| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Karlene T Barrett, Ryan T Dosumu-Johnson, J Andrew Daubenspeck, Rachael D Brust, Vasileios Kreouzis, Jun Chul Kim, Aihua Li, Susan M Dymecki, Eugene E Natti. Partial Raphe Dysfunction in Neurotransmission Is Sufficient to Increase Mortality after Anoxic Exposures in Mice at a Critical Period in Postnatal Development. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 36. issue 14. 2016-08-15. PMID:27053202. |
sudden infant death syndrome (sids) cases often have abnormalities of the brainstem raphe serotonergic (5-ht) system. |
2016-08-15 |
2023-08-13 |
mouse |
| B J Dougherty, E J Gonzalez-Rothi, K Z Lee, H H Ross, P J Reier, D D Fulle. Respiratory outcomes after mid-cervical transplantation of embryonic medullary cells in rats with cervical spinal cord injury. Experimental neurology. vol 278. 2016-07-14. PMID:26808660. |
we hypothesized that intraspinal transplantation of embryonic midline brainstem (mb) cells rich in serotonergic raphé neurons would improve respiratory outcomes after csci. |
2016-07-14 |
2023-08-13 |
rat |
| Petr Šilhán, Monika Jelínková, Uwe Walter, Ján Pavlov Praško, Roman Herzig, Kateřina Langová, David Školoudí. Transcranial sonography of brainstem structures in panic disorder. Psychiatry research. vol 234. issue 1. 2016-06-27. PMID:26371456. |
patients with panic disorder exhibit changes of brainstem raphe on tcs suggesting an alteration of the central serotonergic system. |
2016-06-27 |
2023-08-13 |
human |
| Maria Cristina Greco, Pierluigi Navarra, Giuseppe Tringal. The analgesic agent tapentadol inhibits calcitonin gene-related peptide release from isolated rat brainstem via a serotonergic mechanism. Life sciences. vol 145. 2016-06-13. PMID:26706288. |
the analgesic agent tapentadol inhibits calcitonin gene-related peptide release from isolated rat brainstem via a serotonergic mechanism. |
2016-06-13 |
2023-08-13 |
rat |
| Takashi Matsui, Shingo Soya, Kentaro Kawanaka, Hideaki Soy. Brain Glycogen Decreases During Intense Exercise Without Hypoglycemia: The Possible Involvement of Serotonin. Neurochemical research. vol 40. issue 7. 2016-04-11. PMID:26037553. |
activated serotonergic regulation is a possible underlying mechanism for intense exercise-induced glycogenolysis at least in the hippocampus and brainstem. |
2016-04-11 |
2023-08-13 |
rat |
| Eunkyung Park, Yu Mi Hwang, Min Kyung Chu, Ki-Young Jun. Increased Brainstem Serotonergic Transporter Availability in Adult Migraineurs: an [(18)F]FP-CIT PET Imaging Pilot Study. Nuclear medicine and molecular imaging. vol 50. issue 1. 2016-03-04. PMID:26941862. |
increased brainstem serotonergic transporter availability in adult migraineurs: an [(18)f]fp-cit pet imaging pilot study. |
2016-03-04 |
2023-08-13 |
Not clear |
| Janina Deusser, Stefanie Schmidt, Benjamin Ettle, Sonja Plötz, Sabine Huber, Christian P Müller, Eliezer Masliah, Jürgen Winkler, Zacharias Koh. Serotonergic dysfunction in the A53T alpha-synuclein mouse model of Parkinson's disease. Journal of neurochemistry. vol 135. issue 3. 2016-02-10. PMID:26201615. |
we detected human α-synuclein in the perikarya of brainstem median and dorsal raphe neurons as well as in serotonergic fibers in the hippocampus. |
2016-02-10 |
2023-08-13 |
mouse |
| Dario Arnaldi, Francesco Famà, Fabrizio De Carli, Silvia Morbelli, Michela Ferrara, Agnese Picco, Jennifer Accardo, Alberto Primavera, Gianmario Sambuceti, Flavio Nobil. The Role of the Serotonergic System in REM Sleep Behavior Disorder. Sleep. vol 38. issue 9. 2016-01-14. PMID:25845692. |
we aimed to study brain stem serotonergic system integrity, by means of (123)i-fp-cit-spect, in a group of irbd patients as compared to normal subjects. |
2016-01-14 |
2023-08-13 |
human |
| Hector Mateo-Carrasco, Eva María Muñoz-Aguilera, Juan Manuel García-Torrecillas, Hiba Abu Al-Rob. Serotonin syndrome probably triggered by a morphine-phenelzine interaction. Pharmacotherapy. vol 35. issue 6. 2016-01-04. PMID:25903219. |
serotonin syndrome is a potentially life-threatening condition caused by excessive central and peripheral stimulation of serotonin brainstem receptors, usually triggered by inadvertent interactions between agents with serotonergic activity. |
2016-01-04 |
2023-08-13 |
Not clear |
| Zahi Qamhawi, David Towey, Bina Shah, Gennaro Pagano, John Seibyl, Kenneth Marek, Per Borghammer, David James Brooks, Nicola Paves. Clinical correlates of raphe serotonergic dysfunction in early Parkinson's disease. Brain : a journal of neurology. vol 138. issue Pt 10. 2015-12-28. PMID:26209314. |
post-mortem and neuroimaging studies suggest that the serotonergic system, which originates from the brainstem raphe nuclei, is disrupted in parkinson's disease. |
2015-12-28 |
2023-08-13 |
human |
| Patrice G Guyenet, Douglas A Baylis. Neural Control of Breathing and CO2 Homeostasis. Neuron. vol 87. issue 5. 2015-11-23. PMID:26335642. |
