| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| M P Laakso, K Juottonen, K Partanen, P Vainio, H Soinine. MRI volumetry of the hippocampus: the effect of slice thickness on volume formation. Magnetic resonance imaging. vol 15. issue 2. 1997-07-28. PMID:9106155. |
the hippocampus is a mesial temporal lobe structure of complex shape and the main locus of temporal lobe epilepsy and also affected early in alzheimer's disease. |
1997-07-28 |
2023-08-12 |
human |
| B M Longo, L E Mell. Blockade of pilocarpine- or kainate-induced mossy fiber sprouting by cycloheximide does not prevent subsequent epileptogenesis in rats. Neuroscience letters. vol 226. issue 3. 1997-07-22. PMID:9175592. |
post-injury sprouting of hippocampal mossy fibers has been suggested to be a causal mechanism underlying the development of temporal lobe epilepsy. |
1997-07-22 |
2023-08-12 |
rat |
| G W Mathern, E H Bertram, T L Babb, J K Pretorius, P A Kuhlman, S Spradlin, D Mendoz. In contrast to kindled seizures, the frequency of spontaneous epilepsy in the limbic status model correlates with greater aberrant fascia dentata excitatory and inhibitory axon sprouting, and increased staining for N-methyl-D-aspartate, AMPA and GABA(A) receptors. Neuroscience. vol 77. issue 4. 1997-07-01. PMID:9130782. |
the hippocampal pathological findings in the limbic status model are similar to those in humans with hippocampal sclerosis and mesial temporal lobe epilepsy, and support the hypothesis that synaptic reorganization of both excitatory and inhibitory systems in the fascia dentata is an important pathophysiological mechanism that probably contributes to or generates chronic limbic seizures. |
1997-07-01 |
2023-08-12 |
rat |
| J Tuunanen, T Halonen, A Pitkäne. Decrease in somatostatin-immunoreactive neurons in the rat amygdaloid complex in a kindling model of temporal lobe epilepsy. Epilepsy research. vol 26. issue 2. 1997-06-25. PMID:9095393. |
in human temporal lobe epilepsy, seizures can begin in the hippocampus, amygdala, or surrounding cortical areas. |
1997-06-25 |
2023-08-12 |
human |
| J Tuunanen, T Halonen, A Pitkäne. Decrease in somatostatin-immunoreactive neurons in the rat amygdaloid complex in a kindling model of temporal lobe epilepsy. Epilepsy research. vol 26. issue 2. 1997-06-25. PMID:9095393. |
we used an amygdala-kindling model of temporal lobe epilepsy to study whether seizures of amygdaloid origin cause damage to the amygdala and hippocampus. |
1997-06-25 |
2023-08-12 |
human |
| L Marsh, M J Morrell, P K Shear, E V Sullivan, H Freeman, A Marie, K O Lim, A Pfefferbau. Cortical and hippocampal volume deficits in temporal lobe epilepsy. Epilepsia. vol 38. issue 5. 1997-06-20. PMID:9184604. |
cortical and hippocampal volume deficits in temporal lobe epilepsy. |
1997-06-20 |
2023-08-12 |
Not clear |
| M Quigg, E H Bertram, T Jackson, E Law. Volumetric magnetic resonance imaging evidence of bilateral hippocampal atrophy in mesial temporal lobe epilepsy. Epilepsia. vol 38. issue 5. 1997-06-20. PMID:9184605. |
volumetric magnetic resonance imaging evidence of bilateral hippocampal atrophy in mesial temporal lobe epilepsy. |
1997-06-20 |
2023-08-12 |
Not clear |
| M Quigg, E H Bertram, T Jackson, E Law. Volumetric magnetic resonance imaging evidence of bilateral hippocampal atrophy in mesial temporal lobe epilepsy. Epilepsia. vol 38. issue 5. 1997-06-20. PMID:9184605. |
we measured absolute volumes and volume differences of hippocampi in patients with mesial temporal lobe epilepsy (mtle) using volumetric magnetic resonance imaging (mri) to determine the extent of bilateral atrophy in mtle and to relate hippocampal volumes (hv) to outcome of temporal lobectomy. |
1997-06-20 |
2023-08-12 |
Not clear |
| b' I E Holopainen, N M Lundbom, E L Mets\\xc3\\xa4honkala, M E Komu, P H Sonninen, M T Haaparanta, J R Bergman, M L Sillanp\\xc3\\xa4\\xc3\\xa. Temporal lobe pathology in epilepsy: proton magnetic resonance spectroscopy and positron emission tomography study. Pediatric neurology. vol 16. issue 2. 1997-06-19. PMID:9090682.' |
hippocampal atrophy characterized by neuronal loss is a common feature in intractable temporal lobe epilepsy in adults. |
1997-06-19 |
2023-08-12 |
Not clear |
| K Hashizume, K Takizawa, M Kunimoto, K Yoshida, T Tanaka, Y Yonemasu, S Ochi, K Hash. [Hippocampal hemosiderin deposit due to large pituitary adenoma presenting temporal lobe epilepsy--a case report]. No to shinkei = Brain and nerve. vol 49. issue 4. 1997-06-18. PMID:9125746. |
[hippocampal hemosiderin deposit due to large pituitary adenoma presenting temporal lobe epilepsy--a case report]. |
1997-06-18 |
2023-08-12 |
Not clear |
| K Hashizume, K Takizawa, M Kunimoto, K Yoshida, T Tanaka, Y Yonemasu, S Ochi, K Hash. [Hippocampal hemosiderin deposit due to large pituitary adenoma presenting temporal lobe epilepsy--a case report]. No to shinkei = Brain and nerve. vol 49. issue 4. 1997-06-18. PMID:9125746. |
