| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| L Tebartz Van Elst, D Baeumer, L Lemieux, F G Woermann, M Koepp, S Krishnamoorthy, P J Thompson, D Ebert, M R Trimbl. Amygdala pathology in psychosis of epilepsy: A magnetic resonance imaging study in patients with temporal lobe epilepsy. Brain : a journal of neurology. vol 125. issue Pt 1. 2002-03-01. PMID:11834599. |
in order to test the hypothesis that atrophy of the hippocampus and the amygdala is found in patients with tle and poe, we performed a retrospective study of all patients with tle who were admitted to the assessment unit of the chalfont centre for epilepsy from 1995 until 1999. |
2002-03-01 |
2023-08-12 |
human |
| L Tebartz Van Elst, D Baeumer, L Lemieux, F G Woermann, M Koepp, S Krishnamoorthy, P J Thompson, D Ebert, M R Trimbl. Amygdala pathology in psychosis of epilepsy: A magnetic resonance imaging study in patients with temporal lobe epilepsy. Brain : a journal of neurology. vol 125. issue Pt 1. 2002-03-01. PMID:11834599. |
the finding of amygdala enlargement agrees with the observation of an association between dysphoric disorders of epilepsy and poe described nearly 100 years ago. |
2002-03-01 |
2023-08-12 |
human |
| T Salmenperä, R Kälviäinen, K Partanen, A Pitkäne. Hippocampal and amygdaloid damage in partial epilepsy: a cross-sectional MRI study of 241 patients. Epilepsy research. vol 46. issue 1. 2001-07-19. PMID:11395291. |
magnetic resonance imaging was used to measure the volumes of the hippocampus and the amygdala in 241 patients with different durations of epilepsy. |
2001-07-19 |
2023-08-12 |
Not clear |
| H Potschka, E Friderichs, W Lösche. Anticonvulsant and proconvulsant effects of tramadol, its enantiomers and its M1 metabolite in the rat kindling model of epilepsy. British journal of pharmacology. vol 131. issue 2. 2001-02-22. PMID:10991912. |
in the present study, we evaluated (+/-)-tramadol, its enantiomers, and its m1 metabolite ((+)-enantiomer) in the amygdala kindling model of epilepsy in rats. |
2001-02-22 |
2023-08-12 |
rat |
| P S Mangan, C A Scott, J M Williamson, E H Bertra. Aberrant neuronal physiology in the basal nucleus of the amygdala in a model of chronic limbic epilepsy. Neuroscience. vol 101. issue 2. 2001-02-01. PMID:11074161. |
aberrant neuronal physiology in the basal nucleus of the amygdala in a model of chronic limbic epilepsy. |
2001-02-01 |
2023-08-12 |
rat |
| P S Mangan, C A Scott, J M Williamson, E H Bertra. Aberrant neuronal physiology in the basal nucleus of the amygdala in a model of chronic limbic epilepsy. Neuroscience. vol 101. issue 2. 2001-02-01. PMID:11074161. |
this study used in vitro intracellular recording and histology to examine alterations to the physiology and anatomy of the basal nucleus of the amygdala in a rat model of chronic limbic epilepsy characterized by spontaneously recurring seizures. |
2001-02-01 |
2023-08-12 |
rat |
| A L Velasco, M Velasco, F Velasco, D Menes, F Gordon, L Rocha, M Briones, I Márque. Subacute and chronic electrical stimulation of the hippocampus on intractable temporal lobe seizures: preliminary report. Archives of medical research. vol 31. issue 3. 2000-12-22. PMID:11036183. |
recent animal experiments show that the application of an electrical stimulus to the amygdala or hippocampus following the kindling stimulus produced a significant and long-lasting suppressive effect on this experimental model of epilepsy. |
2000-12-22 |
2023-08-12 |
Not clear |
| H G Wiese. Mesial temporal lobe epilepsy versus amygdalar epilepsy: late seizure recurrence after initially successful amygdalotomy and regained seizure control following hippocampectomy. Epileptic disorders : international epilepsy journal with videotape. vol 2. issue 3. 2000-12-05. PMID:11022139. |
we summarise the concept of mesial temporal lobe epilepsy and the pros and cons in order to define amygdala epilepsy. |
2000-12-05 |
2023-08-12 |
Not clear |
| H G Wiese. Mesial temporal lobe epilepsy versus amygdalar epilepsy: late seizure recurrence after initially successful amygdalotomy and regained seizure control following hippocampectomy. Epileptic disorders : international epilepsy journal with videotape. vol 2. issue 3. 2000-12-05. PMID:11022139. |
this case underscores the important role of the amygdala in generating the semiology, and raises several questions concerning the existence of "amygdalar epilepsy". |
2000-12-05 |
2023-08-12 |
Not clear |
| X Zou, X Ruan, Y Wei, C Zh. [Alterations of substance P-immune reaction positive neurons of cerebral tissues in epileptic rats]. Hua xi yi ke da xue xue bao = Journal of West China University of Medical Sciences = Huaxi yike daxue xuebao. vol 28. issue 2. 2000-03-28. PMID:10683920. |
