| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| X Q Zhang, R L Moore, T Tenhave, J Y Cheun. [Ca2+]i transients in hypertensive and postinfarction myocytes. The American journal of physiology. vol 269. issue 3 Pt 1. 1995-11-06. PMID:7573393. |
the observation that isoproterenol lowers diastolic [ca2+]i in hyp myocytes toward normal may provide a cellular mechanism for the lack of efficacy of beta-adrenergic blockers to improve diastolic compliance in patients with hypertensive hypertrophic cardiomyopathy. |
1995-11-06 |
2023-08-12 |
rat |
| I Vugman, A R Han. Quantitative immunocytochemical study of secretory protein expression in parotid glands of rats chronically treated with isoproterenol. Microscopy research and technique. vol 31. issue 2. 1995-10-05. PMID:7544654. |
chronic treatment of mice and rats with isoproterenol (ipr) causes marked hypertrophy and hyperplasia of the salivary glands, and alters the expression of several secretory proteins. |
1995-10-05 |
2023-08-12 |
mouse |
| J Mészáros, B Pászto. Effect of ouabain in catecholamine-induced cardiac hypertrophy. Acta physiologica Hungarica. vol 83. issue 1. 1995-10-02. PMID:7660838. |
hypertrophy was induced by administration of 5 mg/kg isoprenaline once daily for 7 days. |
1995-10-02 |
2023-08-12 |
rat |
| M O Boluyt, X Long, T Eschenhagen, U Mende, W Schmitz, M T Crow, E G Lakatt. Isoproterenol infusion induces alterations in expression of hypertrophy-associated genes in rat heart. The American journal of physiology. vol 269. issue 2 Pt 2. 1995-09-22. PMID:7653628. |
chronic infusion of isoproterenol (iso) in rats results in cardiac hypertrophy via incompletely understood mechanisms. |
1995-09-22 |
2023-08-12 |
rat |
| T A Slotkin, S E Lappi, F J Seidle. Beta-adrenergic control of c-fos expression in fetal and neonatal rat tissues: relationship to cell differentiation and teratogenesis. Toxicology and applied pharmacology. vol 133. issue 2. 1995-09-21. PMID:7544033. |
in keeping with the view that fetal/neonatal beta-adrenergic stimulation of c-fos is related to cell differentiation rather than simply to growth, repeated administration of isoproterenol to neonatal rats did not elicit cardiac hypertrophy, whereas the same treatment did produce hypertrophy in adult rats. |
1995-09-21 |
2023-08-12 |
rat |
| S P Lubic, K M Giacomini, J C Giacomin. The effects of modulation of calcium influx through the voltage-sensitive calcium channel on cardiomyocyte hypertrophy. Journal of molecular and cellular cardiology. vol 27. issue 3. 1995-08-10. PMID:7602609. |
norepinephrine (ne) and, to a lesser degree, isoproterenol (iso) modulated serum-stimulated hypertrophy. |
1995-08-10 |
2023-08-12 |
human |
| T Omura, S Kim, K Takeuchi, H Iwao, T Taked. Transforming growth factor beta 1 and extracellular matrix gene expression in isoprenaline induced cardiac hypertrophy: effects of inhibition of the renin-angiotensin system. Cardiovascular research. vol 28. issue 12. 1995-03-28. PMID:7867037. |
transforming growth factor beta 1 and extracellular matrix gene expression in isoprenaline induced cardiac hypertrophy: effects of inhibition of the renin-angiotensin system. |
1995-03-28 |
2023-08-12 |
Not clear |
| T Omura, S Kim, K Takeuchi, H Iwao, T Taked. Transforming growth factor beta 1 and extracellular matrix gene expression in isoprenaline induced cardiac hypertrophy: effects of inhibition of the renin-angiotensin system. Cardiovascular research. vol 28. issue 12. 1995-03-28. PMID:7867037. |
the aim was to investigate changes in cardiac transforming growth factor beta 1 (tgf-beta 1), fibronectin, and collagen types i and iii mrna levels in isoprenaline induced cardiac hypertrophy, and the effects of delapril, an angiotensin converting enzyme inhibitor, and tcv-116, an angiotensin ii type 1 receptor antagonist, on this hypertrophy. |
1995-03-28 |
2023-08-12 |
Not clear |
| S Kawano, K Iida, K Fujieda, K Yukisada, E S Magdi, Y Iwasaki, F Tabei, I Yamaguchi, Y Sugishit. Response to isoproterenol as a prognostic indicator of evolution from hypertrophic cardiomyopathy to a phase resembling dilated cardiomyopathy. Journal of the American College of Cardiology. vol 25. issue 3. 1995-03-23. PMID:7860914. |
response to isoproterenol as a prognostic indicator of evolution from hypertrophic cardiomyopathy to a phase resembling dilated cardiomyopathy. |
1995-03-23 |
2023-08-12 |
Not clear |
| S Kawano, K Iida, K Fujieda, K Yukisada, E S Magdi, Y Iwasaki, F Tabei, I Yamaguchi, Y Sugishit. Response to isoproterenol as a prognostic indicator of evolution from hypertrophic cardiomyopathy to a phase resembling dilated cardiomyopathy. Journal of the American College of Cardiology. vol 25. issue 3. 1995-03-23. PMID:7860914. |
we sought to assess whether isoproterenol stress echocardiography could detect in advance in which patients hypertrophic cardiomyopathy would progress to a phase resembling dilated cardiomyopathy. |
