| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| L J Ptáce. Autosomal dominant spinocerebellar atrophy with retinal degeneration. Clinical neuroscience (New York, N.Y.). vol 3. issue 1. 1995-08-24. PMID:7614091. |
the autosomal dominant cerebellar ataxias are a clinically and genetically heterogeneous group of disorders. |
1995-08-24 |
2023-08-12 |
Not clear |
| S Belal, F Hentati, C Ben Hamida, M Ben Hamid. Friedreich's ataxia-vitamin E responsive type. The chromosome 8 locus. Clinical neuroscience (New York, N.Y.). vol 3. issue 1. 1995-08-24. PMID:7614093. |
the most common autosomal recessive ataxia is friedreich's ataxia (fa), characterized mainly by an early onset, absent tendon reflexes, deep sensory loss, cerebellar and babinski signs. |
1995-08-24 |
2023-08-12 |
Not clear |
| B Bonnefoi-Kyriacou, E Trouche, E Legallet, F Vialle. Planning and execution of pointing movements in cerebellar patients. Movement disorders : official journal of the Movement Disorder Society. vol 10. issue 2. 1995-06-21. PMID:7753059. |
twelve patients with cerebellar dysfunction including a limb ataxia and 12 age-matched controls performed pointing movements with an arm. |
1995-06-21 |
2023-08-12 |
Not clear |
| P Haggard, R C Miall, D Wade, S Fowler, A Richardson, P Anslow, J Stei. Damage to cerebellocortical pathways after closed head injury: a behavioural and magnetic resonance imaging study. Journal of neurology, neurosurgery, and psychiatry. vol 58. issue 4. 1995-06-05. PMID:7738549. |
all patients had had a closed head injury and all had cerebellar type ataxia. |
1995-06-05 |
2023-08-12 |
Not clear |
| P Haggard, R C Miall, D Wade, S Fowler, A Richardson, P Anslow, J Stei. Damage to cerebellocortical pathways after closed head injury: a behavioural and magnetic resonance imaging study. Journal of neurology, neurosurgery, and psychiatry. vol 58. issue 4. 1995-06-05. PMID:7738549. |
ataxia seen after severe head injury can arise from damage to the superior cerebellar peduncle, which may interfere with the cerebellocortical circuits involved in coordinated movement. |
1995-06-05 |
2023-08-12 |
Not clear |
| H Ackermann, I Hertrich, T Heh. Oral diadochokinesis in neurological dysarthrias. Folia phoniatrica et logopaedica : official organ of the International Association of Logopedics and Phoniatrics (IALP). vol 47. issue 1. 1995-06-01. PMID:7728177. |
the present study performed an acoustic analysis of rapid syllable repetitions in patients suffering from idiopathic parkinson's disease (n = 17), huntington's chorea (n = 14), friedreich's ataxia (n = 9), or from a purely cerebellar syndrome (n = 13). |
1995-06-01 |
2023-08-12 |
human |
| P Trouillas, G Serratrice, D Laplane, A Rascol, P Augustin, G Barroche, M Clanet, C F Degos, C Desnuelle, R Duma. Levorotatory form of 5-hydroxytryptophan in Friedreich's ataxia. Results of a double-blind drug-placebo cooperative study. Archives of neurology. vol 52. issue 5. 1995-05-26. PMID:7733839. |
to study the effect of the levorotatory form of 5-hydroxytryptophan on the cerebellar symptoms of friedreich's ataxia. |
1995-05-26 |
2023-08-12 |
Not clear |
| I N Horowitz, N A Nipark. Vertebral artery dissection with bilateral hemiparesis. Pediatric neurology. vol 11. issue 3. 1995-04-12. PMID:7880341. |
right vertebral artery dissection after trauma, with subsequent cerebellar and thalamic infarcts, is described in a 5-year-old child who presented with mild hemiparesis and ataxia. |
1995-04-12 |
2023-08-12 |
Not clear |
| H Devanne, M Gentil, B Mato. Biomechanical analysis of simple jaw movements in Friedreich's ataxia. Electroencephalography and clinical neurophysiology. vol 97. issue 1. 1995-04-11. PMID:7533718. |
these characteristics are similar to those, previously described, of limb movements in subjects with cerebellar dysfunctions and suggest that the alterations of jaw movement in friedreich's ataxia could be due to a deficit in cerebellar control. |
1995-04-11 |
2023-08-12 |
human |
| R P Symonds, R B Hogg, I Bon. Paraneoplastic neurological syndromes associated with lymphomas. Leukemia & lymphoma. vol 15. issue 5-6. 1995-04-06. PMID:7874006. |
the other with a non hodgkin's lymphoma of thyroid suffered from ataxia owing to cerebellar degeneration. |
1995-04-06 |
2023-08-12 |
Not clear |
| M Giroud, L Septien, J L Pelletier, N Dueret, R Duma. Decrease in cerebellar blood flow in patients with Friedreich's ataxia: A TC-HMPAO SPECT study of three cases. Neurological research. vol 16. issue 5. 1995-03-27. PMID:7870272. |
