All Relations between Alzheimer Disease and cerebellum

Publication Sentence Publish Date Extraction Date Species
N Ujihira, M Ohya, C Mabuchi, T Indo, Y Hashizum. [An autopsied case of type II citrullinemia--transient effectiveness with either citrate or benzoate to the consciousness disturbance]. Rinsho shinkeigaku = Clinical neurology. vol 29. issue 4. 1990-03-12. PMID:2692930. neuropathologically, characteristic findings were brain edema with cerebellar tonsilar herniation, laminar necrosis with spongy formation in cerebral cortex, and alzheimer type ii glia. 1990-03-12 2023-08-11 Not clear
T Suenaga, A Hirano, J F Llena, H Ksiezak-Reding, S H Yen, D W Dickso. Modified Bielschowsky and immunocytochemical studies on cerebellar plaques in Alzheimer's disease. Journal of neuropathology and experimental neurology. vol 49. issue 1. 1990-02-26. PMID:1688924. modified bielschowsky and immunocytochemical studies on cerebellar plaques in alzheimer's disease. 1990-02-26 2023-08-11 Not clear
T Suenaga, A Hirano, J F Llena, H Ksiezak-Reding, S H Yen, D W Dickso. Modified Bielschowsky and immunocytochemical studies on cerebellar plaques in Alzheimer's disease. Journal of neuropathology and experimental neurology. vol 49. issue 1. 1990-02-26. PMID:1688924. senile plaques (sp) in the cerebellum of 23 cases of alzheimer's disease (ad), three with widespread amyloid angiopathy, were studied with a modified bielschowsky stain and immunocytochemical methods using antibodies to a beta-amyloid synthetic peptide (beta asp), phosphorylated neurofilament proteins, ubiquitin, tau protein, and glial fibrillary acidic protein (gfap). 1990-02-26 2023-08-11 Not clear
E Sofic, J Halket, A Przyborowska, P Riederer, H Beckmann, M Sandler, K Jellinge. Brain quinolinic acid in Alzheimer's dementia. European archives of psychiatry and neurological sciences. vol 239. issue 3. 1990-02-07. PMID:2532134. quinolinic acid (qa) content was measured in postmortem frontal and temporal cortex, putamen and cerebellum obtained from patients with senile dementia of alzheimer type (sdat), huntington's disease (hd) and controls, using a gas chromatography/mass spectrometry method. 1990-02-07 2023-08-11 Not clear
R J Mayer, J Lowe, G Lennox, F Doherty, M Lando. Intermediate filaments and ubiquitin: a new thread in the understanding of chronic neurodegenerative diseases. Progress in clinical and biological research. vol 317. 1990-01-31. PMID:2557642. the conditions include parkinson's disease, motor neurone disease, alzheimer's disease, pick's disease, and alcoholic liver disease as well as cerebellar astrocytomas and a myopathy. 1990-01-31 2023-08-11 human
R N Kalaria, S I Hari. Abnormalities of the glucose transporter at the blood-brain barrier and in brain in Alzheimer's disease. Progress in clinical and biological research. vol 317. 1990-01-31. PMID:2602426. in this study, we assessed the gt protein in the frontal cortex, hippocampus, cerebellum, and cerebral microvessels in subjects with alzheimer's disease (ad) and controls. 1990-01-31 2023-08-11 human
C Bancher, I Grundke-Iqbal, K Iqbal, K S Kim, H M Wisniewsk. A 31 kilodalton beta-protein immunoreactive polypeptide in neuronal lipofuscin. Progress in clinical and biological research. vol 317. 1990-01-31. PMID:2690128. this material is immunoreactive in every brain area examined (cerebral and cerebellar cortices, basal ganglia, brainstem) and its reactivity is independent from the presence of alzheimer's disease. 1990-01-31 2023-08-11 Not clear
K Okamoto, H Yamaguchi, S Hirai, M Shoji, K Inoue, M Takatam. Immunogold electron microscopic study of cerebrovascular and senile plaque amyloid using anti-beta protein antibody. Progress in clinical and biological research. vol 317. 1990-01-31. PMID:2690130. diffuse (very primitive) plaques of the cerebral cortex and cerebellum in subjects with senile dementia of the alzheimer type (sdat) were seen as ill-defined areas of the fine fibrillar materials labeled by beta protein immunostain and silver impregnation. 1990-01-31 2023-08-11 human
H Braak, E Braak, J Bohl, W Lan. Alzheimer's disease: amyloid plaques in the cerebellum. Journal of the neurological sciences. vol 93. issue 2-3. 1990-01-19. PMID:2556503. alzheimer's disease: amyloid plaques in the cerebellum. 1990-01-19 2023-08-11 Not clear
H Braak, E Braak, J Bohl, W Lan. Alzheimer's disease: amyloid plaques in the cerebellum. Journal of the neurological sciences. vol 93. issue 2-3. 1990-01-19. PMID:2556503. two specific silver-staining methods demonstrating either extracellular amyloid and/or precursors of amyloid or intraneuronal neurofibrillary changes were used to examine cerebellar pathology in cases of presenile and senile dementia of the alzheimer type, cases of down's syndrome, and non-demented controls. 1990-01-19 2023-08-11 Not clear
