| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| A Pitkäne. Treatment with antiepileptic drugs: possible neuroprotective effects. Neurology. vol 47. issue 1 Suppl 1. 1996-08-15. PMID:8677031. |
findings from studies involving both rat models of epilepsy and humans with temporal lobe epilepsy suggest that epileptic seizures may induce hippocampal neuronal damage. |
1996-08-15 |
2023-08-12 |
rat |
| A Pitkänen, M Laakso, R Kälviäinen, K Partanen, P Vainio, M Lehtovirta, P Riekkinen, H Soinine. Severity of hippocampal atrophy correlates with the prolongation of MRI T2 relaxation time in temporal lobe epilepsy but not in Alzheimer's disease. Neurology. vol 46. issue 6. 1996-07-25. PMID:8649578. |
severity of hippocampal atrophy correlates with the prolongation of mri t2 relaxation time in temporal lobe epilepsy but not in alzheimer's disease. |
1996-07-25 |
2023-08-12 |
human |
| A Pitkänen, M Laakso, R Kälviäinen, K Partanen, P Vainio, M Lehtovirta, P Riekkinen, H Soinine. Severity of hippocampal atrophy correlates with the prolongation of MRI T2 relaxation time in temporal lobe epilepsy but not in Alzheimer's disease. Neurology. vol 46. issue 6. 1996-07-25. PMID:8649578. |
we analyzed hippocampal volumes and t2 relaxation times by mri from 78 control subjects, 24 patients with temporal lobe epilepsy, and 55 patients with alzheimer's disease (ad). |
1996-07-25 |
2023-08-12 |
human |
| A Pitkänen, M Laakso, R Kälviäinen, K Partanen, P Vainio, M Lehtovirta, P Riekkinen, H Soinine. Severity of hippocampal atrophy correlates with the prolongation of MRI T2 relaxation time in temporal lobe epilepsy but not in Alzheimer's disease. Neurology. vol 46. issue 6. 1996-07-25. PMID:8649578. |
these data show that the degree of prolongation of t2 relaxation time is associated with severity of hippocampal atrophy in temporal lobe epilepsy but not in ad. |
1996-07-25 |
2023-08-12 |
human |
| W M O'Connor, L Masukawa, A Freese, M R Sperling, J A French, M J O'Conno. Hippocampal cell distributions in temporal lobe epilepsy: a comparison between patients with and without an early risk factor. Epilepsia. vol 37. issue 5. 1996-06-10. PMID:8617172. |
hippocampal cell distributions in temporal lobe epilepsy: a comparison between patients with and without an early risk factor. |
1996-06-10 |
2023-08-12 |
Not clear |
| W M O'Connor, L Masukawa, A Freese, M R Sperling, J A French, M J O'Conno. Hippocampal cell distributions in temporal lobe epilepsy: a comparison between patients with and without an early risk factor. Epilepsia. vol 37. issue 5. 1996-06-10. PMID:8617172. |
our results support the hypothesis of a predominantly anterior hippocampal insult in temporal lobe epilepsy (tle). |
1996-06-10 |
2023-08-12 |
Not clear |
| S L Free, L M Li, D R Fish, S D Shorvon, J M Steven. Bilateral hippocampal volume loss in patients with a history of encephalitis or meningitis. Epilepsia. vol 37. issue 4. 1996-05-10. PMID:8603647. |
volumetric analysis of high-quality magnetic resonance imaging (mri) scans identifies asymmetric hippocampal atrophy in most patients with temporal lobe epilepsy. |
1996-05-10 |
2023-08-12 |
human |
| C Schwarzer, G Sper. Hippocampal granule cells express glutamic acid decarboxylase-67 after limbic seizures in the rat. Neuroscience. vol 69. issue 3. 1996-04-18. PMID:8596641. |
on the other hand, increased expression of glutamic acid decarboxylase, the rate-limiting enzyme of gaba synthesis, has been found in interneurons of the hippocampus in patients with temporal lobe epilepsy and in rats after kainic acid-induced limbic seizures, indicating increased gaba-ergic transmission. |
1996-04-18 |
2023-08-12 |
rat |
| C Schwarzer, J M Williamson, E W Lothman, A Vezzani, G Sper. Somatostatin, neuropeptide Y, neurokinin B and cholecystokinin immunoreactivity in two chronic models of temporal lobe epilepsy. Neuroscience. vol 69. issue 3. 1996-04-18. PMID:8596652. |
somatostatin-, neuropeptide y-, neurokinin b- and cholecystokinin-containing neurons were investigated in the rat hippocampus in two chronic models of temporal lobe epilepsy, i.e. |
1996-04-18 |
2023-08-12 |
rat |
| R Jooma, H S Yeh, M D Privitera, D Rigrish, M Gartne. Seizure control and extent of mesial temporal resection. Acta neurochirurgica. vol 133. issue 1-2. 1996-02-26. PMID:8561035. |
our results suggest that although most patients with temporal lobe epilepsy require hippocampal resection of varying degrees, there is a subset in whom the amygdala may be the crucial element of a mesial temporal epileptogenic network. |
1996-02-26 |
2023-08-12 |
Not clear |
| E H Buhl, T S Otis, I Mod. Zinc-induced collapse of augmented inhibition by GABA in a temporal lobe epilepsy model. Science (New York, N.Y.). vol 271. issue 5247. 1996-02-21. PMID:8553076. |
