All Relations between reward and nucleus accumbens

Publication Sentence Publish Date Extraction Date Species
I N Acworth, K Ressler, R J Wurtma. Feeding-associated alterations in striatal neurotransmitter release. Annals of the New York Academy of Sciences. vol 575. 1996-10-07. PMID:11538862. dopamine release from the nucleus accumbens did increase with free access to pellets post starvation or after food reward. 1996-10-07 2023-08-12 rat
S Ikemoto, J Panksep. Dissociations between appetitive and consummatory responses by pharmacological manipulations of reward-relevant brain regions. Behavioral neuroscience. vol 110. issue 2. 1996-10-02. PMID:8731060. microinjections of the dopamine antagonist cis-flupentixol or the cholinergic antagonist atropine into the nucleus accumbens (acb) severely reduced the approach speed and anticipatory shuttlebox activity while leaving the consumption of the 20% sucrose reward intact. 1996-10-02 2023-08-12 rat
W A Carlezon, R A Wis. Rewarding actions of phencyclidine and related drugs in nucleus accumbens shell and frontal cortex. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 16. issue 9. 1996-06-18. PMID:8622141. when considered with independent evidence, the present results suggest a model of drug reward within which the critical event is inhibition of medium spiny neurons in nucleus accumbens. 1996-06-18 2023-08-12 rat
K C Berridg. Food reward: brain substrates of wanting and liking. Neuroscience and biobehavioral reviews. vol 20. issue 1. 1996-06-14. PMID:8622814. mediation of wanting related to food reward involves mesotelencephalic dopamine systems, and divisions of nucleus accumbens and amygdala. 1996-06-14 2023-08-12 Not clear
F G Kaddis, N J Uretsky, L J Wallac. DNQX in the nucleus accumbens inhibits cocaine-induced conditioned place preference. Brain research. vol 697. issue 1-2. 1996-04-11. PMID:8593597. several lines of evidence suggest that activation of both ampa/kainate receptors and dopaminergic receptors in the nucleus accumbens may be required for psychostimulant drug induced reward. 1996-04-11 2023-08-12 rat
F C Zhou, J K Zhang, L Lumeng, T K L. Mesolimbic dopamine system in alcohol-preferring rats. Alcohol (Fayetteville, N.Y.). vol 12. issue 5. 1996-01-22. PMID:8519434. the mesolimbic dopamine system of the brain, in particular the nucleus accumbens (acb), is long known to be involved in reward behavior. 1996-01-22 2023-08-12 rat
M A Parada, M Puig de Parada, B G Hoebe. Rats self-inject a dopamine antagonist in the lateral hypothalamus where it acts to increase extracellular dopamine in the nucleus accumbens. Pharmacology, biochemistry, and behavior. vol 52. issue 1. 1996-01-16. PMID:7501663. local injection of sulpiride to block dopamine (primarily d2-type) receptors in the perifornical lateral hypothalamus (pf-lh) can induce locomotion, feeding, and drinking, and in the present study, local sulpiride induced reward and dopamine (da) release in the nucleus accumbens. 1996-01-16 2023-08-12 rat
P I Johnson, J B Goodman, R Condon, J R Stella. Reward shifts and motor responses following microinjections of opiate-specific agonists into either the core or shell of the nucleus accumbens. Psychopharmacology. vol 120. issue 2. 1995-12-07. PMID:7480552. reward shifts and motor responses following microinjections of opiate-specific agonists into either the core or shell of the nucleus accumbens. 1995-12-07 2023-08-12 rat
P I Johnson, J B Goodman, R Condon, J R Stella. Reward shifts and motor responses following microinjections of opiate-specific agonists into either the core or shell of the nucleus accumbens. Psychopharmacology. vol 120. issue 2. 1995-12-07. PMID:7480552. differences in pharmacology, anatomical connections, and receptor densities between the "core" and "shell" of the nucleus accumbens suggest that behavioral activity normally modulated by the accumbens, such as reward and motor functions, may be differentially regulated across the mediolateral axis. 1995-12-07 2023-08-12 rat
C S Maldonado-Irizarry, C J Swanson, A E Kelle. Glutamate receptors in the nucleus accumbens shell control feeding behavior via the lateral hypothalamus. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 15. issue 10. 1995-12-04. PMID:7472436. these findings demonstrate a selective role for non-nmda receptors in the nucleus accumbens shell in ingestive behavior, and suggest an important functional link between two major brain regions involved in reward, the nucleus accumbens and lateral hypothalamus. 1995-12-04 2023-08-12 rat
