| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| B Crosso. Subcortical functions in language: a working model. Brain and language. vol 25. issue 2. 1986-01-03. PMID:3904918. |
the current paper explains a model of subcortical language functions that focuses on dynamic interactions between the cortex, the thalamus, and the basal ganglia in the production of spoken language. |
1986-01-03 |
2023-08-11 |
Not clear |
| H A Kretzschmar, S J DeArmond, L S Forn. Measurement of GFAP in hepatic encephalopathy by ELISA and transblots. Journal of neuropathology and experimental neurology. vol 44. issue 5. 1985-10-10. PMID:3897466. |
basal ganglia, cerebral cortex and subcortical white matter were studied for glial fibrillary acidic protein (gfap) in four cases of hepatic encephalopathy (he) with alzheimer ii gliosis and five age-matched controls. |
1985-10-10 |
2023-08-11 |
Not clear |
| H A Kretzschmar, S J DeArmond, L S Forn. Measurement of GFAP in hepatic encephalopathy by ELISA and transblots. Journal of neuropathology and experimental neurology. vol 44. issue 5. 1985-10-10. PMID:3897466. |
both methods revealed a 70% decrease in the average amount of gfap in the cerebral cortex and a 60% decrease in the basal ganglia in he cases. |
1985-10-10 |
2023-08-11 |
Not clear |
| M Ebadi, P C Jobe, H E Lair. The status of vitamin B6 metabolism in brains of genetically epilepsy-prone rats. Epilepsia. vol 26. issue 4. 1985-08-22. PMID:4006895. |
the status of vitamin b6 in brains of genetically epilepsy-prone and epilepsy-resistant rats was ascertained by measuring the concentrations of pyridoxal phosphate and the activities of pyridoxal kinase in the hippocampus, basal ganglia, cerebellum, and cerebral cortex, as well as in the entire brain. |
1985-08-22 |
2023-08-11 |
rat |
| K Stengaard-Pedersen, M Scho. Opioid peptides and receptors in relation to affective illness. Effects of desipramine and lithium on opioid receptors in rat brain. Acta pharmacologica et toxicologica. vol 56 Suppl 1. 1985-05-16. PMID:2984886. |
treatment with desipramine in vivo (10 mg/kg body weight/day) caused a down-regulation of 3h-enkephalinamide binding in the basal ganglia and the hippocampus, while no effects could be observed in the cerebral cortex and the rest of the forebrain. |
1985-05-16 |
2023-08-11 |
rat |
| E E Estévez, D Jerusalinsky, J H Medina, E De Roberti. Cholinergic muscarinic receptors in rat cerebral cortex, basal ganglia and cerebellum undergo rapid and reversible changes after acute stress. Neuroscience. vol 13. issue 4. 1985-04-09. PMID:6527794. |
cholinergic muscarinic receptors in rat cerebral cortex, basal ganglia and cerebellum undergo rapid and reversible changes after acute stress. |
1985-04-09 |
2023-08-12 |
rat |
| B Azzarelli, J Muller, B Ghetti, M Dyken, P M Conneall. Cerebellar plaques in familial Alzheimer's disease (Gerstmann-Sträussler-Scheinker variant?). Acta neuropathologica. vol 65. issue 3-4. 1985-04-01. PMID:3883687. |
neuropathologically, there were plaques and neurofibrillary tangles in the cerebral cortex as well as some in the basal ganglia, particularly reminiscent of the plaques seen in kuru; there was only minimal spinal cord disease (pyramidal tract field). |
1985-04-01 |
2023-08-11 |
Not clear |
| J L Cummings, D F Benso. Subcortical dementia. Review of an emerging concept. Archives of neurology. vol 41. issue 8. 1984-08-27. PMID:6235797. |
disorders manifesting subcortical dementia have pathologic changes that involve primarily the thalamus, basal ganglia, and related brain-stem nuclei with relative sparing of the cerebral cortex. |
1984-08-27 |
2023-08-12 |
Not clear |
| L P Chao, K S Kan, F M Hun. Immunohistochemical localization of choline acetyltransferase in rabbit forebrain. Brain research. vol 235. issue 1. 1984-08-13. PMID:6765220. |
choline acetyltransferase was localized in olfactory bulb, olfactory tract, olfactory tubercle, piriform cortex, septum, diagonal band, basal ganglia, thalamus, hypothalamus, subthalamus, habenula, cerebral cortex, hippocampal region, corpus callosum, internal capsule, fornix, longitudinal striae and other areas. |
1984-08-13 |
2023-08-12 |
rabbit |
| B O Kjos, M Brant-Zawadzki, R G Youn. Early CT findings of global central nervous system hypoperfusion. AJR. American journal of roentgenology. vol 141. issue 6. 1984-01-07. PMID:6606321. |
the abnormal ct findings can be ascribed to increased vulnerability of the cerebral cortex and basal ganglia to hypotensive episodes. |
1984-01-07 |
2023-08-12 |
Not clear |
| M M Singh, R E Becker, R K Pitman, H A Nasrallah, H La. Sustained improvement in tardive dyskinesia with diazepam: indirect evidence for corticolimbic involvement. Brain research bulletin. vol 11. issue 2. 1983-12-20. PMID:6313141. |
