All Relations between Movement Disorders and basal ganglia

Publication Sentence Publish Date Extraction Date Species
J L Gastaut, F Nicoli, H Somma-Mauvais, F Bartolomei, A Dalecky, M Bruzzo, C Dhiver, J A Gastau. [Hemichorea-hemiballismus and toxoplasmosis in AIDS]. Revue neurologique. vol 148. issue 12. 1993-06-24. PMID:1303576. this rarity seems to be explained by the frequent association of other lesions of the basal ganglia which may prevent the movement disorders. 1993-06-24 2023-08-11 Not clear
F Zipp, H Baas, P A Fische. Lamotrigine--antiparkinsonian activity by blockade of glutamate release? Journal of neural transmission. Parkinson's disease and dementia section. vol 5. issue 1. 1993-03-31. PMID:8094961. these findings are based upon a hypothetical pathway-circuit including the basal ganglia as a model reflecting hypo- and hyperkinetic movement disorders. 1993-03-31 2023-08-12 Not clear
A W Flaherty, A M Graybie. Two input systems for body representations in the primate striatal matrix: experimental evidence in the squirrel monkey. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 13. issue 3. 1993-03-30. PMID:7680067. the striatum is important in basal ganglia motor control and movement disorders. 1993-03-30 2023-08-12 squirrel_monkey
J K Krauss, F Nobbe, A K Wakhloo, M Mohadjer, W Vach, F Mundinge. Movement disorders in astrocytomas of the basal ganglia and the thalamus. Journal of neurology, neurosurgery, and psychiatry. vol 55. issue 12. 1993-02-11. PMID:1479396. movement disorders in astrocytomas of the basal ganglia and the thalamus. 1993-02-11 2023-08-11 Not clear
J K Krauss, F Nobbe, A K Wakhloo, M Mohadjer, W Vach, F Mundinge. Movement disorders in astrocytomas of the basal ganglia and the thalamus. Journal of neurology, neurosurgery, and psychiatry. vol 55. issue 12. 1993-02-11. PMID:1479396. in a series of 225 patients with astrocytomas (grades i-iv) of the basal ganglia and the thalamus, 20 had a movement disorder. 1993-02-11 2023-08-11 Not clear
C G Goetz, E J Papper. Trauma and movement disorders. Neurologic clinics. vol 10. issue 4. 1992-12-09. PMID:1435664. the pathophysiology of movement disorders relates to basal ganglia dysfunction, and traumatic lesions of peripheral nervous system structures or cortical regions could theoretically alter basal ganglia function indirectly and influence movement disorders. 1992-12-09 2023-08-11 Not clear
T N Witt, A Danek, M Reiter, M U Heim, J Dirschinger, E G Olse. McLeod syndrome: a distinct form of neuroacanthocytosis. Report of two cases and literature review with emphasis on neuromuscular manifestations. Journal of neurology. vol 239. issue 6. 1992-09-28. PMID:1512605. the movement disorder suggested accompanying basal ganglia dysfunction. 1992-09-28 2023-08-11 Not clear
A P Auchus, V M Picke. Quantitative light microscopic demonstration of increased pallidal and striatal met5-enkephalin-like immunoreactivity in rats following chronic treatment with haloperidol but not with clozapine: implications for the pathogenesis of neuroleptic-induced movement disorders. Experimental neurology. vol 117. issue 1. 1992-08-04. PMID:1618284. although the neural mechanisms underlying neuroleptic-induced movement disorders remain unknown, alterations in basal ganglia function are likely involved. 1992-08-04 2023-08-11 rat
A P Auchus, V M Picke. Quantitative light microscopic demonstration of increased pallidal and striatal met5-enkephalin-like immunoreactivity in rats following chronic treatment with haloperidol but not with clozapine: implications for the pathogenesis of neuroleptic-induced movement disorders. Experimental neurology. vol 117. issue 1. 1992-08-04. PMID:1618284. these results support the concept that alterations in endogenous opiate peptides in basal ganglia may contribute to movement disorders seen in patients receiving typical neuroleptic drugs. 1992-08-04 2023-08-11 rat
L Fornazzari, M Ichise, G Remington, I Smit. Rabbit syndrome, antidepressant use, and cerebral perfusion SPECT scan findings. Journal of psychiatry & neuroscience : JPN. vol 16. issue 4. 1992-03-24. PMID:1786266. repeated cerebral perfusion spect scans revealed decreased basal ganglia perfusion while the movement disorder was present, and a return to normal perfusion when the rabbit syndrome resolved. 1992-03-24 2023-08-11 rabbit