P J Fletche. Effects of d-fenfluramine and metergoline on responding for conditioned reward and the response potentiating effect of nucleus accumbens d-amphetamine. Psychopharmacology. vol 118. issue 2. 1995-08-18. PMID:7617802. effects of d-fenfluramine and metergoline on responding for conditioned reward and the response potentiating effect of nucleus accumbens d-amphetamine. 1995-08-18 2023-08-12 rat
P J Fletche. Effects of d-fenfluramine and metergoline on responding for conditioned reward and the response potentiating effect of nucleus accumbens d-amphetamine. Psychopharmacology. vol 118. issue 2. 1995-08-18. PMID:7617802. these studies investigated the effects of the 5-hydroxytryptamine (5-ht) releaser, and re-uptake inhibitor, d-fenfluramine, and the non-selective 5-ht receptor antagonist metergoline, on responding for conditioned reward (cr), and on the potentiation of responding for cr following amphetamine injected into the nucleus accumbens. 1995-08-18 2023-08-12 rat
F J Vaccarino, J S Mogil, L Stinu. Chronic dopamine antagonism facilitates opiate-induced feeding. Journal of psychiatry & neuroscience : JPN. vol 20. issue 3. 1995-07-27. PMID:7786882. chronic interference with dopamine (da) transmission has been found to facilitate opiate reward and opiate-induced behavioral activation derived from the nucleus accumbens. 1995-07-27 2023-08-12 rat
F Boix, M Pfister, J P Huston, R K Schwartin. Substance P decreases extracellular concentrations of acetylcholine in neostriatum and nucleus accumbens in vivo: possible relevance for the central processing of reward and aversion. Behavioural brain research. vol 63. issue 2. 1995-01-25. PMID:7528024. substance p decreases extracellular concentrations of acetylcholine in neostriatum and nucleus accumbens in vivo: possible relevance for the central processing of reward and aversion. 1995-01-25 2023-08-12 rat
A Heinz, L G Schmidt, F M Reischie. Anhedonia in schizophrenic, depressed, or alcohol-dependent patients--neurobiological correlates. Pharmacopsychiatry. vol 27 Suppl 1. 1995-01-04. PMID:7984706. these symptoms may be caused by a functional deficit of dopaminergic transmission in the dopaminergic reward system, ascending from the mesencephalon to the ventral striatum (nucleus accumbens). 1995-01-04 2023-08-12 Not clear
E A Kiyatkin, A Gratto. Electrochemical monitoring of extracellular dopamine in nucleus accumbens of rats lever-pressing for food. Brain research. vol 652. issue 2. 1994-12-29. PMID:7953734. high-speed chronoamperometry and monoamine-selective electrochemical probes were used to monitor, during each of 5-6 consecutive daily sessions, changes in dopamine (da)-related oxidation current in nucleus accumbens (nacc) of rats lever-pressing for a food reward. 1994-12-29 2023-08-12 rat
F J Vaccarin. Nucleus accumbens dopamine-CCK interactions in psychostimulant reward and related behaviors. Neuroscience and biobehavioral reviews. vol 18. issue 2. 1994-09-15. PMID:7914685. nucleus accumbens dopamine-cck interactions in psychostimulant reward and related behaviors. 1994-09-15 2023-08-12 Not clear
F J Vaccarin. Nucleus accumbens dopamine-CCK interactions in psychostimulant reward and related behaviors. Neuroscience and biobehavioral reviews. vol 18. issue 2. 1994-09-15. PMID:7914685. based on evidence derived from these different behavioral paradigms, this paper reviews data indicating that nucleus accumbens cck and da play an important role in mediating reward. 1994-09-15 2023-08-12 Not clear
P I Johnson, J R Stella. N-methyl-D-aspartic acid-induced lesions of the nucleus accumbens and/or ventral pallidum fail to attenuate lateral hypothalamic self-stimulation reward. Brain research. vol 646. issue 1. 1994-09-15. PMID:8055342. n-methyl-d-aspartic acid-induced lesions of the nucleus accumbens and/or ventral pallidum fail to attenuate lateral hypothalamic self-stimulation reward. 1994-09-15 2023-08-12 Not clear
P I Johnson, J R Stella. N-methyl-D-aspartic acid-induced lesions of the nucleus accumbens and/or ventral pallidum fail to attenuate lateral hypothalamic self-stimulation reward. Brain research. vol 646. issue 1. 1994-09-15. PMID:8055342. the above data suggests that the nucleus accumbens and the ventral pallidum are not critical for icss rewards stimulation and that hypothalamic icss reward signals are processed downstream from these limbic structures. 1994-09-15 2023-08-12 Not clear