| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| C Bergeron, A Davis, A E Lan. Corticobasal ganglionic degeneration and progressive supranuclear palsy presenting with cognitive decline. Brain pathology (Zurich, Switzerland). vol 8. issue 2. 1998-06-12. PMID:9546292. |
although some pathological features of cbgd overlap with those of other forms of non-alzheimer non-lewy body dementia, the distribution and relative number of these abnormalities and the distinctive pattern of tau immunodeposits allows the distinction of cbgd from pick's disease and fronto-temporal dementia. |
1998-06-12 |
2023-08-12 |
Not clear |
| T Tapiola, M Lehtovirta, J Ramberg, S Helisalmi, K Linnaranta, P Riekkinen, H Soinine. CSF tau is related to apolipoprotein E genotype in early Alzheimer's disease. Neurology. vol 50. issue 1. 1998-02-06. PMID:9443475. |
however, csf tau values in the non-ad dementia group did not differ significantly from those of ad patients or neurologic control subjects. |
1998-02-06 |
2023-08-12 |
human |
| T Tapiola, M Lehtovirta, J Ramberg, S Helisalmi, K Linnaranta, P Riekkinen, H Soinine. CSF tau is related to apolipoprotein E genotype in early Alzheimer's disease. Neurology. vol 50. issue 1. 1998-02-06. PMID:9443475. |
this study shows that the current elisa test for csf tau is not sensitive and specific enough to distinguish early ad from other dementias and indicates that in the interpretation of csf tau analysis as a diagnostic tool, the apoe genotype should also be taken into account. |
1998-02-06 |
2023-08-12 |
human |
| D R Giffor. Assessment of CSF levels of tau protein in patients with Alzheimer's disease and mild dementia. Neurology. vol 49. issue 6. 1998-01-16. PMID:9409391. |
assessment of csf levels of tau protein in patients with alzheimer's disease and mild dementia. |
1998-01-16 |
2023-08-12 |
Not clear |
| H Arai, Y Morikawa, M Higuchi, T Matsui, C M Clark, M Miura, N Machida, V M Lee, J Q Trojanowski, H Sasak. Cerebrospinal fluid tau levels in neurodegenerative diseases with distinct tau-related pathology. Biochemical and biophysical research communications. vol 236. issue 2. 1997-08-28. PMID:9240421. |
cerebrospinal fluid tau (csf-tau) levels were quantified in 8 patients with frontotemporal dementia (ftd), 6 patients with progressive supranuclear palsy (psp), 3 patients with corticobasal degeneration (cbd), and 6 patients with dementia with lewy bodies (dlb). |
1997-08-28 |
2023-08-12 |
Not clear |
| D W Dickson, M B Feany, S H Yen, L A Mattiace, P Davie. Cytoskeletal pathology in non-Alzheimer degenerative dementia: new lesions in diffuse Lewy body disease, Pick's disease, and corticobasal degeneration. Journal of neural transmission. Supplementum. vol 47. 1997-06-11. PMID:8841955. |
although nf, ubiquitin and tau proteins are present in diverse neuronal and glial inclusions in these disorders, the morphology and distribution of these lesions differentiate non-ad dementias. |
1997-06-11 |
2023-08-12 |
Not clear |
| N Rösler, I Wichart, C Bancher, K A Jellinge. Tau protein and apolipoprotein E in CSF diagnostics of Alzheimer's disease: impact on non Alzheimer's dementia? Journal of neural transmission. Supplementum. vol 47. 1997-06-11. PMID:8841971. |
tau protein and apolipoprotein e in csf diagnostics of alzheimer's disease: impact on non alzheimer's dementia? |
1997-06-11 |
2023-08-12 |
Not clear |
| M G Spillantini, M Goedert, R A Crowther, J R Murrell, M R Farlow, B Ghett. Familial multiple system tauopathy with presenile dementia: a disease with abundant neuronal and glial tau filaments. Proceedings of the National Academy of Sciences of the United States of America. vol 94. issue 8. 1997-05-22. PMID:9108114. |
familial multiple system tauopathy with presenile dementia: a disease with abundant neuronal and glial tau filaments. |
1997-05-22 |
2023-08-12 |
Not clear |
| M G Spillantini, M Goedert, R A Crowther, J R Murrell, M R Farlow, B Ghett. Familial multiple system tauopathy with presenile dementia: a disease with abundant neuronal and glial tau filaments. Proceedings of the National Academy of Sciences of the United States of America. vol 94. issue 8. 1997-05-22. PMID:9108114. |
neurofibrillary lesions made of hyperphosphorylated microtubule-associated protein tau constitute not only one of the defining neuropathological features of alzheimer disease but also are present in a number of other neurodegenerative diseases with dementia. |
1997-05-22 |
2023-08-12 |
Not clear |
| M G Spillantini, M Goedert, R A Crowther, J R Murrell, M R Farlow, B Ghett. Familial multiple system tauopathy with presenile dementia: a disease with abundant neuronal and glial tau filaments. Proceedings of the National Academy of Sciences of the United States of America. vol 94. issue 8. 1997-05-22. PMID:9108114. |