I J Mitchell, J M Brotchie, G D Brown, A R Crossma. Modeling the functional organization of the basal ganglia. A parallel distributed processing approach. Movement disorders : official journal of the Movement Disorder Society. vol 6. issue 3. 1991-11-07. PMID:1922123. despite recent advances in the understanding of the pathophysiology of movement disorders, little is known about the precise function the basal ganglia play in the control of movement. 1991-11-07 2023-08-11 Not clear
I J Mitchell, J M Brotchie, G D Brown, A R Crossma. Modeling the functional organization of the basal ganglia. A parallel distributed processing approach. Movement disorders : official journal of the Movement Disorder Society. vol 6. issue 3. 1991-11-07. PMID:1922123. we discuss both these approaches and outline a scheme of the functional organization of the basal ganglia, which predicts some of the pathophysiological mechanisms that mediate movement disorders and which can be formally modeled on a computer. 1991-11-07 2023-08-11 Not clear
D T Dexter, A Carayon, F Javoy-Agid, Y Agid, F R Wells, S E Daniel, A J Lees, P Jenner, C D Marsde. Alterations in the levels of iron, ferritin and other trace metals in Parkinson's disease and other neurodegenerative diseases affecting the basal ganglia. Brain : a journal of neurology. vol 114 ( Pt 4). 1991-10-10. PMID:1832073. an increase in total iron content appears to be a response to neurodegeneration in affected basal ganglia regions in a number of movement disorders. 1991-10-10 2023-08-11 human
E R Tonholo-Silva, E M Quagliat. [Spasmodic torticollis and frontal meningioma]. Arquivos de neuro-psiquiatria. vol 49. issue 1. 1991-09-05. PMID:1863233. the association of spasmodic torticollis and other movement disorders has been reported with contralateral lesions in the basal ganglia. 1991-09-05 2023-08-11 Not clear
J S Nagel, M Ichise, B L Holma. The scintigraphic evaluation of Huntington's disease and other movement disorders using single photon emission computed tomography perfusion brain scans. Seminars in nuclear medicine. vol 21. issue 1. 1991-03-25. PMID:1825359. basal ganglia and cortical perfusion changes also have been reported in a few patients with a variety of other less common movement disorders. 1991-03-25 2023-08-11 Not clear
G E Stelmach, J G Phillip. Movement disorders--limb movement and the basal ganglia. Physical therapy. vol 71. issue 1. 1991-01-24. PMID:1824578. movement disorders--limb movement and the basal ganglia. 1991-01-24 2023-08-11 Not clear
D J Brook. Positron emission tomographic studies of the subcortical degenerations and dystonia. Seminars in neurology. vol 9. issue 4. 1990-10-05. PMID:2701780. it is likely that more information will become available about the nature of the functional corrections between cortex, thalamus, and basal ganglia as increasingly sophisticated activation paradigms are designed for pet studies on movement disorders. 1990-10-05 2023-08-11 Not clear
P M FitzGerald, J Jankovic, A K Perc. Rett syndrome and associated movement disorders. Movement disorders : official journal of the Movement Disorder Society. vol 5. issue 3. 1990-09-24. PMID:2388636. this study provides evidence that movement disorders seen in rett syndrome reflect age-related neurodegenerative changes in the basal ganglia. 1990-09-24 2023-08-11 human
M R DeLon. Primate models of movement disorders of basal ganglia origin. Trends in neurosciences. vol 13. issue 7. 1990-08-23. PMID:1695404. primate models of movement disorders of basal ganglia origin. 1990-08-23 2023-08-11 Not clear
M R DeLon. Primate models of movement disorders of basal ganglia origin. Trends in neurosciences. vol 13. issue 7. 1990-08-23. PMID:1695404. movement disorders associated with basal ganglia dysfunction comprise a spectrum of abnormalities that range from the hypokinetic disorders (of which parkinson's disease is the best-known example) at one extreme to the hyperkinetic disorders (exemplified by huntington's disease and hemiballismus) at the other. 1990-08-23 2023-08-11 Not clear