All Relations between amygdala and Epilepsy

Publication Sentence Publish Date Extraction Date Species
M Simonato, R Molteni, G Bregola, A Muzzolini, M Piffanelli, L Beani, G Racagni, M Riv. Different patterns of induction of FGF-2, FGF-1 and BDNF mRNAs during kindling epileptogenesis in the rat. The European journal of neuroscience. vol 10. issue 3. 1998-10-22. PMID:9753162. we used the rnaase protection assay to investigate the expression patterns of some ntfs during amygdala kindling, an animal model of epilepsy in which 'pathological' neuronal plasticity appears to occur. 1998-10-22 2023-08-12 rat
Z Toth, X X Yan, S Haftoglou, C E Ribak, T Z Bara. Seizure-induced neuronal injury: vulnerability to febrile seizures in an immature rat model. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 18. issue 11. 1998-06-04. PMID:9592105. whether they induce death of hippocampal and amygdala neurons and consequent limbic (temporal lobe) epilepsy has remained controversial, with conflicting data from prospective and retrospective studies. 1998-06-04 2023-08-12 rat
W Löscher, U Ebert, H Lehmann, C Rosenthal, G Nikkha. Seizure suppression in kindling epilepsy by grafts of fetal GABAergic neurons in rat substantia nigra. Journal of neuroscience research. vol 51. issue 2. 1998-03-30. PMID:9469573. we previously showed that amygdala kindling results in a significant decrease of gaba and its synthesizing enzyme glutamate decarboxylase in substantia nigra (sn), i.e., a region thought to be critically involved in seizure propagation in various models of epilepsy. 1998-03-30 2023-08-12 rat
V Neugebauer, N B Keele, P Shinnick-Gallaghe. Loss of long-lasting potentiation mediated by group III mGluRs in amygdala neurons in kindling-induced epileptogenesis. Journal of neurophysiology. vol 78. issue 6. 1998-02-05. PMID:9405564. long-lasting modifications of synaptic transmission can be induced in the amygdala by electrical stimulation as done in the long-term potentiation (ltp) model of learning and memory and the kindling model of epilepsy. 1998-02-05 2023-08-12 rat
H K Wolf, A F Aliashkevich, I Blümcke, O D Wiestler, J Zentne. Neuronal loss and gliosis of the amygdaloid nucleus in temporal lobe epilepsy. A quantitative analysis of 70 surgical specimens. Acta neuropathologica. vol 93. issue 6. 1997-08-05. PMID:9194900. although clinical and electrophysiological evidence indicates that the amygdaloid body plays an important role in the pathogenesis of temporal lobe epilepsy, there are very few detailed data on histopathological changes in this nucleus in epilepsy patients. 1997-08-05 2023-08-12 Not clear
B N Smith, F E Dude. Enhanced population responses in the basolateral amygdala of kainate-treated, epileptic rats in vitro. Neuroscience letters. vol 222. issue 1. 1997-04-22. PMID:9121710. several lines of evidence suggest that the amygdala is altered functionally in humans with temporal lobe epilepsy, but little is known about neuronal interactions in the amygdala of humans or animals with chronic epilepsy. 1997-04-22 2023-08-12 rat
K H Holmes, N B Keele, P Shinnick-Gallaghe. Loss of mGluR-mediated hyperpolarizations and increase in mGluR depolarizations in basolateral amygdala neurons in kindling-induced epilepsy. Journal of neurophysiology. vol 76. issue 4. 1997-03-11. PMID:8899650. loss of mglur-mediated hyperpolarizations and increase in mglur depolarizations in basolateral amygdala neurons in kindling-induced epilepsy. 1997-03-11 2023-08-12 rat
B Van Swede. Sleep and the temporal lobe. Acta neurologica Belgica. vol 96. issue 1. 1996-08-05. PMID:8669224. experimental amygdala kindling, an animal epilepsy model involving temporal structures, induces disturbed sleep patterns favouring waking and light sleep. 1996-08-05 2023-08-12 Not clear
K Tanaka, K Hirayama, R Murata, S Matsuur. Relation of the enhancement of entorhinal tetanic responses by 50-Hz amygdala stimulation to the progression of kindling in the rat. Neuroscience research. vol 23. issue 3. 1996-02-14. PMID:8545073. we concluded from the results that the enhancement of facilitation of the excitatory synaptic activation and the reduction of the inhibitory synaptic activation in entorhinal tetanic responses by 50-hz amygdala kindling stimulation is involved in the electrophysiological source of the progression of kindling epilepsy. 1996-02-14 2023-08-12 rat
P Bo, D Soragna, E Marchioni, E Candeloro, A Albergati, F Savold. Role of dopamine D-1 and D-2 antagonists in a model of focal epilepsy induced by electrical stimulation of hippocampus and amygdala in the rabbit. Progress in neuro-psychopharmacology & biological psychiatry. vol 19. issue 5. 1996-02-07. PMID:8539428. role of dopamine d-1 and d-2 antagonists in a model of focal epilepsy induced by electrical stimulation of hippocampus and amygdala in the rabbit. 1996-02-07 2023-08-12 rabbit
