All Relations between gaba and glutamatergic

Publication Sentence Publish Date Extraction Date Species
N G Weilan. Glutamic acid decarboxylase messenger ribonucleic acid is regulated by estradiol and progesterone in the hippocampus. Endocrinology. vol 131. issue 6. 1992-12-30. PMID:1446611. normal function depends on precise interactions between the inhibitory gamma-aminobutyric acid (gaba)ergic and excitatory glutamatergic neurons of the hippocampus. 1992-12-30 2023-08-11 rat
T Galli, J M Desce, F Artaud, M L Kemel, A Chéramy, J Glowinsk. Modulation of GABA release by alpha-amino-3-hydroxy-5-methylisoxazole-4-propionate and N-methyl-D-aspartate receptors in matrix-enriched areas of the rat striatum. Neuroscience. vol 50. issue 4. 1992-12-29. PMID:1280348. the selective glutamatergic agonist alpha-amino-3-hydroxy-5-methylisoxazole-4-propionate (1 mm) enhanced the potassium-evoked release of [3h]gaba as well as the basal outflow of [3h]gaba. 1992-12-29 2023-08-11 rat
B Hassel, R E Paulsen, A Johnsen, F Fonnu. Selective inhibition of glial cell metabolism in vivo by fluorocitrate. Brain research. vol 576. issue 1. 1992-10-06. PMID:1515906. differences in the labelling of gamma-aminobutyric acid (gaba) from [14c]acetate and citrate suggest that astrocytes associated with gabaergic and glutamatergic nerve terminals may differ in their preference for amino acid precursors. 1992-10-06 2023-08-11 rat
D H Penning, J Patrick, S Jimmo, J F Brie. Release of glutamate and gamma-aminobutyric acid in the ovine fetal hippocampus: ontogeny and effect of hypoxia. Journal of developmental physiology. vol 16. issue 5. 1992-10-06. PMID:1688088. the lack of calcium ion regulation of glutamate efflux compared with gaba efflux indicates either a difference in maturation of glutamatergic synaptic mechanisms compared with gabaergic mechanisms, or is indicative of glial release of glutamate. 1992-10-06 2023-08-11 Not clear
R Schneggenburger, J López-Barneo, A Konnert. Excitatory and inhibitory synaptic currents and receptors in rat medial septal neurones. The Journal of physiology. vol 445. 1992-09-17. PMID:1354258. thus the major excitatory and inhibitory synaptic inputs were identified as being due to activation of quisqualate/kainate glutamatergic and gamma-aminobutyric acid (gabaa) receptors, respectively. 1992-09-17 2023-08-11 rat
D K Bosman, N E Deutz, M A Maas, H M van Eijk, J J Smit, J G de Haan, R A Chamulea. Amino acid release from cerebral cortex in experimental acute liver failure, studied by in vivo cerebral cortex microdialysis. Journal of neurochemistry. vol 59. issue 2. 1992-08-20. PMID:1629730. both increased gamma-aminobutyric acid (gaba)-ergic and decreased glutamatergic neurotransmission have been suggested relative to the pathophysiology of hepatic encephalopathy. 1992-08-20 2023-08-11 rat
E Cherubini, J L Gaiarsa, Y Ben-Ar. GABA: an excitatory transmitter in early postnatal life. Trends in neurosciences. vol 14. issue 12. 1992-07-15. PMID:1726341. in fact, during the early neonatal period, gaba acting on gabaa receptors provides most of the excitatory drive, whereas excitatory glutamatergic synapses are quiescent. 1992-07-15 2023-08-11 Not clear
L Kerkerian-Le Goff, C Forni, D Samuel, A Bloc, N Dusticier, A Nieoullo. Intracerebroventricular administration of neuropeptide Y affects parameters of dopamine, glutamate and GABA activities in the rat striatum. Brain research bulletin. vol 28. issue 2. 1992-07-07. PMID:1596740. these results indicate that npy may be involved in regulating the activity of nigral dopaminergic and cortical glutamatergic afferent pathways and that of intrinsic gaba neurons in the rat striatum. 1992-07-07 2023-08-11 rat
R Nitsch, M Frotsche. Reduction of posttraumatic transneuronal "early gene" activation and dendritic atrophy by the N-methyl-D-aspartate receptor antagonist MK-801. Proceedings of the National Academy of Sciences of the United States of America. vol 89. issue 11. 1992-06-26. PMID:1534412. the removal of a major hippocampal afferent system, the glutamatergic fibers from the entorhinal cortex, results in transneuronal changes in postsynaptic inhibitory neurons using gamma-aminobutyric acid (gaba) as a neurotransmitter. 1992-06-26 2023-08-11 Not clear
D G Rainnie, E K Asprodini, P Shinnick-Gallaghe. Kindling-induced long-lasting changes in synaptic transmission in the basolateral amygdala. Journal of neurophysiology. vol 67. issue 2. 1992-05-27. PMID:1349037. these results provide evidence that the epileptiform activity observed in bla neurons after kindling results from an increase in excitatory nmda- and non-nmda-receptor-mediated glutamatergic transmission and a decrease in inhibitory gamma-aminobutyric acid (gaba)-receptor-mediated transmission; the enhanced excitatory transmission cannot be accounted for by reduced inhibition. 1992-05-27 2023-08-11 Not clear
