| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Fabrizio Gardoni, Camilla Bellone, Barbara Viviani, Marina Marinovich, Elena Meli, Domenico E Pellegrini-Giampietro, Flaminio Cattabeni, Monica Di Luc. Lack of PSD-95 drives hippocampal neuronal cell death through activation of an alpha CaMKII transduction pathway. The European journal of neuroscience. vol 16. issue 5. 2002-12-09. PMID:12372013. |
lack of psd-95 drives hippocampal neuronal cell death through activation of an alpha camkii transduction pathway. |
2002-12-09 |
2023-08-12 |
Not clear |
| Fabrizio Gardoni, Camilla Bellone, Barbara Viviani, Marina Marinovich, Elena Meli, Domenico E Pellegrini-Giampietro, Flaminio Cattabeni, Monica Di Luc. Lack of PSD-95 drives hippocampal neuronal cell death through activation of an alpha CaMKII transduction pathway. The European journal of neuroscience. vol 16. issue 5. 2002-12-09. PMID:12372013. |
as a direct consequence of alpha camkii association, we found increased glur1 delivery to cell surface in cultured hippocampal neurons paralleled by ampa-dependent increase in [na+]i levels. |
2002-12-09 |
2023-08-12 |
Not clear |
| Ype Elgersma, Nikolai B Fedorov, Sami Ikonen, Esther S Choi, Minetta Elgersma, Ofelia M Carvalho, Karl Peter Giese, Alcino J Silv. Inhibitory autophosphorylation of CaMKII controls PSD association, plasticity, and learning. Neuron. vol 36. issue 3. 2002-11-26. PMID:12408851. |
our data demonstrate that blocking inhibitory phosphorylation increases camkii in the postsynaptic density (psd), lowers the threshold for hippocampal long-term potentiation (ltp), and results in hippocampal-dependent learning that seems more rigid and less fine-tuned. |
2002-11-26 |
2023-08-12 |
mouse |
| Bhavya Trivedi, Richard H Krame. Patch cramming reveals the mechanism of long-term suppression of cyclic nucleotides in intact neurons. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 22. issue 20. 2002-11-25. PMID:12388588. |
the switch between the ca2+ dependence of lts induction to the ca2+ independence of lts maintenance is consistent with camkii autophosphorylation, similar to proposed mechanisms of hippocampal long-term potentiation. |
2002-11-25 |
2023-08-12 |
Not clear |
| Andrea Menegon, Claudia Verderio, Chiara Leoni, Fabio Benfenati, Andrew J Czernik, Paul Greengard, Michela Matteoli, Flavia Valtort. Spatial and temporal regulation of Ca2+/calmodulin-dependent protein kinase II activity in developing neurons. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 22. issue 16. 2002-09-04. PMID:12177199. |
we have studied ca2+/calmodulin-dependent protein kinase ii (camkii) isoform distribution and activity in embryonic hippocampal neurons developing in culture. |
2002-09-04 |
2023-08-12 |
Not clear |
| Rafael Bejar, Rie Yasuda, Harmen Krugers, Kristin Hood, Mark Mayfor. Transgenic calmodulin-dependent protein kinase II activation: dose-dependent effects on synaptic plasticity, learning, and memory. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 22. issue 13. 2002-07-29. PMID:12097524. |
genetic disruption of calmodulin-dependent protein kinase ii (camkii) function alters hippocampal synaptic plasticity and memory in mice. |
2002-07-29 |
2023-08-12 |
mouse |
| Rafael Bejar, Rie Yasuda, Harmen Krugers, Kristin Hood, Mark Mayfor. Transgenic calmodulin-dependent protein kinase II activation: dose-dependent effects on synaptic plasticity, learning, and memory. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 22. issue 13. 2002-07-29. PMID:12097524. |
these results demonstrate that in the hippocampus, camkii activation facilitates the induction of low-frequency ltp, but at high levels of expression, compensatory mechanisms act to inhibit the induction of this form of ltp. |
2002-07-29 |
2023-08-12 |
mouse |
| Christopher Rong. A fresh look at the role of CaMKII in hippocampal synaptic plasticity and memory. BioEssays : news and reviews in molecular, cellular and developmental biology. vol 24. issue 3. 2002-04-09. PMID:11891759. |
a fresh look at the role of camkii in hippocampal synaptic plasticity and memory. |
2002-04-09 |
2023-08-12 |
mouse |
| Christopher Rong. A fresh look at the role of CaMKII in hippocampal synaptic plasticity and memory. BioEssays : news and reviews in molecular, cellular and developmental biology. vol 24. issue 3. 2002-04-09. PMID:11891759. |
genetic analysis of the alpha isoform of camkii in mice support the hypothesis that camkii signaling is required to initiate the formation of new spatial memories in the hippocampus. |
2002-04-09 |
2023-08-12 |
mouse |
| Christopher Rong. A fresh look at the role of CaMKII in hippocampal synaptic plasticity and memory. BioEssays : news and reviews in molecular, cellular and developmental biology. vol 24. issue 3. 2002-04-09. PMID:11891759. |