tau protein extracted from filaments of familial multiple system tauopathy with presenile dementia shows a minor 72-kda band and two major bands of 64 and 68 kda that contain mainly hyperphosphorylated four-repeat tau isoforms of 383 and 412 amino acids. |
1997-05-22 |
2023-08-12 |
Not clear |
| M G Spillantini, R A Crowther, M Goeder. Comparison of the neurofibrillary pathology in Alzheimer's disease and familial presenile dementia with tangles. Acta neuropathologica. vol 92. issue 1. 1997-03-06. PMID:8811124. |
we have compared the tau pathology in this familial form of presenile dementia with that of ad. |
1997-03-06 |
2023-08-12 |
Not clear |
| M G Spillantini, R A Crowther, M Goeder. Comparison of the neurofibrillary pathology in Alzheimer's disease and familial presenile dementia with tangles. Acta neuropathologica. vol 92. issue 1. 1997-03-06. PMID:8811124. |
these results indicate that in the two diseases tau undergoes the same modifications; they confirm that neurofibrillary tangles and neuropil threads like those in ad can exist independently of beta-amyloid deposits and that their presence is associated with dementia. |
1997-03-06 |
2023-08-12 |
Not clear |
| T Kampers, P Friedhoff, J Biernat, E M Mandelkow, E Mandelko. RNA stimulates aggregation of microtubule-associated protein tau into Alzheimer-like paired helical filaments. FEBS letters. vol 399. issue 3. 1997-01-30. PMID:8985176. |
the microtubule-associated protein tau is the main component of the paired helical filaments (phfs) of alzheimer's disease, the most common senile dementia. |
1997-01-30 |
2023-08-12 |
Not clear |
| M Riemenschneider, K Buch, M Schmolke, A Kurz, W G Gude. Cerebrospinal protein tau is elevated in early Alzheimer's disease. Neuroscience letters. vol 212. issue 3. 1997-01-06. PMID:8843109. |
protein tau concentration was determined by an enzyme linked immunoassay (elisa) in cerebrospinal fluid (csf) of 22 patients with clinically diagnosed alzheimer's disease (ad), in three patients with dementia caused by other neurological disorders (ond) and in 19 cognitively healthy controls. |
1997-01-06 |
2023-08-12 |
Not clear |
| M Riemenschneider, K Buch, M Schmolke, A Kurz, W G Gude. Cerebrospinal protein tau is elevated in early Alzheimer's disease. Neuroscience letters. vol 212. issue 3. 1997-01-06. PMID:8843109. |
a significantly elevated protein tau concentration was found in ad patients as compared with controls, even in 11 patients with very mild dementia. |
1997-01-06 |
2023-08-12 |
Not clear |
| C M Wischik, P C Edwards, R Y Lai, M Roth, C R Harringto. Selective inhibition of Alzheimer disease-like tau aggregation by phenothiazines. Proceedings of the National Academy of Sciences of the United States of America. vol 93. issue 20. 1996-11-25. PMID:8855335. |
in alzheimer disease (ad) the microtubule-associated protein tau is redistributed exponentially into paired helical filaments (phfs) forming neurofibrillary tangles, which correlate with pyramidal cell destruction and dementia. |
1996-11-25 |
2023-08-12 |
Not clear |
| C Strong, B H Anderton, R H Perry, E K Perry, P G Ince, S Loveston. Abnormally phosphorylated tau protein in senile dementia of Lewy body type and Alzheimer disease: evidence that the disorders are distinct. Alzheimer disease and associated disorders. vol 9. issue 4. 1996-10-24. PMID:8749611. |
abnormally phosphorylated tau protein in senile dementia of lewy body type and alzheimer disease: evidence that the disorders are distinct. |
1996-10-24 |
2023-08-12 |
Not clear |
| I Skoog, E Vanmechelen, L A Andreasson, B Palmertz, P Davidsson, C Hesse, K Blenno. A population-based study of tau protein and ubiquitin in cerebrospinal fluid in 85-year-olds: relation to severity of dementia and cerebral atrophy, but not to the apolipoprotein E4 allele. Neurodegeneration : a journal for neurodegenerative disorders, neuroprotection, and neuroregeneration. vol 4. issue 4. 1996-10-24. PMID:8846237. |
a population-based study of tau protein and ubiquitin in cerebrospinal fluid in 85-year-olds: relation to severity of dementia and cerebral atrophy, but not to the apolipoprotein e4 allele. |
1996-10-24 |
2023-08-12 |
Not clear |
| M Jackson, J Low. The new neuropathology of degenerative frontotemporal dementias. Acta neuropathologica. vol 91. issue 2. 1996-09-26. PMID:8787144. |
dementia of frontal type is the preferred term for cases in which no intraneuronal inclusions are seen with antisera to tau and ubiquitin. |
1996-09-26 |
2023-08-12 |
Not clear |
| K Kanemaru, H Yamanouchi, S Matsushit. [Effect of apolipoprotein E phenotype on cerebrospinal fluid levels of tau in patients with dementia of Alzheimer type]. Nihon Ronen Igakkai zasshi. Japanese journal of geriatrics. vol 33. issue 2. 1996-07-26. PMID:8656579. |
[effect of apolipoprotein e phenotype on cerebrospinal fluid levels of tau in patients with dementia of alzheimer type]. |
1996-07-26 |
2023-08-12 |
Not clear |