because activation of basal ganglia cb(1) receptors can modulate neurotransmission and contribute to synaptic plasticity in a manner similar to that described in other brain regions, it also appears that endocannabinoids might modulate cell-cell signalling via effects on neurotransmitter re-uptake and postsynaptic actions mediating cross talk between multiple receptor types. |
2003-07-01 |
2023-08-12 |
Not clear |
| P Gubellini, E Saulle, D Centonze, C Costa, D Tropepi, G Bernardi, F Conquet, P Calabres. Corticostriatal LTP requires combined mGluR1 and mGluR5 activation. Neuropharmacology. vol 44. issue 1. 2003-04-30. PMID:12559117. |
the differential function of mglur1 and mglur5 in corticostriatal synaptic plasticity may play a role in the modulation of the motor activity mediated by the basal ganglia, thus providing a substrate for the pharmacological treatment of motor disorders involving the striatum. |
2003-04-30 |
2023-08-12 |
mouse |
| Daphna Joel, Yael Niv, Eytan Ruppi. Actor-critic models of the basal ganglia: new anatomical and computational perspectives. Neural networks : the official journal of the International Neural Network Society. vol 15. issue 4-6. 2003-02-11. PMID:12371510. |
prominent in these are actor-critic models of basal ganglia functioning, which build on the strong resemblance between dopamine neuron activity and the temporal difference prediction error signal in the critic, and between dopamine-dependent long-term synaptic plasticity in the striatum and learning guided by a prediction error signal in the actor. |
2003-02-11 |
2023-08-12 |
Not clear |
| I G Sil'ki. A possible mechanism for the dopamine-evoked synergistic disinhibition of thalamic neurons via the "direct" and "indirect" pathways in the basal ganglia. Neuroscience and behavioral physiology. vol 32. issue 3. 2002-08-19. PMID:12135331. |
the mechanism of synaptic plasticity which we have previously proposed for striatal spiny neurons, along with published data on the predominance of dopamine-sensitive d1/d2 receptors on strionigral/striopallidal neurons, was used as the basis to propose the hypothesis that the induction of long-term potentiation/depression of the efficiency of the cortical inputs to these cells may result from the excitatory/inhibitory actions of dopamine on the activity of the neurons originating the "direct" and "indirect" pathways through the basal ganglia. |
2002-08-19 |
2023-08-12 |
Not clear |
| I G Sil'ki. [A possible mechanism of dopamine-induced synergistic disinhibition of thalamic cells via "direct" and "indirect" pathways in basal ganglia]. Zhurnal vysshei nervnoi deiatelnosti imeni I P Pavlova. vol 51. issue 3. 2002-02-19. PMID:11550635. |
on the basis of the mechanism of synaptic plasticity that we have earlier suggested for striatal spiny neurons and with regard to the known data about the predominance of dopamine-sensitive d1/d2 receptors on the striatonigral/striatopallidal cells it is hypothesized that the induction of the long-term potentiation/depression of the efficacy of excitatory cortical inputs to these cells can underlie the excitatory/inhibitory effect of dopamine on the activity of neurons that originate the "direct"/"indirect" pathways through the basal ganglia. |
2002-02-19 |
2023-08-12 |
Not clear |