All Relations between ca1 field and isocortex

Publication Sentence Publish Date Extraction Date Species
J W Shar. PCP and ketamine inhibit non-NMDA glutamate receptor mediated hsp70 induction. Brain research. vol 728. issue 2. 1997-01-15. PMID:8864485. kainate receptors probably mediate excitotoxicity via direct calcium conductance with glutamate release being important in the ca1 area and neocortex. 1997-01-15 2023-08-12 Not clear
R Schmidt-Kastner, C Humpel, C Wetmore, L Olso. Cellular hybridization for BDNF, trkB, and NGF mRNAs and BDNF-immunoreactivity in rat forebrain after pilocarpine-induced status epilepticus. Experimental brain research. vol 107. issue 3. 1996-10-30. PMID:8821376. bdnf mrna increased in hippocampus, neocortex, piriform cortex, striatum, and thalamus with a maximum at 3-6 h. hybridization levels increased earlier in the resistant granule and ca1 cells as compared to the vulnerable ca3 neurons. 1996-10-30 2023-08-12 rat
D I Kerr, J Ong, D J Doolette, K Schafer, R H Prage. The (S)-enantiomer of 2-hydroxysaclofen is the active GABAB receptor antagonist in central and peripheral preparations. European journal of pharmacology. vol 287. issue 2. 1996-10-24. PMID:8749034. the depression of excitatory field potentials by baclofen ( 5 mu m) in rat ca1 hippocampal slices was antagonised by (s)-2-hydroxysaclofen (100 mu m) (pa2 = 4.3), whilst in rat neocortex, (s)-2-hyroxysaclofen (50-500 mu m) antagonised the baclofen (10 mu m)-induced suppression of spontaneous discharges, the (r)-enantiomer being inactive. 1996-10-24 2023-08-12 rat
V R Heale, K Petersen, C H Vanderwol. Effect of colchicine-induced cell loss in the dentate gyrus and Ammon's horn on the olfactory control of feeding in rats. Brain research. vol 712. issue 2. 1996-10-04. PMID:8814895. lesions of the dentate gyrus and ca1 (induced by local injections of colchicine) and/or the neocortex overlying the hippocampus produce a complex pattern of changes in these normal olfactory reactions, but do not affect the normal reaction to food flavored with sucrose or quinine. 1996-10-04 2023-08-12 rat
P Giannakopoulos, P R Hof, P G Vallet, A S Giannakopoulos, Y Charnay, C Boura. Quantitative analysis of neuropathologic changes in the cerebral cortex of centenarians. Progress in neuro-psychopharmacology & biological psychiatry. vol 19. issue 4. 1996-03-22. PMID:8588057. furthermore, they indicate that both the neurofibrillary tangle densities in the ca1 field and senile plaque densities in the superficial layers of the neocortex must be considered for the neuropathological diagnosis of alzheimer's disease in this age group. 1996-03-22 2023-08-12 Not clear
B J Alps, C Calder, A D Wilson, D E McBean, J M Armstron. Reduction by lifarizine of the neuronal damage induced by cerebral ischaemia in rodents. British journal of pharmacology. vol 115. issue 8. 1996-03-04. PMID:8564203. in rats, the protective effect of lifarizine was regionally and cumulatively assessed in six brain regions (anterior and posterior neocortex, hippocampal ca1 subfield, thalamus, striatum, cerebellar purkinje cells-brain stem) at each dose level. 1996-03-04 2023-08-12 rat
C Largo, P Cuevas, G G Somjen, R Martín del Río, O Herrera. The effect of depressing glial function in rat brain in situ on ion homeostasis, synaptic transmission, and neuron survival. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 16. issue 3. 1996-02-28. PMID:8558250. after 4 hr, spreading depression (sd) waves that originated from the neocortex invaded hippocampal ca1, [k+]o increased to higher levels, pho became very acid, and there were steep increases in taurine, glutamate, and gaba levels. 1996-02-28 2023-08-12 rat
K Shimoda, H Ikeshima, K Matsuo, J Hata, K Maejima, T Takan. Spatial and temporal regulation of the rat calmodulin gene III directed by a 877-base promoter and 103-base leader segment in the mature and embryonal central nervous system of transgenic mice. Brain research. Molecular brain research. vol 31. issue 1-2. 1995-12-07. PMID:7476034. the transgene was expressed prominently in pyramidal cells of the cerebral neocortex and the hippocampal regions ca1 to ca3, in purkinje cells of the cerebellar cortex, and in neurons of the spinal cord, and moderately in granule cells of the dentate gyrus and the cerebellar cortex. 1995-12-07 2023-08-12 mouse
N J Sucher, S Akbarian, C L Chi, C L Leclerc, M Awobuluyi, D L Deitcher, M K Wu, J P Yuan, E G Jones, S A Lipto. Developmental and regional expression pattern of a novel NMDA receptor-like subunit (NMDAR-L) in the rodent brain. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 15. issue 10. 1995-12-04. PMID:7472413. on p14, nmdar-l mrna was expressed in layers ii-vi of the neocortex, in the entorhinal and piriform cortex, in the subiculum and ca1 field, and in the nucleus of the lateral olfactory tract. 1995-12-04 2023-08-12 xenopus_laevis
D S Warner, H Martin, P Ludwig, A McAllister, J F Keana, E Webe. In vivo models of cerebral ischemia: effects of parenterally administered NMDA receptor glycine site antagonists. Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism. vol 15. issue 2. 1995-03-17. PMID:7860652. in the forebrain ischemia model, glycine receptor antagonism had no effect on delayed neuronal necrosis in the hippocampal ca1 sector, neocortex, or caudoputamen. 1995-03-17 2023-08-12 rat