camkii is also required for the correct induction of long-term potentiation (ltp) in the hippocampus, consistent with the widely held belief that ltp is a mechanism for learning and memory. |
2002-04-09 |
2023-08-12 |
mouse |
| K I Katsura, J Kurihara, H Kato, Y Katayam. Ischemic pre-conditioning affects the subcellular distribution of protein kinase C and calcium/calmodulin-dependent protein kinase II in the gerbil hippocampal CA1 neurons. Neurological research. vol 23. issue 7. 2002-03-20. PMID:11680516. |
the pre-conditioning generally downregulated pkcgamma and camkii in the ca1 hippocampus. |
2002-03-20 |
2023-08-12 |
Not clear |
| C C Huang, Y C Liang, K S Hs. Characterization of the mechanism underlying the reversal of long term potentiation by low frequency stimulation at hippocampal CA1 synapses. The Journal of biological chemistry. vol 276. issue 51. 2002-01-31. PMID:11679581. |
these results suggest that lfs erases ltp through an nmda receptor-mediated activation of pp1 to dephosphorylate amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid receptors and camkii in the ca1 region of the hippocampus. |
2002-01-31 |
2023-08-12 |
Not clear |
| M C Lee, S S Ban, Y J Woo, S U Ki. Calcium/calmodulin kinase II activity of hippocampus in kainate-induced epilepsy. Journal of Korean medical science. vol 16. issue 5. 2002-01-03. PMID:11641537. |
this study investigated calcium/calmodulin kinase ii (camkii) activity related to long-standing neuronal injury of the hippocampus in kainate (ka)-induced experimental temporal lobe epilepsy. |
2002-01-03 |
2023-08-12 |
Not clear |
| M C Lee, S S Ban, Y J Woo, S U Ki. Calcium/calmodulin kinase II activity of hippocampus in kainate-induced epilepsy. Journal of Korean medical science. vol 16. issue 5. 2002-01-03. PMID:11641537. |
clinical seizures, histopathologic changes and camkii activity of the hippocampus were evaluated. |
2002-01-03 |
2023-08-12 |
Not clear |
| M C Lee, S S Ban, Y J Woo, S U Ki. Calcium/calmodulin kinase II activity of hippocampus in kainate-induced epilepsy. Journal of Korean medical science. vol 16. issue 5. 2002-01-03. PMID:11641537. |
hippocampal camkii activity increased significantly 4 and 8 weeks after intra-amygdaloid injection of ka, when late seizures developed. |
2002-01-03 |
2023-08-12 |
Not clear |
| M C Lee, S S Ban, Y J Woo, S U Ki. Calcium/calmodulin kinase II activity of hippocampus in kainate-induced epilepsy. Journal of Korean medical science. vol 16. issue 5. 2002-01-03. PMID:11641537. |
the increased activity of camkii may correlate with appearance of distant damage in the hippocampus. |
2002-01-03 |
2023-08-12 |
Not clear |
| M C Lee, S S Ban, Y J Woo, S U Ki. Calcium/calmodulin kinase II activity of hippocampus in kainate-induced epilepsy. Journal of Korean medical science. vol 16. issue 5. 2002-01-03. PMID:11641537. |
the above results indicate that intra-amygdaloid injection of ka produces excitatory signals for ipsilateral ca3 neurons in the hippocampus and that subsequently increased levels of camkii in postsynaptic neurons induce neuronal injury via phosphorylation of n-methyl-d-aspartate type glutamate receptor. |
2002-01-03 |
2023-08-12 |
Not clear |
| J H Tao-Cheng, L Vinade, C Smith, C A Winters, R Ward, M W Brightman, T S Reese, A Dosemec. Sustained elevation of calcium induces Ca(2+)/calmodulin-dependent protein kinase II clusters in hippocampal neurons. Neuroscience. vol 106. issue 1. 2001-12-04. PMID:11564417. |
treatment of cultured hippocampal neurons with the mitochondrial uncoupler carbonyl cyanide m-chlorophenylhydrazone (cccp) in the absence of glucose mimics ischemic energy depletion and induces formation of ca(2+)/calmodulin-dependent protein kinase ii (camkii) clusters, spherical structures with diameters of 75-175 nm [dosemeci et al., j. neurosci. |
2001-12-04 |
2023-08-12 |
rat |
| A Dosemeci, J H Tao-Cheng, L Vinade, C A Winters, L Pozzo-Miller, T S Rees. Glutamate-induced transient modification of the postsynaptic density. Proceedings of the National Academy of Sciences of the United States of America. vol 98. issue 18. 2001-10-04. PMID:11517322. |
thin-section immunoelectron microscopy of hippocampal cultures indicates that at least part of the observed thickening of psds can be accounted for by an accumulation of ca(2+)/calmodulin-dependent protein kinase ii (camkii) on their cytoplasmic faces. |
2001-10-04 |
2023-08-12 |
human |
| J Kasahara, K Fukunaga, E Miyamot. Activation of calcium/calmodulin-dependent protein kinase IV in long term potentiation in the rat hippocampal CA1 region. The Journal of biological chemistry. vol 276. issue 26. 2001-08-16. PMID:11306573. |
the importance of well characterized calcium/calmodulin-dependent protein kinase (camk) ii in hippocampal long term potentiation (ltp) is widely well established; however, several camks other than camkii are not yet clearly characterized and understood. |
2001-08-16 |
2023-08-12 |
rat |