the hemosiderin deposition in the hippocampus was inferred to have resulted from intratumoral hemorrhage due to bromocriptine therapy, and it may have caused the temporal lobe epilepsy in this patient. |
1997-06-18 |
2023-08-12 |
Not clear |
| H Beck, R Steffens, U Heinemann, C E Elge. Properties of voltage-activated Ca2+ currents in acutely isolated human hippocampal granule cells. Journal of neurophysiology. vol 77. issue 3. 1997-06-13. PMID:9084617. |
properties of ba2+ currents through voltage-dependent ca2+ channels (iba) were investigated in 61 dentate granule cells acutely isolated from the resected hippocampus of nine patients with therapy-refractory temporal lobe epilepsy (tle). |
1997-06-13 |
2023-08-12 |
human |
| K Hashizume, T Tanaka, M Kunimoto, T Maeda, Y Yonemas. [Image-guided surgery for epilepsy]. No shinkei geka. Neurological surgery. vol 25. issue 4. 1997-06-04. PMID:9125716. |
one of the temporal lobe epilepsy cases had focal cortical dysplasia in the left posterior temporal lobe, and the other one showed that left hippocampal atrophy on mr images. |
1997-06-04 |
2023-08-12 |
Not clear |
| B P Hermann, M Seidenberg, J Schoenfeld, K Davie. Neuropsychological characteristics of the syndrome of mesial temporal lobe epilepsy. Archives of neurology. vol 54. issue 4. 1997-05-08. PMID:9109737. |
to identify the neuropsychological features of the syndrome of mesial temporal lobe epilepsy (mtle), a surgically remediable epileptic syndrome defined by the presence of hippocampal sclerosis, using a broad and comprehensive neuropsychological test battery. |
1997-05-08 |
2023-08-12 |
Not clear |
| G W Mathern, T L Babb, P E Micevych, C E Blanco, J K Pretoriu. Granule cell mRNA levels for BDNF, NGF, and NT-3 correlate with neuron losses or supragranular mossy fiber sprouting in the chronically damaged and epileptic human hippocampus. Molecular and chemical neuropathology. vol 30. issue 1-2. 1997-05-06. PMID:9138429. |
this study determined in temporal lobe epilepsy patients if there were correlations among hippocampal granule cell expression of neurotrophin mrnas, aberrant supragranular mossy fiber sprouting, and neuron losses. |
1997-05-06 |
2023-08-12 |
human |
| J M Babity, J N Armstrong, J C Plumier, R W Currie, H A Robertso. A novel seizure-induced synaptotagmin gene identified by differential display. Proceedings of the National Academy of Sciences of the United States of America. vol 94. issue 6. 1997-04-24. PMID:9122248. |
systemic administration of kainic acid, a cyclic analogue of glutamate, produces many of the clinical features of human temporal lobe epilepsy and status epilepticus in rats, including the induction of motor convulsions and the degeneration of neurons in the hippocampus and piriform cortex. |
1997-04-24 |
2023-08-12 |
human |
| M Simonato, P Romuald. Dynorphin and epilepsy. Progress in neurobiology. vol 50. issue 5-6. 1997-04-17. PMID:9015827. |
those characteristic of temporal lobe epilepsy, because; (1) dynorphin is highly expressed in various parts of the limbic system, and particularly in the granule cells of the hippocampus; (2) dynorphin appears to be released in the hippocampus (and in other brain areas) during complex partial seizures; (3) released dynorphin inhibits excitatory neurotransmission at multiple synapses in the hippocampus via activation of kappa opioid receptors; (4) kappa opioid receptor agonists are highly effective against limbic seizures. |
1997-04-17 |
2023-08-12 |
Not clear |
| A K Shetty, D A Turne. Development of fetal hippocampal grafts in intact and lesioned hippocampus. Progress in neurobiology. vol 50. issue 5-6. 1997-04-17. PMID:9015829. |
fetal hippocampal grafting paradigms are of considerable significance because of their potential to treat neurological disorders affecting primarily hippocampus, including temporal lobe epilepsy, cerebral ischemia, stroke, and head injury. |
1997-04-17 |
2023-08-12 |
Not clear |
| M Isokaw. Preservation of dendrites with the presence of reorganized mossy fiber collaterals in hippocampal dentate granule cells in patients with temporal lobe epilepsy. Brain research. vol 744. issue 2. 1997-04-16. PMID:9027394. |
preservation of dendrites with the presence of reorganized mossy fiber collaterals in hippocampal dentate granule cells in patients with temporal lobe epilepsy. |
1997-04-16 |
2023-08-12 |
human |
| M Isokaw. Preservation of dendrites with the presence of reorganized mossy fiber collaterals in hippocampal dentate granule cells in patients with temporal lobe epilepsy. Brain research. vol 744. issue 2. 1997-04-16. PMID:9027394. |
dendritic morphology was studied in human hippocampal dentate granule cells (dgcs) by intracellularly-injecting biocytin in slice preparations that were obtained from temporal lobe epilepsy patients who underwent a surgical treatment for medically-intractable seizures. |
1997-04-16 |
2023-08-12 |
human |