| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| U Müller, K G Kupk. The genetics of primary torsion dystonia. Human genetics. vol 84. issue 2. 1990-02-27. PMID:2404852. |
primary torsion dystonia is an idiopathic movement disorder presumably caused by abnormal function of the basal ganglia. |
1990-02-27 |
2023-08-11 |
Not clear |
| F Barinagarrementeria, F Vega, O H DelBrutt. Acute hemichorea due to infarction in the corona radiata. Journal of neurology. vol 236. issue 6. 1989-11-14. PMID:2795109. |
the movement disorder may have been caused by subcortical lesion without direct involvement of the basal ganglia. |
1989-11-14 |
2023-08-11 |
Not clear |
| P Tinuper, P Montagna, S Laudadio, L Ripamonti, E Lugares. Paroxysmal blinking provoked by head movements. European neurology. vol 29. issue 5. 1989-11-07. PMID:2792153. |
of several hypotheses (brainstem or basal ganglia dysfunction, vestibular origin, and tics), none fully explains this peculiar movement disorder, the origin of which remains unclear. |
1989-11-07 |
2023-08-11 |
Not clear |
| M Menz. A simple model of the interactions of dopamine, acetylcholine and GABA in movement disorders seen in psychiatry. Medical hypotheses. vol 27. issue 4. 1989-04-05. PMID:3226360. |
many of the movement disorders seen in psychiatric patients are determined, in large part, by the dynamic balance of dopamine and acetylcholine in the basal ganglia. |
1989-04-05 |
2023-08-11 |
Not clear |
| J A Wilcox, H Nasralla. Sydenham's chorea and psychopathology. Neuropsychobiology. vol 19. issue 1. 1988-12-06. PMID:3185898. |
sydenham's chorea is a movement disorder seen in rheumatic fever with basal ganglia pathology. |
1988-12-06 |
2023-08-11 |
human |
| P A Kempster, D P Brenton, A N Gale, G M Ster. Dystonia in homocystinuria. Journal of neurology, neurosurgery, and psychiatry. vol 51. issue 6. 1988-09-21. PMID:3404194. |
movement disorder associated with homocystinuria may result from the neurochemical changes in the basal ganglia related to the inherited defect in sulphur amino acid metabolism. |
1988-09-21 |
2023-08-11 |
Not clear |
| R Sandyk, C R Bamfor. The hypothalamus in dystonic movement disorders. The International journal of neuroscience. vol 40. issue 1-2. 1988-08-31. PMID:2840407. |
although dystonic movement disorders have been presumed to originate from dysfunction of the basal ganglia, examination of the brains of patients who have died with idiopathic or hereditary dystonia musculorum deformans have revealed no consistent neuropathological or neurochemical abnormalities (zeman, 1970). |
1988-08-31 |
2023-08-11 |
Not clear |
| J F Marshall, J N Joyc. Basal ganglia dopamine receptor autoradiography and age-related movement disorders. Annals of the New York Academy of Sciences. vol 515. 1988-06-07. PMID:3364885. |
basal ganglia dopamine receptor autoradiography and age-related movement disorders. |
1988-06-07 |
2023-08-11 |
Not clear |
| S I Goodman, F E Frerman, J P Loeh. Recent progress in understanding glutaric acidemias. Enzyme. vol 38. issue 1-4. 1988-04-18. PMID:3126045. |
there may also be a clinical phenotype of glutaric acidemia type ii which, like glutaryl-coa dehydrogenase deficiency, is characterized by a movement disorder and by degeneration of the basal ganglia. |
1988-04-18 |
2023-08-11 |
Not clear |
| A Pauranik, A Verma, M C Maheshwar. Reversible movement disorder in a patient with post traumatic basal ganglia haematoma. Journal of neurology, neurosurgery, and psychiatry. vol 50. issue 8. 1987-11-20. PMID:3655820. |
reversible movement disorder in a patient with post traumatic basal ganglia haematoma. |
1987-11-20 |
2023-08-11 |
Not clear |
| M Segaw. [Disorders of higher motor function: changes in movement disorders with growth--age dependency of clinical findings in essential basal nuclei diseases]. Nihon rinsho. Japanese journal of clinical medicine. vol 45. issue 2. 1987-06-08. PMID:3553668. |