A Saukkonen, R Kälviäinen, K Partanen, P Vainio, P Riekkinen, A Pitkäne. Do seizures cause neuronal damage? A MRI study in newly diagnosed and chronic epilepsy. Neuroreport. vol 6. issue 1. 1995-05-09. PMID:7703420. we approached this question by measuring the volumes of the amygdala, hippocampus and parahippocampal gyrus with magnetic resonance imaging in 15 controls, 13 unmedicated patients with newly diagnosed epilepsy and 16 patients with chronic drug-refractory epilepsy. 1995-05-09 2023-08-12 human
A Gambardella, J Gotman, F Cendes, F Anderman. The relation of spike foci and of clinical seizure characteristics to different patterns of mesial temporal atrophy. Archives of neurology. vol 52. issue 3. 1995-03-24. PMID:7872883. these results explain some of the variability in the clinical and electrographic manifestations of temporal epilepsy and outline the specific role of amygdala involvement in addition to the commonly reported hippocampal atrophy. 1995-03-24 2023-08-12 Not clear
D D Spencer, S S Spence. Hippocampal resections and the use of human tissue in defining temporal lobe epilepsy syndromes. Hippocampus. vol 4. issue 3. 1995-03-03. PMID:7842044. through the continued evolution of surgical feedback to our preoperative localization studies, and from our basic science studies on resected tissue, we propose that 1) the hippocampus is critical to developing medial temporal lobe excitability, and that the inhibitory hilar interneurons which suffer most during developmental injury are likely to be involved in this process; 2) the hippocampus is not the only potentially hyperexcitable medial temporal lobe structure, but seems to function (both normally and pathologically) in a recurrent loop consisting of the entorhinal cortex, amygdala, and temporal neocortices; and 3) hypotheses derived from animal model studies, and data obtained directly from analysis of human tissue can be used to refine epilepsy classifications. 1995-03-03 2023-08-12 human
P Bo, D Soragna, R Murelli, A Albergati, F Savold. [Role of the dopaminergic system in experimental models of epilepsy]. Bollettino della Societa italiana di biologia sperimentale. vol 69. issue 6. 1994-05-09. PMID:8148116. the aim of this work was to investigate whether the d-1 antagonist sch 23390 differs from haloperidol (d-2 antagonist) in models of experimental epilepsy induced by electrical stimulation of selected brain regions (hippocampus and amygdala), in rabbits. 1994-05-09 2023-08-12 Not clear
N Mor. Roles of excitatory amino acids in expression of secondarily generalized limbic seizures: observations in the kindling model of epilepsy. Fukushima journal of medical science. vol 39. issue 1. 1994-04-13. PMID:7907308. amygdaloid kindling, in which repeated application of low intensity electrical stimulation to the amygdala at appropriate intervals results in the progressive development of generalized convulsive seizures, is an animal model of complex partial epilepsy. 1994-04-13 2023-08-12 Not clear
S Matsuura, K Hirayama, R Murat. Enhancement of synaptic facilitation during the progression of kindling epilepsy by amygdala stimulations. Journal of neurophysiology. vol 70. issue 2. 1993-11-08. PMID:8410160. enhancement of synaptic facilitation during the progression of kindling epilepsy by amygdala stimulations. 1993-11-08 2023-08-12 rat
B S Meldrum, M D Craggs, N Dürmüller, S E Smith, A G Chapma. The effects of AMPA receptor antagonists on kindled seizures and on reflex epilepsy in rodents and primates. Epilepsy research. Supplement. vol 9. 1993-03-15. PMID:1337444. two potent glutamate antagonists, nbqx and gyki 52466, that act selectively on non-nmda receptors, have been tested for anticonvulsant activity in 3 models of reflex epilepsy (sound-induced seizures in dba/2 mice and in genetically epilepsy-prone rats and photically-induced myoclonus in papio papio) and in amygdala kindled rats. 1993-03-15 2023-08-11 mouse
B S Meldrum, M D Craggs, N Dürmüller, S E Smith, A G Chapma. The effects of AMPA receptor antagonists on kindled seizures and on reflex epilepsy in rodents and primates. Epilepsy research. Supplement. vol 9. 1993-03-15. PMID:1337444. gyki 52466 also reduces behavioral seizures and afterdischarge duration in amygdala kindled rats, but with a lower potency than it suppresses reflex epilepsy. 1993-03-15 2023-08-11 mouse
H J Beldhuis, J M Koolhaas, B Bohu. Effect of different agonistic experiences on behavioural seizures in fully amygdala kindled rats. Neuroscience letters. vol 141. issue 1. 1992-09-22. PMID:1508389. fully amygdala kindled rats were exposed to two different inter-male agonistic experiences in order to study the interaction between epilepsy and acute social stress. 1992-09-22 2023-08-11 rat
S A Nieminen, J Sirviö, K Teittinen, A Pitkänen, M M Airaksinen, P Riekkine. Amygdala kindling increased fear-response, but did not impair spatial memory in rats. Physiology & behavior. vol 51. issue 4. 1992-07-01. PMID:1594684. the behavioral effects of amygdala kindling, a model of experimental epilepsy in rats, are reported. 1992-07-01 2023-08-11 rat