in the kindling model of temporal lobe epilepsy, several physiological indicators of inhibition by gamma-aminobutyric acid (gaba) in the hippocampal dentate gyrus are consistent with an augmented, rather than a diminished, inhibition. |
1996-02-21 |
2023-08-12 |
human |
| A Pitkänen, T Halone. Prevention of neuronal cell death by anticonvulsants in experimental epilepsy (extended abstract). Acta neurologica Scandinavica. Supplementum. vol 162. 1996-01-11. PMID:7495184. |
in human temporal lobe epilepsy, neuronal cell damage is found in the hippocampus (1) as well as in the amygdaloid complex (2) and entorhinal cortex (3). |
1996-01-11 |
2023-08-12 |
human |
| A Williamson, S S Spencer, D D Spence. Depth electrode studies and intracellular dentate granule cell recordings in temporal lobe epilepsy. Annals of neurology. vol 38. issue 5. 1995-12-28. PMID:7486870. |
hippocampal depth electrodes are often used to localize seizure onset in patients who may have temporal lobe epilepsy (tle). |
1995-12-28 |
2023-08-12 |
Not clear |
| D A Rempe, P S Mangan, E W Lothma. Regional heterogeneity of pathophysiological alterations in CA1 and dentate gyrus in a chronic model of temporal lobe epilepsy. Journal of neurophysiology. vol 74. issue 2. 1995-12-07. PMID:7472385. |
extracellular and intracellular recording techniques were employed in brain slice preparations to characterize responses of hippocampal tissue in the post-self sustaining limbic status epilepticus (post-sslse) model of chronic temporal lobe epilepsy (tle) as compared with responses in slices from control animals. |
1995-12-07 |
2023-08-12 |
Not clear |
| J Niquet, I Jorquera, A Faissner, Y Ben-Ari, A Repres. Gliosis and axonal sprouting in the hippocampus of epileptic rats are associated with an increase of tenascin-C immunoreactivity. Journal of neurocytology. vol 24. issue 8. 1995-12-07. PMID:7595669. |
temporal lobe epilepsy is associated with neuronal death, gliosis and sprouting of mossy fibres in the hippocampus of human and rats. |
1995-12-07 |
2023-08-12 |
human |
| G W Mathern, T L Babb, J K Pretorius, M Melendez, M F Lévesqu. The pathophysiologic relationships between lesion pathology, intracranial ictal EEG onsets, and hippocampal neuron losses in temporal lobe epilepsy. Epilepsy research. vol 21. issue 2. 1995-12-06. PMID:7588588. |
the pathophysiologic relationships between lesion pathology, intracranial ictal eeg onsets, and hippocampal neuron losses in temporal lobe epilepsy. |
1995-12-06 |
2023-08-12 |
Not clear |
| G W Mathern, T L Babb, J K Pretorius, M Melendez, M F Lévesqu. The pathophysiologic relationships between lesion pathology, intracranial ictal EEG onsets, and hippocampal neuron losses in temporal lobe epilepsy. Epilepsy research. vol 21. issue 2. 1995-12-06. PMID:7588588. |
in temporal lobe epilepsy (tle) lesion patients the pathology, location of intracranial ictal eeg onsets, and hippocampal neuron losses were compared. |
1995-12-06 |
2023-08-12 |
Not clear |
| N C de Lanerolle, M Gunel, S Sundaresan, M Y Shen, M L Brines, D D Spence. Vasoactive intestinal polypeptide and its receptor changes in human temporal lobe epilepsy. Brain research. vol 686. issue 2. 1995-11-30. PMID:7583284. |
the distribution of the vip receptor in the human hippocampus was studied by receptor autoradiography using [3-iodotyrosyl-125i]vasoactive intestinal peptide (vip) as a ligand, and the relationship of receptor distribution to the distribution of the peptide (visualized by immunocytochemistry) was examined in hippocampi surgically removed from patients with medically intractable temporal lobe epilepsy (tle) and hippocampi obtained at autopsy from neurologically normal subjects. |
1995-11-30 |
2023-08-12 |
human |
| F Guillem, B N'Kaoua, A Rougier, B Claveri. Effects of temporal versus temporal plus extra-temporal lobe epilepsies on hippocampal ERPs: physiopathological implications for recognition memory studies in humans. Brain research. Cognitive brain research. vol 2. issue 3. 1995-11-29. PMID:7580396. |
hippocampal event-related potentials (erps) were recorded during a recognition memory task for pictures in patients with unilateral temporal lobe epilepsy and in patients with combined temporal and extra-temporal (frontal or parietal) epileptogenic foci. |
1995-11-29 |
2023-08-12 |
Not clear |
| I Blümcke, H K Wolf, P R Hof, J H Morrison, O D Wiestle. Regional distribution of the AMPA glutamate receptor subunits GluR2(4) in human hippocampus. Brain research. vol 682. issue 1-2. 1995-11-09. PMID:7552321. |
these findings may provide basic information for studies of the glur2(4) subunit in human hippocampus during various neuropathological conditions, such as temporal lobe epilepsy, ischemia and alzheimer's disease. |
1995-11-09 |
2023-08-12 |
human |