M Kelley, N Kowal. Corticotropin-releasing factor immunoreactive neurons persist throughout the brain in Alzheimer's disease. Brain research. vol 501. issue 2. 1990-01-08. PMID:2819447. corticotropin-releasing factor (crf) immunoreactivity was examined in the hippocampal formation, cerebellum and hypothalamus of normal aged and alzheimer's disease (ad) brains. 1990-01-08 2023-08-11 Not clear
S Flament, A Delacourte, B Hémon, A Défosse. Characterization of two pathological tau protein, variants in Alzheimer brain cortices. Journal of the neurological sciences. vol 92. issue 2-3. 1989-12-01. PMID:2509637. tau 64 and 69 were not found in regions of alzheimer brains where the alzheimer pathology was absent (caudate nucleus or cerebellum for example). 1989-12-01 2023-08-11 Not clear
M Tabaton, S Cammarata, V Manetto, G Perry, G Mancard. Tau-reactive neurofibrillary tangles in cerebellar cortex from patients with Alzheimer's disease. Neuroscience letters. vol 103. issue 3. 1989-11-28. PMID:2478932. in 4 cases of alzheimer's disease (ad) a tau antiserum immunostained thin, round or flame-shaped profiles disposed around the nuclei of the cerebellar fusiform-type golgi cells. 1989-11-28 2023-08-11 Not clear
M D Johnson, J M Kamso-Pratt, W O Whetsell, R B Pepinsk. Lipocortin-1 immunoreactivity in the normal human central nervous system and lesions with astrocytosis. American journal of clinical pathology. vol 92. issue 4. 1989-11-07. PMID:2529760. l-1 immunoreactivity was evaluated in the frontal cortex, parahippocampal gyrus/lateral ventricle, cerebellum, medulla, and spinal cord from 27 normal human fetuses, neonates, and adults without neurologic disease and in these same regions and representative lesions from 35 patients with diseases producing varying degrees of astrocytosis, including intraparenchymal hemorrhage; embolic, thrombotic, or traumatic infarctions; and alzheimer's disease (ad). 1989-11-07 2023-08-11 human
W D Heiss, K Herholz, G Pawlik, I Hebold, P Klinkhammer, B Szelie. Positron emission tomography findings in dementia disorders: contributions to differential diagnosis and objectivizing of therapeutic effects. The Keio journal of medicine. vol 38. issue 2. 1989-10-20. PMID:2674513. degenerative dementia of the alzheimer type is characterized by a metabolic disturbance most prominent in the parietooccipito-temporal association cortex and later in the frontal lobe, while primary cortical areas, basal ganglia, thalamus, and cerebellum are not affected. 1989-10-20 2023-08-11 Not clear
C L Joachim, J H Morris, D J Selko. Diffuse senile plaques occur commonly in the cerebellum in Alzheimer's disease. The American journal of pathology. vol 135. issue 2. 1989-10-20. PMID:2675616. diffuse senile plaques occur commonly in the cerebellum in alzheimer's disease. 1989-10-20 2023-08-11 Not clear
G Cole, P Williams, D Alldryck, S Singhar. Amyloid plaques in the cerebellum in Alzheimer's disease. Clinical neuropathology. vol 8. issue 4. 1989-10-19. PMID:2528435. amyloid plaques in the cerebellum in alzheimer's disease. 1989-10-19 2023-08-11 Not clear
G Cole, P Williams, D Alldryck, S Singhar. Amyloid plaques in the cerebellum in Alzheimer's disease. Clinical neuropathology. vol 8. issue 4. 1989-10-19. PMID:2528435. the significance of cerebellar plaques is uncertain, but their presence in alzheimer's disease is by no means as rare as previously thought. 1989-10-19 2023-08-11 Not clear
R N Kalaria, S I Hari. Reduced glucose transporter at the blood-brain barrier and in cerebral cortex in Alzheimer disease. Journal of neurochemistry. vol 53. issue 4. 1989-10-12. PMID:2769254. we studied the hexose transporter protein of the frontal and temporal neocortex, hippocampus, putamen, cerebellum, and cerebral microvessels (which constitute the blood-brain barrier) in alzheimer disease and control subjects by reversible and covalent binding with [3h]cytochalasin b and by immunological reactivity. 1989-10-12 2023-08-11 human
R N Kalaria, S I Hari. Reduced glucose transporter at the blood-brain barrier and in cerebral cortex in Alzheimer disease. Journal of neurochemistry. vol 53. issue 4. 1989-10-12. PMID:2769254. in alzheimer disease subjects, we found a marked decrease in the hexose transporter in brain microvessels and in the cerebral neocortex and hippocampus, regions that are most affected in alzheimer disease, but there were no abnormalities in the putamen or cerebellum. 1989-10-12 2023-08-11 human