All Relations between Epilepsy, Temporal Lobe and ca1 field

Publication Sentence Publish Date Extraction Date Species
A Kivi, T N Lehmann, R Kovács, A Eilers, R Jauch, H J Meencke, A von Deimling, U Heinemann, S Gabrie. Effects of barium on stimulus-induced rises of [K+]o in human epileptic non-sclerotic and sclerotic hippocampal area CA1. The European journal of neuroscience. vol 12. issue 6. 2000-08-11. PMID:10886343. in the hippocampus of patients with therapy-refractory temporal lobe epilepsy, glial cells of area ca1 might be less able to take up potassium ions via barium-sensitive inwardly rectifying and voltage-independent potassium channels. 2000-08-11 2023-08-12 human
M R Looney, F C Dohan, K G Davies, M Seidenberg, B P Hermann, J B Schweitze. Synaptophysin immunoreactivity in temporal lobe epilepsy-associated hippocampal sclerosis. Acta neuropathologica. vol 98. issue 2. 1999-09-09. PMID:10442558. sector ca4 seemed to respond in a more graded or continuous way to the pathological insults occurring in temporal lobe epilepsy than did ca1, which appeared to exhibit an all or nothing response. 1999-09-09 2023-08-12 Not clear
R Gutiérrez, V Armand, S Schuchmann, U Heineman. Epileptiform activity induced by low Mg2+ in cultured rat hippocampal slices. Brain research. vol 815. issue 2. 1999-02-26. PMID:9878794. besides the establishment of reciprocal connections between area ca1 and the dentate gyrus (dg), collateral excitatory connections between granule cells are formed which are similar to those appearing in several epilepsy models and in the dg from patients with temporal lobe epilepsy. 1999-02-26 2023-08-12 rat
P S Mangan, E W Lothma. Profound disturbances of pre- and postsynaptic GABAB-receptor-mediated processes in region CA1 in a chronic model of temporal lobe epilepsy. Journal of neurophysiology. vol 76. issue 2. 1998-03-16. PMID:8871236. profound disturbances of pre- and postsynaptic gabab-receptor-mediated processes in region ca1 in a chronic model of temporal lobe epilepsy. 1998-03-16 2023-08-12 Not clear
P S Mangan, E W Lothma. Profound disturbances of pre- and postsynaptic GABAB-receptor-mediated processes in region CA1 in a chronic model of temporal lobe epilepsy. Journal of neurophysiology. vol 76. issue 2. 1998-03-16. PMID:8871236. this report examines alterations in presynaptic and postsynaptic processes mediated by gamma-aminobutyric acid-b (gabab) receptors within hippocampal region ca1 in a model of chronic temporal lobe epilepsy (tle). 1998-03-16 2023-08-12 Not clear
M Esclapez, J C Hirsch, R Khazipov, Y Ben-Ari, C Bernar. Operative GABAergic inhibition in hippocampal CA1 pyramidal neurons in experimental epilepsy. Proceedings of the National Academy of Sciences of the United States of America. vol 94. issue 22. 1997-12-04. PMID:9342378. patch-clamp recordings of ca1 interneurons and pyramidal cells were performed in hippocampal slices from kainate- or pilocarpine-treated rat models of temporal lobe epilepsy. 1997-12-04 2023-08-12 rat
D A Rempe, E H Bertram, J M Williamson, E W Lothma. Interneurons in area CA1 stratum radiatum and stratum oriens remain functionally connected to excitatory synaptic input in chronically epileptic animals. Journal of neurophysiology. vol 78. issue 3. 1997-11-06. PMID:9310439. in conclusion, our data suggest that stimulus-evoked and spontaneous excitatory synaptic input to area ca1 interneurons remains functional in an animal model of chronic temporal lobe epilepsy. 1997-11-06 2023-08-12 human
P S Mangan, E H Bertra. Shortened-duration GABA(A) receptor-mediated synaptic potentials underlie enhanced CA1 excitability in a chronic model of temporal lobe epilepsy. Neuroscience. vol 80. issue 4. 1997-10-07. PMID:9284063. shortened-duration gaba(a) receptor-mediated synaptic potentials underlie enhanced ca1 excitability in a chronic model of temporal lobe epilepsy. 1997-10-07 2023-08-12 rat
P S Mangan, E H Bertra. Shortened-duration GABA(A) receptor-mediated synaptic potentials underlie enhanced CA1 excitability in a chronic model of temporal lobe epilepsy. Neuroscience. vol 80. issue 4. 1997-10-07. PMID:9284063. intracellular recording techniques were used to examine gaba(a) receptor-mediated synaptic inhibition in pyramidal cells of the ca1 region of the rat hippocampus in the post-self sustaining limbic status epilepticus model of temporal lobe epilepsy. 1997-10-07 2023-08-12 rat