decrease in cerebellar blood flow in patients with friedreich's ataxia: a tc-hmpao spect study of three cases. |
1995-03-27 |
2023-08-12 |
Not clear |
| R Sandy. Rapid normalization of visual evoked potentials by picoTesla range magnetic fields in chronic progressive multiple sclerosis. The International journal of neuroscience. vol 77. issue 3-4. 1995-02-03. PMID:7814217. |
this communication concerns a 55 year old woman with a 5 year history of chronic progressive multiple sclerosis (ms) in whom a single external application of extremely low magnetic fields (mf) (7.5 picotesla; 5 hz frequency) of 20 minutes duration resulted in a rapid improvement in symptoms including vision, cerebellar symptomatology (ataxia and dysarthria), bladder functions, mood, sleep, cognitive functions and fatigue. |
1995-02-03 |
2023-08-12 |
Not clear |
| R W Balo. Paraneoplastic cerebellar disorders. Otolaryngology--head and neck surgery : official journal of American Academy of Otolaryngology-Head and Neck Surgery. vol 112. issue 1. 1995-02-03. PMID:7816446. |
paraneoplastic cerebellar degeneration typically begins with rapidly progressive ataxia of the trunk and extremities. |
1995-02-03 |
2023-08-12 |
Not clear |
| J Arpa, Y Campos, A Cruz Martínez, M Gutiérrez Molina, J Arenas, M Alonso, I Plaza, C Morales, F Palomo, P Barreir. [Clinical and investigative approaches in mitochondrial diseases. A review of 15 cases]. Neurologia (Barcelona, Spain). vol 9. issue 8. 1995-01-30. PMID:7803049. |
five clinical groups were studied including 5 exercise intolerances (2 with inflammatory myopathy), 3 with myopathies (1 with dilated cardiomyopathy), 2 with progressive external oftalmoplegia (1 associated with cerebellar ataxia+epilepsy+hypertrophic cardiomyopathy+pes cavus), 4 with encephalopathies (3 with myoclonic encephalopathies with ataxia and dementia and 1 with epilepsy and tremor), and 1 with metabolic acidosis and cardiomyopathy. |
1995-01-30 |
2023-08-12 |
Not clear |
| G B Young, S R Oppenheimer, B A Gordon, G A Wells, L P Assis, J H Kreeft, N A Lohuis, W T Blum. Ataxia in institutionalized patients with epilepsy. The Canadian journal of neurological sciences. Le journal canadien des sciences neurologiques. vol 21. issue 3. 1995-01-26. PMID:8000981. |
ataxia of gait was inconsistently associated with cerebellar atrophy. |
1995-01-26 |
2023-08-12 |
Not clear |
| G Luef, A Chemelli, G Birbamer, F Aichner, G Baue. Phenytoin overdosage and cerebellar atrophy in epileptic patients: clinical and MRI findings. European neurology. vol 34 Suppl 1. 1995-01-26. PMID:8001616. |
mild overdosage of phenytoin produces reversible cerebellar symptoms (nystagmus, double vision, dysarthria and ataxia). |
1995-01-26 |
2023-08-12 |
Not clear |
| S Banfi, H Y Zoghb. Molecular genetics of hereditary ataxias. Bailliere's clinical neurology. vol 3. issue 2. 1994-12-28. PMID:7952848. |
the hereditary ataxias are a very heterogeneous group of disorders characterized by cerebellar dysfunction that can be either isolated or accompanied by other neurological manifestations. |
1994-12-28 |
2023-08-12 |
Not clear |
| S Banfi, H Y Zoghb. Molecular genetics of hereditary ataxias. Bailliere's clinical neurology. vol 3. issue 2. 1994-12-28. PMID:7952848. |
last, significant research progress has been accomplished in the field of hereditary ataxias associated with dna repair defects which should facilitate our understanding of mechanisms involved in cerebellar degeneration. |
1994-12-28 |
2023-08-12 |
Not clear |
| G G Miao, R J Smeyne, G D'Arcangelo, N G Copeland, N A Jenkins, J I Morgan, T Curra. Isolation of an allele of reeler by insertional mutagenesis. Proceedings of the National Academy of Sciences of the United States of America. vol 91. issue 23. 1994-12-12. PMID:7972007. |
the reeler (rl/rl) mouse displays a disruption of laminar structures in both the cerebellum and the forebrain and it exhibits tremors, dystonia, and ataxia. |
1994-12-12 |
2023-08-12 |
mouse |
| M Otsuka, Y Ichiya, Y Kuwabara, S Hosokawa, Y Akashi, T Yoshida, T Fukumura, K Masuda, I Goto, M Kat. Striatal 18F-dopa uptake and brain glucose metabolism by PET in patients with syndrome of progressive ataxia. Journal of the neurological sciences. vol 124. issue 2. 1994-12-07. PMID:7964871. |
five of the 11 patients were diagnosed as having cerebellar cortical degeneration (ccd), including 3 with late cerebellar cortical atrophy and 2 with holmes type hereditary ataxia while 6 demonstrated olivopontocerebellar atrophy (opca). |
1994-12-07 |
2023-08-12 |
Not clear |