P Kugler, G Baie. Mitochondrial enzymes related to glutamate and GABA metabolism in the hippocampus of young and aged rats: a quantitative histochemical study. Neurochemical research. vol 17. issue 2. 1992-03-31. PMID:1347164. from the present study it is concluded that, 1. the enzymatic complement of mitochondria in neurons and glia depends upon layer specific metabolic processes of the hippocampus (also with respect to glutamatergic and gabaergic terminal fields) indicating a layer specific interaction of the enzymes studied to produce or catabolize glutamate and gaba, and 2. the age dependent changes of the studied enzymes are very restricted. 1992-03-31 2023-08-11 rat
F Zafra, E Castrén, H Thoenen, D Lindhol. Interplay between glutamate and gamma-aminobutyric acid transmitter systems in the physiological regulation of brain-derived neurotrophic factor and nerve growth factor synthesis in hippocampal neurons. Proceedings of the National Academy of Sciences of the United States of America. vol 88. issue 22. 1991-12-26. PMID:1658793. here we demonstrate that the balance between the activity of the glutamatergic and gamma-aminobutyric acid (gaba)ergic systems controls the physiological levels of bdnf and ngf mrnas in hippocampal neurons in vitro and in vivo. 1991-12-26 2023-08-11 Not clear
M Y Globus, M D Ginsberg, R Bust. Excitotoxic index--a biochemical marker of selective vulnerability. Neuroscience letters. vol 127. issue 1. 1991-09-30. PMID:1679223. glycine and gamma-aminobutyric acid (gaba), which modulate glutamatergic activity, may also play a significant role. 1991-09-30 2023-08-11 rat
M Palmi, S Brooke, A D Smith, J P Bola. GABA-like immunoreactivity in different cellular populations of cerebellar cortex of rats before and after treatment with amino-oxyacetic acid. Brain research. vol 543. issue 2. 1991-08-07. PMID:2059835. thus, in both control and treated rats, the highest gaba immunoreactivity was present in the terminals of gabaergic cells, and the lowest in putative glutamatergic cells. 1991-08-07 2023-08-11 rat
P Calabresi, N B Mercuri, M De Murtas, G Bernard. Endogenous GABA mediates presynaptic inhibition of spontaneous and evoked excitatory synaptic potentials in the rat neostriatum. Neuroscience letters. vol 118. issue 1. 1991-01-31. PMID:2259476. the effect of the blockade of the gamma-aminobutyric acid (gaba) uptake system on the amplitude of glutamatergic synaptic potentials was studied by using a corticostriatal slice preparation. 1991-01-31 2023-08-11 rat
P Calabresi, N B Mercuri, M De Murtas, G Bernard. Endogenous GABA mediates presynaptic inhibition of spontaneous and evoked excitatory synaptic potentials in the rat neostriatum. Neuroscience letters. vol 118. issue 1. 1991-01-31. PMID:2259476. these findings show that phaclofen-insensitive gabab receptors, activated by endogenous gaba, mediate presynaptic inhibition of cortical glutamatergic inputs in the neostriatum. 1991-01-31 2023-08-11 rat
D Becquet, M Faudon, F Her. In vivo evidence for an inhibitory glutamatergic control of serotonin release in the cat caudate nucleus: involvement of GABA neurons. Brain research. vol 519. issue 1-2. 1990-10-16. PMID:1975768. in vivo evidence for an inhibitory glutamatergic control of serotonin release in the cat caudate nucleus: involvement of gaba neurons. 1990-10-16 2023-08-11 cat
H Akiyama, T Kaneko, N Mizuno, P L McGee. Distribution of phosphate-activated glutaminase in the human cerebral cortex. The Journal of comparative neurology. vol 297. issue 2. 1990-08-23. PMID:2196285. phosphate-activated glutaminase (pag), which catalyses conversion of glutamine to glutamate, is a potential marker for glutamatergic, and possibly gaba, neurons in the central nervous system. 1990-08-23 2023-08-11 human
H Akiyama, T Kaneko, N Mizuno, P L McGee. Distribution of phosphate-activated glutaminase in the human cerebral cortex. The Journal of comparative neurology. vol 297. issue 2. 1990-08-23. PMID:2196285. since pyramidal cells are believed to be glutamatergic and large basket cells gabaergic, these results suggest that pag plays a role in generating not only transmitter glutamate, but also gaba precursor glutamate. 1990-08-23 2023-08-11 human
M P Meeley, M D Underwood, W T Talman, D J Rei. Content and in vitro release of endogenous amino acids in the area of the nucleus of the solitary tract of the rat. Journal of neurochemistry. vol 53. issue 6. 1989-12-21. PMID:2809593. we conclude that (a) glu is a transmitter of primary afferents in the nucleus of the solitary tract; (b) glutamatergic afferents may interact with gamma-aminobutyric acid system(s) in this region; (c) gly also may participate in the mediation and/or modulation of cardiovascular or other visceral reflexes; and (d) amino acid neurotransmission may play an integral role in the neurogenic control of arterial pressure. 1989-12-21 2023-08-11 rat