since benzodiazepine receptors and sites of action seem to be mainly in the neocortex (especially frontal), limbic cortex, and deep limbs nuclei, and these structures provide most of the input into the nigrostriatopallidal system that probably regulates its role in voluntary movement, it may be suggested that impaired corticolimbic control of basal ganglia may be a factor in the pathogenesis of tardive dyskinesia. |
1983-12-20 |
2023-08-12 |
Not clear |
| A Tsuda, M Tanaka, Y Kohno, Y Ida, Y Hoaki, K Iimori, R Nakagawa, T Nishikawa, N Nagasak. Daily increase in noradrenaline turnover in brain regions of activity-stressed rats. Pharmacology, biochemistry, and behavior. vol 19. issue 3. 1983-12-20. PMID:6634888. |
changes in contents of noradrenaline (na) and its major metabolite, 3-methoxy-4-hydroxyphenylethyleneglycol sulfate (mhpg-so4) in brain regions (the hypothalamus, amygdala, thalamus, hippocampus, midbrain, cerebral cortex, pons plus medulla oblongata and basal ganglia) of male wistar rats were evaluated after 1, 3 or 5 days of exposure to the activity-stress paradigm, wherein rats were housed in a cage with a running-wheel and restricted to 1-hr of feeding per day. |
1983-12-20 |
2023-08-12 |
rat |
| b' V Kv\\xc3\\xadcala, J Vymazal, S Nevsimalov\\xc3\\xa. Computed tomography of Wilson disease. AJNR. American journal of neuroradiology. vol 4. issue 3. 1983-09-20. PMID:6410762.' |
in 10 patients, ct abnormalities were graded as mild: there were atrophic changes around the basal ganglia and in the cortex and cerebellum. |
1983-09-20 |
2023-08-12 |
Not clear |
| K R Reuhl, E A Smallridge, L W Chang, B A Mackenzi. Developmental effects of trimethyltin intoxication in the neonatal mouse. I. Light microscopic studies. Neurotoxicology. vol 4. issue 1. 1983-09-20. PMID:6683824. |
cerebellar purkinje and granule neurons, basal ganglia and cerebral cortex were involved to a lesser extent. |
1983-09-20 |
2023-08-12 |
mouse |
| R Hes. [Neurophysiological mechanisms of pain perception]. Methods and findings in experimental and clinical pharmacology. vol 4. issue 7. 1983-06-10. PMID:6302416. |
from there, pathways to the somatosensory cortex, basal ganglia, and limbic structures were described, which probably mediate conscious perception, subconscious motor reactions and emotional colouring of pain perception, respectively. |
1983-06-10 |
2023-08-12 |
Not clear |
| S E Hays, S M Pau. CCK receptors and human neurological disease. Life sciences. vol 31. issue 4. 1983-01-19. PMID:6292647. |
cck binding was significantly reduced inthe basal ganglia and cerebral cortex of huntington's patients, but was normal in the temporal and cingulate cortex of patients with alzheimer's disease. |
1983-01-19 |
2023-08-12 |
human |
| A Raab, G Koje. A10 lesion and passive avoidance latency: correlation with limbic tyrosine-hydroxylase-activity. Pharmacology, biochemistry, and behavior. vol 17. issue 1. 1982-12-02. PMID:6126891. |
depletion of the thy-activity in the septum (se), the entorhinal cortex (eco), the frontal cortex and the basal ganglia correlated with an impairment in the performance of a passive avoidance task (indicated by testing 24 hours postshock). |
1982-12-02 |
2023-08-12 |
Not clear |
| H J Manz, A R Colo. Neuropathology, pathogenesis, and neuropsychiatric sequelae of Reye syndrome. Journal of the neurological sciences. vol 53. issue 2. 1982-04-22. PMID:7057216. |
the neuropathologic substrate for the clinical manifestations of mental retardation, recurrent seizures, movement disorders, sensori-motor deficits, psychobiologic maladjustment, and mediocre performance in school and on formal psychometric tests consists of multifocal infarction and astrocytosis in cerebral cortex, diencephalon, basal ganglia, and brainstem. |
1982-04-22 |
2023-08-12 |
Not clear |
| S E Hays, F K Goodwin, S M Pau. Cholecystokinin receptors are decreased in basal ganglia and cerebral cortex of Huntington's disease. Brain research. vol 225. issue 2. 1982-02-12. PMID:6272931. |
cholecystokinin (cck) receptors were found to be significantly reduced in basal ganglia and cerebral cortex of post-mortem from huntington's patients with matched controls. |
1982-02-12 |
2023-08-12 |
Not clear |
| S E Hays, F K Goodwin, S M Pau. Cholecystokinin receptors are decreased in basal ganglia and cerebral cortex of Huntington's disease. Brain research. vol 225. issue 2. 1982-02-12. PMID:6272931. |
the magnitude of the reduction in cck binding (69% in basal ganglia, 43% in cerebral cortex) is consistent with the degree of neuronal degeneration in basal ganglia, but suggests a possibly selective loss of cck receptor-containing neurons in cerebral cortex of huntington's patients. |
1982-02-12 |
2023-08-12 |
Not clear |