All Relations between dopaminergic and basal ganglia

Publication Sentence Publish Date Extraction Date Species
J M Casto, G F Bal. Characterization and localization of D1 dopamine receptors in the sexually dimorphic vocal control nucleus, area X, and the basal ganglia of European starlings. Journal of neurobiology. vol 25. issue 7. 1994-10-20. PMID:8089655. this circuit includes a sexually dimorphic nucleus, area x, that is a subregion of the parolfactory lobe of the basal ganglia and is known from work on zebra finches to receive dopaminergic input from the area ventralis of tsai. 1994-10-20 2023-08-12 Not clear
D G Cole, M Di Figli. Reserpine increases Fos activity in the rat basal ganglia via a quinpirole-sensitive mechanism. Neuroscience. vol 60. issue 1. 1994-09-08. PMID:7914358. c-fos and fos protein have been widely used to investigate basal ganglia responses to changes in dopaminergic neurotransmission. 1994-09-08 2023-08-12 rat
D G Cole, M Di Figli. Reserpine increases Fos activity in the rat basal ganglia via a quinpirole-sensitive mechanism. Neuroscience. vol 60. issue 1. 1994-09-08. PMID:7914358. because dopamine is believed to excite striatal neurons via d1 receptors and inhibit them via d2 receptors, we hypothesized that acute dopamine depletion would increase fos activity in basal ganglia circuits normally inhibited by dopaminergic input to d2 receptors. 1994-09-08 2023-08-12 rat
N Tsuda, K Negoro, M Morimats. [A case of multiple cerebral infarctions associated with hemichorea induced by amantadine hydrochloride]. Rinsho shinkeigaku = Clinical neurology. vol 34. issue 4. 1994-08-09. PMID:8026137. these findings suggest hemichorea was related to hyperfunction of dopaminergic neurons in the left basal ganglia. 1994-08-09 2023-08-12 Not clear
T Di Paol. Modulation of brain dopamine transmission by sex steroids. Reviews in the neurosciences. vol 5. issue 1. 1994-08-04. PMID:8019704. biochemical and behavioral studies in animals have also shown the effect of sex steroids on dopaminergic activity in the basal ganglia; however, both activating and inhibiting effects have been reported. 1994-08-04 2023-08-12 rat
E Bächli, C Alban. [Akinetic crisis in Parkinson disease]. Schweizerische medizinische Wochenschrift. vol 124. issue 23. 1994-08-04. PMID:8023100. while nms is related to dopaminergic receptor blockade or dopaminergic depletion, akinetic crisis can occur despite adequate dopaminergic therapy as a symptom of severe basal ganglia dysfunction related to the advanced stages of parkinson's disease. 1994-08-04 2023-08-12 Not clear
F Rodríguez de Fonseca, J L Martín Calderón, R Mechoulam, M Navarr. Repeated stimulation of D1 dopamine receptors enhances (-)-11-hydroxy-delta 8-tetrahydrocannabinol-dimethyl-heptyl-induced catalepsy in male rats. Neuroreport. vol 5. issue 7. 1994-07-29. PMID:7912554. dopaminergic and cannabinoid receptors are localized in the outflow nuclei of the basal ganglia. 1994-07-29 2023-08-12 rat
A Charara, A Paren. Brainstem dopaminergic, cholinergic and serotoninergic afferents to the pallidum in the squirrel monkey. Brain research. vol 640. issue 1-2. 1994-07-21. PMID:7911724. these results suggest that, in addition to their action at striatal and/or nigral levels, the brainstem dopaminergic, cholinergic and serotoninergic neurons influence the output of the primate basal ganglia by acting directly upon gpi neurons. 1994-07-21 2023-08-12 squirrel_monkey
J T Greenamyre, R V Eller, Z Zhang, A Ovadia, R Kurlan, D M Gas. Antiparkinsonian effects of remacemide hydrochloride, a glutamate antagonist, in rodent and primate models of Parkinson's disease. Annals of neurology. vol 35. issue 6. 1994-07-08. PMID:8210221. loss of dopaminergic innervation of the striatum results in overactivity of the glutamatergic pathways from the subthalamic nucleus to the internal segment of the globus pallidus and the substantia nigra pars reticulata, the output nuclei of the basal ganglia. 1994-07-08 2023-08-12 rat
T Hasegawa, M Shimohira, M Itoh, M Hayashi, J Kohyama, S Kumada, Y Iwakaw. [Polygraphical sleep study on typical absence: relationship between the effect of sodium valproate and nigrostriatal function]. No to hattatsu = Brain and development. vol 26. issue 3. 1994-06-23. PMID:8185977. since tms are thought to be controlled by the nigrostriatal dopaminergic pathway, the different response of basal ganglia to vpa among cases with absence epilepsy would have some relation to the different effectiveness of vpa in controlling seizures. 1994-06-23 2023-08-12 Not clear