specific brainstem neurons (e.g., retrotrapezoid nucleus, rtn; serotonergic) are activated by pco2 and stimulate breathing. |
2015-11-23 |
2023-08-13 |
Not clear |
| Blair A Johnston, Serenella Tolomeo, Victoria Gradin, David Christmas, Keith Matthews, J Douglas Steel. Failure of hippocampal deactivation during loss events in treatment-resistant depression. Brain : a journal of neurology. vol 138. issue Pt 9. 2015-11-16. PMID:26133661. |
we conclude that the neuroimaging findings were largely consistent with deaken and graeff's predictions, abnormally increased hippocampal activity during loss events was an especially consistent abnormality, and brainstem serotonergic nuclei merit further study in depressive illness. |
2015-11-16 |
2023-08-13 |
human |
| H E Reinebrant, J A Wixey, K M Bulle. Neonatal hypoxia-ischaemia disrupts descending neural inputs to dorsal raphé nuclei. Neuroscience. vol 248. 2015-11-03. PMID:23806712. |
nonetheless the mechanisms contributing to losses of serotonergic neurons in the brainstem remain to be elucidated. |
2015-11-03 |
2023-08-12 |
rat |
| H E Reinebrant, J A Wixey, K M Bulle. Neonatal hypoxia-ischaemia disrupts descending neural inputs to dorsal raphé nuclei. Neuroscience. vol 248. 2015-11-03. PMID:23806712. |
since losses of descending inputs are associated with decreases in serotonergic neurons in the brainstem raphé nuclei, we propose that disruption of certain descending neural inputs from the forebrain to the dr dorsal and the dr ventral nuclei may contribute to losses of serotonergic neurons after p3 hi. |
2015-11-03 |
2023-08-12 |
rat |
| Raffaele Nardone, Yvonne Höller, Aljoscha Thomschewski, Peter Höller, Piergiorgio Lochner, Stefan Golaszewski, Francesco Brigo, Eugen Trink. Serotonergic transmission after spinal cord injury. Journal of neural transmission (Vienna, Austria : 1996). vol 122. issue 2. 2015-09-25. PMID:24866695. |
moreover, emerging evidence implicates serotonergic descending facilitatory pathways from the brainstem to the spinal cord in the maintenance of pathologic pain. |
2015-09-25 |
2023-08-13 |
Not clear |
| Raffaele Nardone, Yvonne Höller, Aljoscha Thomschewski, Peter Höller, Piergiorgio Lochner, Stefan Golaszewski, Francesco Brigo, Eugen Trink. Serotonergic transmission after spinal cord injury. Journal of neural transmission (Vienna, Austria : 1996). vol 122. issue 2. 2015-09-25. PMID:24866695. |
there are functional relevant connections between the descending serotonergic system from the rostral ventromedial medulla in the brainstem, the 5-ht receptors in the spinal dorsal horn, and the descending pain facilitation after tissue and nerve injury. |
2015-09-25 |
2023-08-13 |
Not clear |
| Mousumi Ghosh, Damien D Pears. The role of the serotonergic system in locomotor recovery after spinal cord injury. Frontiers in neural circuits. vol 8. 2015-07-30. PMID:25709569. |
another approach has been to employ cell therapeutics to replace the loss of descending serotonergic input to the cpg, either through transplanted fetal brainstem 5-ht neurons at the site of injury that can supply 5-ht to below the level of the lesion or by other cell types to provide a substrate at the injury site for encouraging serotonergic axon regrowth across the lesion to the caudal spinal cord for restoring locomotion. |
2015-07-30 |
2023-08-13 |
Not clear |
| Evan D Paul, Philip L Johnson, Anantha Shekhar, Christopher A Lowr. The Deakin/Graeff hypothesis: focus on serotonergic inhibition of panic. Neuroscience and biobehavioral reviews. vol 46 Pt 3. 2015-07-17. PMID:24661986. |
the deakin/graeff hypothesis proposes that different subpopulations of serotonergic neurons through topographically organized projections to forebrain and brainstem structures modulate the response to acute and chronic stressors, and that dysfunction of these neurons increases vulnerability to affective and anxiety disorders, including panic disorder. |
2015-07-17 |
2023-08-12 |
Not clear |
| Giampaolo Perna, Giuseppe Guerriero, Paolo Brambilla, Daniela Caldirol. Panic and the brainstem: clues from neuroimaging studies. CNS & neurological disorders drug targets. vol 13. issue 6. 2015-05-19. PMID:24923341. |
however, preliminary evidence of a role of the brainstem in pd can be found and, secondly, the brainstem serotonergic system seems to be involved in panic modulation with indications of both altered serotonergic receptors and 5-ht transporter bindings. |
2015-05-19 |
2023-08-13 |
Not clear |
| Giampaolo Perna, Giuseppe Guerriero, Paolo Brambilla, Daniela Caldirol. Panic and the brainstem: clues from neuroimaging studies. CNS & neurological disorders drug targets. vol 13. issue 6. 2015-05-19. PMID:24923341. |
in conclusion, our review suggests that the brainstem may be involved in psychopathology of pd and supports the relevant role of subcortical serotonergic system in panic pathogenesis. |
2015-05-19 |
2023-08-13 |
Not clear |