1995-03-23 |
2023-08-12 |
Not clear |
| M Inoko, Y Kihara, S Sasayam. Neurohumoral factors during transition from left ventricular hypertrophy to failure in Dahl salt-sensitive rats. Biochemical and biophysical research communications. vol 206. issue 3. 1995-02-22. PMID:7832791. |
the pie induced by isoproterenol was reduced at the stage of compensatory lv hypertrophy, which was followed by further reduction of pie at chf stage. |
1995-02-22 |
2023-08-12 |
rat |
| T Minor, W Isselhard, J Stur. [Recovery of healthy and hypertrophied hearts after global ischemia and gradual reperfusion]. Annales de cardiologie et d'angeiologie. vol 43. issue 7. 1995-01-10. PMID:7993034. |
to induce normotensive cardiac hypertrophy, male wistar rats received injections of isoprenaline (5 mg/kg s.c. bid for three days). |
1995-01-10 |
2023-08-12 |
rat |
| P S Sunga, S W Rabki. Angiotensin II induced alteration of cyclic adenosine 3',5'-monophosphate generation in the hypertrophic myocardium of Dahl salt-sensitive rat on a high-salt diet. Canadian journal of physiology and pharmacology. vol 72. issue 6. 1994-12-27. PMID:7525030. |
the objective of this study was to explore the action of angiotensin ii (aii) on cardiac contractility and cyclic amp (camp) generation by forskolin and isoproterenol in the hypertrophic myocardium of the salt-sensitive dahl rat. |
1994-12-27 |
2023-08-12 |
rat |
| P S Sunga, S W Rabki. Angiotensin II induced alteration of cyclic adenosine 3',5'-monophosphate generation in the hypertrophic myocardium of Dahl salt-sensitive rat on a high-salt diet. Canadian journal of physiology and pharmacology. vol 72. issue 6. 1994-12-27. PMID:7525030. |
these data indicate a reversal of control by aii in the hypertrophic dahl s heart in response to forskolin, which activates adenylyl cyclase directly on the catalytic subunit, converting the substrate, atp, to camp, independent of the guanine nucleotide activated protein, and in response to isoproterenol, which activates adenylyl cyclase through g protein mechanisms. |
1994-12-27 |
2023-08-12 |
rat |
| E Golomb, Z A Abassi, G Cuda, M Stylianou, V R Panchal, D Trachewsky, H R Keise. Angiotensin II maintains, but does not mediate, isoproterenol-induced cardiac hypertrophy in rats. The American journal of physiology. vol 267. issue 4 Pt 2. 1994-11-18. PMID:7524366. |
the role of angiotensin ii (ang ii) in the development of isoproterenol (iso)-induced cardiac hypertrophy was examined in rats. |
1994-11-18 |
2023-08-12 |
rat |
| I A Atencio, A Meraz, L P Villarrea. The secretory cells of mouse salivary glands are nonpermissive for polyomavirus replication. Virology. vol 200. issue 2. 1994-06-06. PMID:8178470. |
in addition, isoproterenol and tannin, which induce hypertrophy and hyperplasia of the secretory cells of adult male mice salivary glands, did not make these cells permissive to py replication. |
1994-06-06 |
2023-08-12 |
mouse |
| Y Sugishita, K Iida, K Fujieda, K Yukisad. Decreased adrenergic response in hypertensive patients without left ventricular hypertrophy. Clinical cardiology. vol 17. issue 2. 1994-05-23. PMID:8162629. |
to analyze the adrenergic responses and to compare them between hypertensive patients with and without left ventricular hypertrophy (lvh), left ventricular (lv) fractional shortening (fs) and end-systolic wall stress (ess) were measured by echocardiography and the inotropic response to the infusion of isoproterenol (0.02 micrograms/kg/min for 5 min) was studied in 25 hypertensive patients without lvh [h(-)] and 30 hypertensive patients with lvh [h(+)]. |
1994-05-23 |
2023-08-12 |
human |
| E Harmsen, R Schoemaker, J Yu, M Ruzicka, F H Leene. Sensitivity to ischaemic ATP breakdown in different models of cardiac hypertrophy in rats. Journal of hypertension. vol 12. issue 1. 1994-05-17. PMID:8157944. |
to evaluate the sensitivity to ischaemia of rat hearts made hypertrophic by pressure overload [two-kidney, one clip (2-k,1c) rats], volume overload (aortocaval arteriovenous shunt), minoxidil or isoproterenol. |
1994-05-17 |
2023-08-12 |
rat |
| T Minor, W Isselhard, J Stur. [Recovery of healthy and hypertrophic hearts after global ischemia and graduated reperfusion]. Annales de chirurgie. vol 47. issue 8. 1994-03-15. PMID:8311400. |
to induce normotensive cardiac hypertrophy, male wistar rats received injections of isoprenaline (5 mg/kg s.c. bid for three days). |
1994-03-15 |
2023-08-12 |
rat |
| M Shirota, M Hayashi, Y Kajiwara, K Kitabatake, T Uruno, K Kubot. General pharmacology of the non-sulfhydryl angiotensin converting enzyme inhibitor N-[8-amino-1(S)-carboxyoctyl]-L-alanyl-L-proline. Arzneimittel-Forschung. vol 43. issue 11. 1994-02-24. PMID:8292059. |
inhibited the cardiac hypertrophy induced by isoproterenol (isoprenaline). |
1994-02-24 |
2023-08-12 |
mouse |