A Priori, A Berardelli, M Inghilleri, N Accornero, M Manfred. Motor cortical inhibition and the dopaminergic system. Pharmacological changes in the silent period after transcranial brain stimulation in normal subjects, patients with Parkinson's disease and drug-induced parkinsonism. Brain : a journal of neurology. vol 117 ( Pt 2). 1994-06-22. PMID:8186958. dopaminergic drugs modulate the duration of the cortical silent periods in patients and in normal subjects, through mechanisms acting mainly at basal ganglia and possibly also directly at cortical level. 1994-06-22 2023-08-12 human
O Suchowersk. Gilles de la Tourette syndrome. The Canadian journal of neurological sciences. Le journal canadien des sciences neurologiques. vol 21. issue 1. 1994-06-15. PMID:8180905. neuropathophysiology of tourette syndrome is unknown but investigations have suggested that the cause may be an abnormality either in the dopaminergic or endorphin receptor system within the basal ganglia. 1994-06-15 2023-08-12 Not clear
Z H Qin, S P Zhang, B Weis. Dopaminergic and glutamatergic blocking drugs differentially regulate glutamic acid decarboxylase mRNA in mouse brain. Brain research. Molecular brain research. vol 21. issue 3-4. 1994-06-01. PMID:8170353. dopaminergic and glutamatergic inputs play an important role in regulating the activity of gabaergic neurons in basal ganglia. 1994-06-01 2023-08-12 mouse
H S Markus, D C Costa, A J Lee. HMPAO SPECT in Parkinson's disease before and after levodopa: correlation with dopaminergic responsiveness. Journal of neurology, neurosurgery, and psychiatry. vol 57. issue 2. 1994-04-14. PMID:8126501. basal ganglia perfusion assessed by this technique is unlikely to be of use in the diagnosis of parkinson's disease that is responsive to dopaminergic treatment. 1994-04-14 2023-08-12 human
A Takahashi, M Murasak. [Drug-induced movement disorders]. Nihon rinsho. Japanese journal of clinical medicine. vol 51. issue 11. 1994-02-04. PMID:7903986. most of these drugs interfere with dopaminergic transmission within the basal ganglia. 1994-02-04 2023-08-12 Not clear
P Jalil, M C Miranda, C Frenke. [Progressive hereditary dystonia with diurnal fluctuations: report of 2 cases]. Revista medica de Chile. vol 121. issue 5. 1994-02-03. PMID:7903816. the pathogenesis of this disease could be a functional alteration of basal ganglia in the dopaminergic striatonigral region. 1994-02-03 2023-08-12 Not clear
A Benazzouz, C Gross, J Féger, T Boraud, B Bioula. Reversal of rigidity and improvement in motor performance by subthalamic high-frequency stimulation in MPTP-treated monkeys. The European journal of neuroscience. vol 5. issue 4. 1994-01-27. PMID:8261116. in parkinson's disease the loss of dopaminergic neurons in the substantia nigra is associated with global disorganization of basal ganglia activity and, in particular, with increased activity of the excitatory glutamatergic neurons of the subthalamic nucleus. 1994-01-27 2023-08-12 monkey
B K Lipska, D R Weinberge. Delayed effects of neonatal hippocampal damage on haloperidol-induced catalepsy and apomorphine-induced stereotypic behaviors in the rat. Brain research. Developmental brain research. vol 75. issue 2. 1994-01-26. PMID:7903225. the developmental effects of neonatal excitotoxic ventral hippocampal (vh) damage on behaviors related to dopaminergic (da) transmission in the basal ganglia were investigated in the rat. 1994-01-26 2023-08-12 rat
R Hitzemann, Y Qian, B Hitzeman. Dopamine and acetylcholine cell density in the neuroleptic responsive (NR) and neuroleptic nonresponsive (NNR) lines of mice. The Journal of pharmacology and experimental therapeutics. vol 266. issue 1. 1993-08-19. PMID:8101223. overall, these data illustrate that selective breeding for neuroleptic response has yielded lines of mice with significant differences in both the cholinergic and dopaminergic systems of the basal ganglia. 1993-08-19 2023-08-12 mouse
J Kornhuber, M Welle. Amantadine and the glutamate hypothesis of schizophrenia. Experiences in the treatment of neuroleptic malignant syndrome. Journal of neural transmission. General section. vol 92. issue 1. 1993-08-17. PMID:8101093. our observations support the glutamate hypothesis of schizophrenia which suggests that reduced glutamatergic transmission causes a relative dopaminergic excess in the basal ganglia and the limbic system. 1993-08-17 2023-08-12 Not clear