[disorders of higher motor function: changes in movement disorders with growth--age dependency of clinical findings in essential basal nuclei diseases]. |
1987-06-08 |
2023-08-11 |
Not clear |
| M P Merchut, J Brumli. Painful tonic spasms caused by putaminal infarction. Stroke. vol 17. issue 6. 1987-03-09. PMID:3810737. |
lacunar infarcts in the basal ganglia are known to cause various movement disorders, such as chorea, focal dystonia, and hemichorea-hemiballismus. |
1987-03-09 |
2023-08-11 |
Not clear |
| C Reavill, P Jenner, C D Marsde. Gamma-aminobutyric acid and basal ganglia outflow pathways. Ciba Foundation symposium. vol 107. 1985-01-24. PMID:6094124. |
consequently manipulation of gaba action in the outflow zones of the basal ganglia may mimic extrapyramidal movement disorders more closely than the normal functions of these regions of the brain. |
1985-01-24 |
2023-08-12 |
rat |
| J L Cummings, L F Gosenfeld, J P Houlihan, T McCaffre. Neuropsychiatric disturbances associated with idiopathic calcification of the basal ganglia. Biological psychiatry. vol 18. issue 5. 1983-08-26. PMID:6860732. |
a patient with idiopathic calcification of the basal ganglia (icbg) manifested a schizophrenialike psychosis beginning at age 17 and later developed a complex movement disorder and mild dementia. |
1983-08-26 |
2023-08-12 |
Not clear |
| T I Lidsky, T Labuszewski, F M Levin. Are movement disorders the most serious side effects of maintenance therapy with antipsychotic drugs? Biological psychiatry. vol 16. issue 12. 1983-04-07. PMID:6130799. |
movement disorders arise because antipsychotics, in a substantial proportion of patients, disrupt the basal ganglia. |
1983-04-07 |
2023-08-12 |
Not clear |
| K F Swaiman, S A Smith, G L Trock, A R Siddiqu. Sea-blue histiocytes, lymphocytic cytosomes, movement disorder and 59Fe-uptake in basal ganglia: Hallervorden-Spatz disease or ceroid storage disease with abnormal isotope scan? Neurology. vol 33. issue 3. 1983-04-07. PMID:6681873. |
sea-blue histiocytes, lymphocytic cytosomes, movement disorder and 59fe-uptake in basal ganglia: hallervorden-spatz disease or ceroid storage disease with abnormal isotope scan? |
1983-04-07 |
2023-08-12 |
Not clear |
| M S Diamond, Y P Huang, M D Yah. Sudden onset of involuntary movement disorders with arteriovenous malformations of the basal ganglia. The Mount Sinai journal of medicine, New York. vol 49. issue 5. 1983-02-25. PMID:6983658. |
sudden onset of involuntary movement disorders with arteriovenous malformations of the basal ganglia. |
1983-02-25 |
2023-08-12 |
Not clear |
| S M Stahl, K L Davis, P A Berge. The neuropharmacology of tardive dyskinesia, spontaneous dyskinesia, and other dystonias. Journal of clinical psychopharmacology. vol 2. issue 5. 1982-12-18. PMID:6127351. |
this review of movement disorders is organized according to the hypothesis of basal ganglia neurotransmitter imbalance in order to emphasize current research based on the pharmacology of these disorders. |
1982-12-18 |
2023-08-12 |
Not clear |
| H J Manz, A R Colo. Neuropathology, pathogenesis, and neuropsychiatric sequelae of Reye syndrome. Journal of the neurological sciences. vol 53. issue 2. 1982-04-22. PMID:7057216. |
the neuropathologic substrate for the clinical manifestations of mental retardation, recurrent seizures, movement disorders, sensori-motor deficits, psychobiologic maladjustment, and mediocre performance in school and on formal psychometric tests consists of multifocal infarction and astrocytosis in cerebral cortex, diencephalon, basal ganglia, and brainstem. |
1982-04-22 |
2023-08-12 |
Not clear |
| J S Chopra, K Radhakrishnan, R Sridhara. Meige's syndrome: clinical, pharmacological and radiological observations. Clinical neurology and neurosurgery. vol 83. issue 3. 1982-03-13. PMID:6274556. |
neurotransmitter imbalance in the basal ganglia in this disorder remains to be established, and at present there is no satisfactory drug treatment for this progressively disabling movement disorder. |
1982-03-13 |
2023-08-12 |
Not clear |