employing the immunocytochemical analysis, we observed the alterations of substance p-immune reaction (sp-ir) positive neurons of the cerebral cortex, hippocampus and amygdala in rats suffering from epilepsy induced by penicillin (pen). |
2000-03-28 |
2023-08-12 |
rat |
| M Thom, B Griffin, J W Sander, F Scaravill. Amygdala sclerosis in sudden and unexpected death in epilepsy. Epilepsy research. vol 37. issue 1. 1999-11-22. PMID:10515175. |
amygdala sclerosis in sudden and unexpected death in epilepsy. |
1999-11-22 |
2023-08-12 |
Not clear |
| M Thom, B Griffin, J W Sander, F Scaravill. Amygdala sclerosis in sudden and unexpected death in epilepsy. Epilepsy research. vol 37. issue 1. 1999-11-22. PMID:10515175. |
sclerosis of the amygdala is a not uncommon finding in patients with chronic epilepsy. |
1999-11-22 |
2023-08-12 |
Not clear |
| M Thom, B Griffin, J W Sander, F Scaravill. Amygdala sclerosis in sudden and unexpected death in epilepsy. Epilepsy research. vol 37. issue 1. 1999-11-22. PMID:10515175. |
these patterns of cell loss in the amygdala do not differ from previous studies in both humans and animal models of chronic epilepsy suggesting that there is not a specific pattern of amygdaloid sclerosis in sudep patients which could implicate a functional role for this nucleus in the mechanism of the sudden death. |
1999-11-22 |
2023-08-12 |
Not clear |
| J Mellanby, M Oliva, A Peniket, B Nicholl. The effect of experimental epilepsy induced by injection of tetanus toxin into the amygdala of the rat on eating behaviour and response to novelty. Behavioural brain research. vol 100. issue 1-2. 1999-07-28. PMID:10212058. |
the effect of experimental epilepsy induced by injection of tetanus toxin into the amygdala of the rat on eating behaviour and response to novelty. |
1999-07-28 |
2023-08-12 |
rat |
| K Elisevich, S A Rempel, B Smith, K Hirs. Temporal profile of connexin 43 mRNA expression in a tetanus toxin-induced seizure disorder. Molecular and chemical neuropathology. vol 35. issue 1-3. 1999-07-26. PMID:10343969. |
the messenger ribonucleic acid (mrna) of gap junction protein connexin 43 was quantified in the tetanus toxin rat model of focal epilepsy following injection of toxin into the left amygdala. |
1999-07-26 |
2023-08-12 |
rat |
| A Matagne, H Klitgaar. Validation of corneally kindled mice: a sensitive screening model for partial epilepsy in man. Epilepsy research. vol 31. issue 1. 1999-05-19. PMID:9696301. |
the seizure protection and adverse effect potential obtained with proven antiepileptic drugs were similar to the effects observed in amygdala kindled rats and their corresponding clinical profile in partial epilepsy. |
1999-05-19 |
2023-08-12 |
mouse |
| A Cleton, R A Voskuyl, M Danho. Adaptive changes in the pharmacodynamics of midazolam in different experimental models of epilepsy: kindling, cortical stimulation and genetic absence epilepsy. British journal of pharmacology. vol 125. issue 4. 1999-03-25. PMID:9831893. |
for that purpose the pharmacodynamics of midazolam were quantified by an integrated pharmacokinetic-pharmacodynamic approach in three different models of experimental epilepsy: amygdala kindling, cortical stimulation and genetic absence epilepsy. |
1999-03-25 |
2023-08-12 |
rat |
| b' A Pitk\\xc3\\xa4nen, J Tuunanen, R K\\xc3\\xa4lvi\\xc3\\xa4inen, K Partanen, T Salmenper\\xc3\\xa. Amygdala damage in experimental and human temporal lobe epilepsy. Epilepsy research. vol 32. issue 1-2. 1998-12-29. PMID:9761324.' |
the information gathered so far on damage to the amygdala in epilepsy or after status epilepticus suggests that local alterations in inhibitory circuitries may contribute to a lowered seizure threshold and greater excitability within the amygdala. |
1998-12-29 |
2023-08-12 |
human |
| P DE GIACOM. [A hypothesis based on histochemical data regarding the site from which originate the automatic movements present in rhinencephalic epilepsy and an added note concerning the distribution of cholinesterase activity in the amygdaloid complex of man]. Rivista di neurologia. vol 31. 1998-11-01. PMID:13884201. |
[a hypothesis based on histochemical data regarding the site from which originate the automatic movements present in rhinencephalic epilepsy and an added note concerning the distribution of cholinesterase activity in the amygdaloid complex of man]. |
1998-11-01 |
2023-08-12 |
Not clear |
| K Morimoto, K Sato, S Sato, S Suemaru, T Sato, N Yamada, T Hayabar. Increases in mRNA levels for synapsin I but not synapsin II in the hippocampus of the rat kindling model of epilepsy. Seizure. vol 7. issue 3. 1998-10-30. PMID:9700837. |
we studied brain synapsin i and ii mrna levels using the amygdala kindling model of epilepsy. |
1998-10-30 |
2023-08-12 |
rat |