L E Jarrard, B S Meldru. Selective excitotoxic pathology in the rat hippocampus. Neuropathology and applied neurobiology. vol 19. issue 5. 1994-02-07. PMID:8278020. after quis and ka injections the hippocampal damage was limited to hilar cells in the dentate gyrus, ca3 pyramidal cells, and partial loss of ca1 cells; there was extensive extrahippocampal damage including entorhinal cortex, amygdala, layers iii, v, and vi of ventral neocortex, olfactory areas, and various thalamic nuclei. 1994-02-07 2023-08-12 rat
K Matsuo, H Ikeshima, K Shimoda, A Umezawa, J Hata, K Maejima, H Nojima, T Takan. Expression of the rat calmodulin gene II in the central nervous system: a 294-base promoter and 68-base leader segment mediates neuron-specific gene expression in transgenic mice. Brain research. Molecular brain research. vol 20. issue 1-2. 1994-01-11. PMID:8255185. this short camii promoter mediated the transgene expression in pyramidal cells of the cerebral neocortex, the pyriformcortex and the hippocampal regions ca1 to ca3, in granule cells of the dentate gyrus, in purkinje cells of the cerebellum, and in neurons of the lateral vestibular nucleus of pons and the spinal cord of adult transgenic mice. 1994-01-11 2023-08-12 mouse
A Kirkwood, S M Dudek, J T Gold, C D Aizenman, M F Bea. Common forms of synaptic plasticity in the hippocampus and neocortex in vitro. Science (New York, N.Y.). vol 260. issue 5113. 1993-06-28. PMID:8502997. activity-dependent synaptic plasticity in the superficial layers of juvenile cat and adult rat visual neocortex was compared with that in adult rat hippocampal field ca1. 1993-06-28 2023-08-12 rat
A Kirkwood, S M Dudek, J T Gold, C D Aizenman, M F Bea. Common forms of synaptic plasticity in the hippocampus and neocortex in vitro. Science (New York, N.Y.). vol 260. issue 5113. 1993-06-28. PMID:8502997. these results provide strong support for the view that common principles may govern experience-dependent synaptic plasticity in ca1 and throughout the superficial layers of the mammalian neocortex. 1993-06-28 2023-08-12 rat
C del Arco, I Galende, A Pazo. Autoradiographic mapping of 5-HT1 receptors in the guinea-pig brain with particular reference to the 5-HT1D receptor sites. Naunyn-Schmiedeberg's archives of pharmacology. vol 347. issue 3. 1993-05-27. PMID:8479545. areas presenting high densities of 5-ht1a receptors included the neocortex (internal layers), hippocampal formation (dentate gyrus, ca1 field), septum and raphe nuclei, while 5-ht1c sites accounted for most of the [3h]-5-ht binding to the choroid plexus. 1993-05-27 2023-08-12 Not clear
K Dineley-Miller, J Patric. Gene transcripts for the nicotinic acetylcholine receptor subunit, beta4, are distributed in multiple areas of the rat central nervous system. Brain research. Molecular brain research. vol 16. issue 3-4. 1993-04-01. PMID:1337943. moderate hybridization signals were seen in the isocortex (layers i-iii), in olfactory regions, in fields ca1 through ca4 of ammon's horn and the entorhinal cortex of the hippocampal formation, in the supramammillary nucleus, in the pontine nucleus, in the cerebellum, and in the locus coeruleus. 1993-04-01 2023-08-11 rat
M Horn, W Schlot. Delayed neuronal death and delayed neuronal recovery in the human brain following global ischemia. Acta neuropathologica. vol 85. issue 1. 1993-03-11. PMID:1285498. the severity of the resultant ischemic injury in hippocampus ca1, among purkinje cells, or in frontal neocortex, respectively, was quantified by direct counting of necrotic neurons. 1993-03-11 2023-08-11 human
C E Ribak, L Seress, C Lerant. Electron microscopic immunocytochemical study of the distribution of parvalbumin-containing neurons and axon terminals in the primate dentate gyrus and Ammon's horn. The Journal of comparative neurology. vol 327. issue 2. 1993-03-02. PMID:8425946. one prominent pv-positive cell type in ca1 was a large multipolar neuron that resembled the large basket cells of the neocortex. 1993-03-02 2023-08-12 monkey
J L Yau, R C Dow, G Fink, J R Seck. Medial septal cholinergic lesions increase hippocampal mineralocorticoid and glucocorticoid receptor messenger RNA expression. Brain research. vol 577. issue 1. 1992-10-09. PMID:1325858. expression of both mr and gr mrna was significantly increased in a subregions of the hippocampus, but not neocortex, with the greatest increase in the ca1 area for mr mrna and dentate gyrus for gr mrna. 1992-10-09 2023-08-11 rat
C Marie, C Mossiat, A Beley, J Brale. Alpha-methyl-para-tyrosine pretreatment protects from striatal neuronal death induced by four-vessel occlusion in the rat. Neurochemical research. vol 17. issue 10. 1992-09-24. PMID:1354843. evaluation of brain damage 72 hours after ischemia showed that amt treatment significantly decreased neuronal necrosis in the striatum but had no cytoprotective effect in the ca1 sector of the hippocampus and in the neocortex. 1992-09-24 2023-08-11 rat