| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| F J White, X T Hu, X F Zhang, M E Wol. Repeated administration of cocaine or amphetamine alters neuronal responses to glutamate in the mesoaccumbens dopamine system. The Journal of pharmacology and experimental therapeutics. vol 273. issue 1. 1995-05-17. PMID:7714800. |
after five daily injections of either cocaine (15.0 mg/kg) or d-amphetamine (5.0 mg/kg), rats were subjected to in vivo single cell recording to determine the efficacy of iontophoretically administered glutamate in altering the firing of ventral tegmental area da neurons and nucleus accumbens neurons. |
1995-05-17 |
2023-08-12 |
rat |
| R Karler, L D Calder, J B Bedingfiel. Cocaine behavioral sensitization and the excitatory amino acids. Psychopharmacology. vol 115. issue 3. 1995-03-24. PMID:7871069. |
these results suggest that the mechanism of induction to cocaine is similar to that of amphetamine; both the glutamate and dopaminergic systems appear to be involved in induction. |
1995-03-24 |
2023-08-12 |
mouse |
| R Karler, L D Calder, L H Thai, J B Bedingfiel. A dopaminergic-glutamatergic basis for the action of amphetamine and cocaine. Brain research. vol 658. issue 1-2. 1995-03-02. PMID:7834358. |
cpp, an nmda-type glutamate antagonist, given systemically or intrastriatally, blocked stereotypy induced by either amphetamine or cocaine. |
1995-03-02 |
2023-08-12 |
mouse |
| U C Garg, H Turndorf, M Bansinat. Effect of cocaine on macromolecular syntheses and cell proliferation in cultured glial cells. Neuroscience. vol 57. issue 2. 1994-03-29. PMID:7509470. |
furthermore, cocaine had no effect on glutamate uptake either in cortical glia or in c6 glioma cells. |
1994-03-29 |
2023-08-12 |
Not clear |
| J M Witki. Blockade of the locomotor stimulant effects of cocaine and methamphetamine by glutamate antagonists. Life sciences. vol 53. issue 24. 1994-01-03. PMID:7902523. |
blockade of the locomotor stimulant effects of cocaine and methamphetamine by glutamate antagonists. |
1994-01-03 |
2023-08-12 |
mouse |
| S Berretta, H A Robertson, A M Graybie. Dopamine and glutamate agonists stimulate neuron-specific expression of Fos-like protein in the striatum. Journal of neurophysiology. vol 68. issue 3. 1992-11-27. PMID:1359024. |
we administered the indirect monoamine agonists cocaine and amphetamine intraperitoneally and gave the glutamate agonist quinolinic acid by direct intrastriatal injection. |
1992-11-27 |
2023-08-11 |
rat |
| S Berretta, H A Robertson, A M Graybie. Dopamine and glutamate agonists stimulate neuron-specific expression of Fos-like protein in the striatum. Journal of neurophysiology. vol 68. issue 3. 1992-11-27. PMID:1359024. |
the induction by cocaine and amphetamine was blocked by pretreatment with the dopamine d1-like receptor antagonist sch23390, and the induction by quinolinic acid was blocked by pretreatment with mk-801, a noncompetitive antagonist of the n-methyl-d-aspartate (nmda) glutamate receptor. |
1992-11-27 |
2023-08-11 |
rat |
| L Peng, L Hert. Long-lasting abolishment of noradrenaline induced stimulation of oxidative metabolism after chronic exposure of developing mouse astrocytes to cocaine. Brain research. vol 581. issue 2. 1992-10-26. PMID:1356581. |
rate of 14co2 production from [l-14c]glutamate was determined as a measurement of oxidative metabolism in developing primary cultures of astrocytes, obtained from the neonatal mouse brain and grown in the absence (control) or presence of cocaine. |
1992-10-26 |
2023-08-11 |
mouse |
| S Kitayama, S Shimada, H Xu, L Markham, D M Donovan, G R Uh. Dopamine transporter site-directed mutations differentially alter substrate transport and cocaine binding. Proceedings of the National Academy of Sciences of the United States of America. vol 89. issue 16. 1992-09-15. PMID:1502198. |
replacement of aspartate at position 79 with alanine, glycine, or glutamate dramatically reduced uptake of [3h]dopamine and the tritium-labeled parkinsonism-inducing neurotoxin 1-methyl-4-phenylpyridinium (mpp+) and reduced the mutants' affinity for the tritium-labeled cocaine analog (-)-2 beta-carbomethoxy-3 beta-(4-fluorophenyl)tropane (cft) without affecting bmax. |
1992-09-15 |
2023-08-11 |
Not clear |
| Y Itzhak, I Stei. Sensitization to the toxic effects of cocaine in mice is associated with the regulation of N-methyl-D-aspartate receptors in the cortex. The Journal of pharmacology and experimental therapeutics. vol 262. issue 2. 1992-09-11. PMID:1386882. |
the present study was undertaken to examine the role of the n-methyl-d-aspartate (nmda) type of glutamate receptors in the development of sensitization to the convulsive and lethal effects of cocaine in swiss webster mice. |
1992-09-11 |
2023-08-11 |
mouse |
| C A Jimenez-Rivera, B D Waterhous. Effects of systemically and locally applied cocaine on cerebrocortical neuron responsiveness to afferent synaptic inputs and glutamate. Brain research. vol 546. issue 2. 1991-08-22. PMID:1676928. |
effects of systemically and locally applied cocaine on cerebrocortical neuron responsiveness to afferent synaptic inputs and glutamate. |
1991-08-22 |
2023-08-11 |
rat |
| S A Turkanis, L M Partlow, R Karle. Effects of cocaine on neuromuscular transmission in the lobster. Neuropharmacology. vol 28. issue 9. 1989-11-29. PMID:2554188. |
in addition, cocaine shifted the concentration-depolarization curve for glutamate to the right, demonstrating a postsynaptic depression of the response to glutamate. |
1989-11-29 |
2023-08-11 |
lobster |
| R Karler, L D Calder, I A Chaudhry, S A Turkani. Blockade of "reverse tolerance" to cocaine and amphetamine by MK-801. Life sciences. vol 45. issue 7. 1989-10-12. PMID:2671566. |
the data suggest that the glutamate system participates in the mechanism of "reverse tolerance" to the dopaminergic effects of cocaine and amphetamine, as well as to the convulsant effect of cocaine. |
1989-10-12 |
2023-08-11 |
mouse |
| L Pulvirenti, N R Swerdlow, G F Koo. Microinjection of a glutamate antagonist into the nucleus accumbens reduces psychostimulant locomotion in rats. Neuroscience letters. vol 103. issue 2. 1989-10-12. PMID:2771183. |
in order to study a possible modulatory effect of glutamatergic afferents to the nucleus accumbens (nac) on psychostimulant-induced locomotion, l-glutamic acid diethyl ester (gdee), a glutamate antagonist, was injected in the nac of rats acutely treated with cocaine, amphetamine or caffeine. |
1989-10-12 |
2023-08-11 |
rat |
| R Karler, C Petty, L Calder, S A Turkani. Proconvulsant and anticonvulsant effects in mice of acute and chronic treatment with cocaine. Neuropharmacology. vol 28. issue 7. 1989-09-11. PMID:2761680. |
cocaine did not affect convulsion thresholds for strychnine, arecoline or aminophylline; these data suggest that the anticonvulsant action of cocaine is relatively selective for the gamma-aminobutyric acid (gaba) and glutamate systems. |
1989-09-11 |
2023-08-11 |
mouse |