J W Gibbs, M D Shumate, D A Coulte. Differential epilepsy-associated alterations in postsynaptic GABA(A) receptor function in dentate granule and CA1 neurons. Journal of neurophysiology. vol 77. issue 4. 1997-07-02. PMID:9114245. alterations in gabaergic function associated with the development of temporal lobe epilepsy (tle) were examined with the use of patch-clamp recording techniques in dentate granule (dg) and ca1 neurons acutely isolated from control and spontaneously epileptic rats in which tle was elicited by pilocarpine injection 3-17 wk before use. 1997-07-02 2023-08-12 rat
O Quesada, J C Hirsch, H Gozlan, Y Ben-Ari, C Bernar. Epileptiform activity but not synaptic plasticity is blocked by oxidation of NMDA receptors in a chronic model of temporal lobe epilepsy. Epilepsy research. vol 26. issue 2. 1997-06-25. PMID:9095399. in this ex vivo experimental model of human temporal lobe epilepsy, stimulation of the surviving commissural fibres in stratum radiatum produced graded epileptiform activity in the ca1 area. 1997-06-25 2023-08-12 human
D A Rempe, P S Mangan, E W Lothma. Regional heterogeneity of pathophysiological alterations in CA1 and dentate gyrus in a chronic model of temporal lobe epilepsy. Journal of neurophysiology. vol 74. issue 2. 1995-12-07. PMID:7472385. regional heterogeneity of pathophysiological alterations in ca1 and dentate gyrus in a chronic model of temporal lobe epilepsy. 1995-12-07 2023-08-12 Not clear
P S Mangan, D A Rempe, E W Lothma. Changes in inhibitory neurotransmission in the CA1 region and dentate gyrus in a chronic model of temporal lobe epilepsy. Journal of neurophysiology. vol 74. issue 2. 1995-12-07. PMID:7472386. changes in inhibitory neurotransmission in the ca1 region and dentate gyrus in a chronic model of temporal lobe epilepsy. 1995-12-07 2023-08-12 Not clear
P S Mangan, D A Rempe, E W Lothma. Changes in inhibitory neurotransmission in the CA1 region and dentate gyrus in a chronic model of temporal lobe epilepsy. Journal of neurophysiology. vol 74. issue 2. 1995-12-07. PMID:7472386. in this report we compare changes in inhibitory neurotransmission within the ca1 region and the dentate gyrus (dg) in a model of chronic temporal lobe epilepsy (tle). 1995-12-07 2023-08-12 Not clear
E W Lothman, D A Rempe, P S Manga. Changes in excitatory neurotransmission in the CA1 region and dentate gyrus in a chronic model of temporal lobe epilepsy. Journal of neurophysiology. vol 74. issue 2. 1995-12-07. PMID:7472387. changes in excitatory neurotransmission in the ca1 region and dentate gyrus in a chronic model of temporal lobe epilepsy. 1995-12-07 2023-08-12 Not clear
E W Lothman, D A Rempe, P S Manga. Changes in excitatory neurotransmission in the CA1 region and dentate gyrus in a chronic model of temporal lobe epilepsy. Journal of neurophysiology. vol 74. issue 2. 1995-12-07. PMID:7472387. in this report we compare changes of excitatory neurotransmission within the ca1 region and the dentate gyrus (dg) in a model of chronic temporal lobe epilepsy (tle). 1995-12-07 2023-08-12 Not clear
A Williamson, D D Spence. Electrophysiological characterization of CA2 pyramidal cells from epileptic humans. Hippocampus. vol 4. issue 2. 1994-12-20. PMID:7951697. the ca2 region of the hippocampus is more resistant to the principal cell loss seen in ca1 and ca3 in both animal models of temporal lobe epilepsy and in medial temporal lobe sclerosis (mts), a common neuropathological finding in human temporal lobe epilepsy. 1994-12-20 2023-08-12 human
S Nakajima, J E Franck, D Bilkey, P A Schwartzkroi. Local circuit synaptic interactions between CA1 pyramidal cells and interneurons in the kainate-lesioned hyperexcitable hippocampus. Hippocampus. vol 1. issue 1. 1994-01-13. PMID:1669343. following kainate (ka)-induced lesions of subfield ca3--a lesion relevant to human temporal lobe epilepsy--remaining pyramidal cells in ca1 display synchronous hyperexcitability associated with a loss of synaptic inhibition. 1994-01-13 2023-08-11 human
D W Zaidel, M M Esiri, J M Oxbur. Regional differentiation of cell densities in the left and right hippocampi of epileptic patients. Journal of neurology. vol 240. issue 5. 1993-08-11. PMID:8326341. neuronal cell densities in surgically removed left or right hippocampal tissue of epileptic patients suffering from temporal lobe epilepsy were determined in ca1, ca4, and the dentate gyrus (dg). 1993-08-11 2023-08-12 Not clear
C L Meier, A Obenaus, F E Dude. Persistent hyperexcitability in isolated hippocampal CA1 of kainate-lesioned rats. Journal of neurophysiology. vol 68. issue 6. 1993-03-03. PMID:1491262. subcutaneous kainate injection in rats evoked acute seizures and led to cell loss in the hilus and areas ca1 and ca3, which resembled the pattern of hippocampal sclerosis often associated with temporal lobe epilepsy in humans. 1993-03-03 2023-08-11 rat